Roles of Toll-like receptor 2 (TLR2) and superantigens on adaptive immune responses during CNS staphylococcal infection

This publication has 62 references indexed in Scilit:

• Toll-like Receptor 2 Signaling in CD4+ T Lymphocytes Promotes T Helper 17 Responses and Regulates the Pathogenesis of Autoimmune Disease
  Immunity, 2010
• Astrocyte-Restricted Ablation of Interleukin-17-Induced Act1-Mediated Signaling Ameliorates Autoimmune Encephalomyelitis
  Immunity, 2010
• Functional interleukin-17 receptor A is expressed in central nervous system glia and upregulated in experimental autoimmune encephalomyelitis
  Journal of Neuroinflammation, 2009
• MyD88 Expression by CNS-Resident Cells is Pivotal for Eliciting Protective Immunity in Brain Abscesses
  ASN Neuro, 2009
• Mouse γδ T cells are capable of expressing MHC class II molecules, and of functioning as antigen-presenting cells
  Journal of Neuroimmunology, 2008
• Interleukin-17 Production in Central Nervous System-Infiltrating T Cells and Glial Cells Is Associated with Active Disease in Multiple Sclerosis
  The American Journal of Pathology, 2008
• Both TLR2 and TLR4 Are Required for the Effective Immune Response in Staphylococcus aureus-Induced Experimental Murine Brain Abscess
  The American Journal of Pathology, 2008
• Minocycline Modulates Neuroinflammation Independently of Its Antimicrobial Activity in Staphylococcus aureus-Induced Brain Abscess
  The American Journal of Pathology, 2007
• TH-17 cells in the circle of immunity and autoimmunity
  Nature Immunology, 2007
• Effects of low dose GM-CSF on microglial inflammatory profiles to diverse pathogen-associated molecular patterns (PAMPs)
  Journal of Neuroinflammation, 2007