Claudin-18 coupled with EGFR/ERK signaling contributes to the malignant potentials of bile duct cancer
Authors:
- 1 September 2017
- journal article
- research article
- Published by Elsevier in Cancer Letters
- Vol. 403, 66-73
Abstract
No abstract availableKeywords
Funding Information
- JSPS KAKENHI (JP16K08693, JP16K21250, JP26460421)
This publication has 20 references indexed in Scilit:
- Nuclear localization of tricellulin promotes the oncogenic property of pancreatic cancerScientific Reports, 2016
- Claudins in cancer: bench to bedsidePflügers Archiv - European Journal of Physiology, 2016
- An immunohistochemical marker panel including claudin-18, maspin, and p53 improves diagnostic accuracy of bile duct neoplasms in surgical and presurgical biopsy specimensVirchows Archiv, 2014
- Claudins and the Modulation of Tight Junction PermeabilityPhysiological Reviews, 2013
- Tight junction‐related human diseasesPathology International, 2013
- Claudin-18 in biliary neoplasms. Its significance in the classification of intrahepatic cholangiocarcinomaVirchows Archiv, 2011
- Transcriptional regulation of claudin‐18 via specific protein kinase C signaling pathways and modification of DNA methylation in human pancreatic cancer cellsJournal of Cellular Biochemistry, 2011
- Vandetanib (ZD6474), an inhibitor of VEGFR and EGFR signalling, as a novel molecular-targeted therapy against cholangiocarcinomaBritish Journal of Cancer, 2009
- Transcriptional activation of the humanclaudin-18gene promoter through two AP-1 motifs in PMA-stimulated MKN45 gastric cancer cellsAmerican Journal of Physiology-Gastrointestinal and Liver Physiology, 2008
- Biliary tract cancer treatment: results from the Biliary Tract Cancer Statistics Registry in JapanJournal of Hepato-Biliary-Pancreatic Surgery, 2002