Deletion of Atf6α enhances kainate-induced neuronal death in mice
Authors:
- 1 January 2016
- journal article
- Published by Elsevier in Neurochemistry International
- Vol. 92, 67-74
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education, Science, Technology, Sports, and Culture of Japan (24500419)
This publication has 27 references indexed in Scilit:
- ATF6alpha Promotes Astroglial Activation and Neuronal Survival in a Chronic Mouse Model of Parkinson’s DiseasePLOS ONE, 2012
- Unfolded proteins and endoplasmic reticulum stress in neurodegenerative disordersJournal of Cellular and Molecular Medicine, 2011
- Kainic acid-induced early genes activation and neuronal death in the medial extended amygdala of ratsExperimental and Toxicologic Pathology, 2011
- The Endoplasmic Reticulum Stress Sensor, ATF6α, Protects against Neurotoxin-induced Dopaminergic Neuronal DeathJournal of Biological Chemistry, 2011
- Induction of Liver Steatosis and Lipid Droplet Formation in ATF6α-Knockout Mice Burdened with Pharmacological Endoplasmic Reticulum StressMolecular Biology of the Cell, 2010
- Increased vulnerability of hippocampal pyramidal neurons to the toxicity of kainic acid in OASIS‐deficient miceJournal of Neurochemistry, 2009
- Transcriptional Induction of Mammalian ER Quality Control Proteins Is Mediated by Single or Combined Action of ATF6α and XBP1Developmental Cell, 2007
- Signal integration in the endoplasmic reticulum unfolded protein responseNature Reviews Molecular Cell Biology, 2007
- Endoplasmic Reticulum Stress Inhibition Protects against Excitotoxic Neuronal Injury in the Rat BrainThe Journal of Neuroscience, 2007
- Kainate receptors in hippocampal CA3 subregion: evidence for a role in regulating neurotransmitter releaseNeurochemistry International, 1998