LATS kinase–mediated CTCF phosphorylation and selective loss of genomic binding
Open Access
- 21 February 2020
- journal article
- research article
- Published by American Association for the Advancement of Science (AAAS) in Science Advances
- Vol. 6 (8) , eaaw4651
Abstract
LATS kinase–activating signals selectively modulate CTCF genomic occupancy and 3D genome organization.Keywords
This publication has 83 references indexed in Scilit:
- Structural Basis for the Versatile and Methylation-Dependent Binding of CTCF to DNAMolecular Cell, 2017
- Editing DNA Methylation in the Mammalian GenomeCell, 2016
- CTCF: making the right connectionsGenes & Development, 2016
- CTCF: an architectural protein bridging genome topology and functionNature Reviews Genetics, 2014
- Topological domains in mammalian genomes identified by analysis of chromatin interactionsNature, 2012
- Global mitotic phosphorylation of C2H2zinc finger protein linker peptidesCell Cycle, 2011
- Regulation of the Transcription Factor YY1 in Mitosis through Phosphorylation of Its DNA-binding DomainMolecular Biology of the Cell, 2009
- Ultrafast and memory-efficient alignment of short DNA sequences to the human genomeGenome Biology, 2009
- Tumor Suppressor LATS1 Is a Negative Regulator of Oncogene YAPJournal of Biological Chemistry, 2008
- DNA Recognition by Cys2His2 Zinc Finger ProteinsAnnual Review of Biophysics, 2000