Abstract
BEFORE the introduction of aureomycin it was generally conceded that the so-called primary atypical or "viral" pneumonias were not benefited by the available antimicrobial agents.1 Indeed, the failure of a patient with an acute pulmonary infection to respond favorably to treatment with sulfonamides and penicillin was often accepted as one of the major criteria for the diagnosis of primary atypical pneumonia, provided that evidence for the presence of a definite inciting cause or agent could not be demonstrated. With the establishment of effectiveness of aureomycin and chloramphenicol against rickettsial infections and against infections with organisms of the psittacosis-lymphogranuloma-venereum-trachoma group,2 , 3 it . . .

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