Alterations in Cell Signaling in Sepsis
Open Access
- 15 November 2005
- journal article
- review article
- Published by Oxford University Press (OUP) in Clinical Infectious Diseases
- Vol. 41 (Supplement) , S459-S464
Abstract
Multiple intracellular signaling pathways involving kinases, transcriptional factors, and the expression of immunoregulatory mediators are altered inKeywords
This publication has 58 references indexed in Scilit:
- Human Toll‐Like Receptor 4 Mutations but Not CD14 Polymorphisms Are Associated with an Increased Risk of Gram‐Negative InfectionsThe Journal of Infectious Diseases, 2002
- NF-κB at the crossroads of life and deathNature Immunology, 2002
- The role of TLR4 in endotoxin responsiveness in humansInnate Immunity, 2001
- Transcription Factor NFκB Expression and Postsurgical Organ DysfunctionAnnals of Surgery, 2001
- Molecular mechanisms of NF-κB activation induced by bacterial lipopolysaccharide through Toll-like receptorsInnate Immunity, 2000
- NF-κB activationCritical Care Medicine, 2000
- Activators and target genes of Rel/NF-κB transcription factorsOncogene, 1999
- Endotoxin-tolerant Mice Have Mutations in Toll-like Receptor 4 (Tlr4)Journal of Experimental Medicine, 1999
- Phosphorylation of NF-κB p65 by PKA Stimulates Transcriptional Activity by Promoting a Novel Bivalent Interaction with the Coactivator CBP/p300Molecular Cell, 1998
- Signal transduction through NF-κBImmunology Today, 1998