Atrogin1/MAFbx
Authors:
- 8 July 2011
- journal article
- editorial
- Published by Wolters Kluwer Health in Circulation Research
- Vol. 109 (2) , 123-126
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- Endogenous Muscle Atrophy F-Box Mediates Pressure Overload–Induced Cardiac Hypertrophy Through Regulation of Nuclear Factor-κBCirculation Research, 2011
- During muscle atrophy, thick, but not thin, filament components are degraded by MuRF1-dependent ubiquitylationJournal of Cell Biology, 2009
- Proteasome inhibition promotes regression of left ventricular hypertrophyAmerican Journal of Physiology-Heart and Circulatory Physiology, 2008
- Atrophy-related ubiquitin ligases atrogin-1 and MuRF-1 are associated with uterine smooth muscle involution in the postpartum periodAmerican Journal of Physiology-Regulatory, Integrative and Comparative Physiology, 2007
- Rapid disuse and denervation atrophy involve transcriptional changes similar to those of muscle wasting during systemic diseasesThe FASEB Journal, 2006
- Degradation of MyoD Mediated by the SCF (MAFbx) Ubiquitin LigaseJournal of Biological Chemistry, 2005
- Foxo Transcription Factors Induce the Atrophy-Related Ubiquitin Ligase Atrogin-1 and Cause Skeletal Muscle AtrophyCell, 2004
- Multiple types of skeletal muscle atrophy involve a common program of changes in gene expressionThe FASEB Journal, 2004
- Identification of Ubiquitin Ligases Required for Skeletal Muscle AtrophyScience, 2001
- Atrogin-1, a muscle-specific F-box protein highly expressed during muscle atrophyProceedings of the National Academy of Sciences, 2001