p53 activity contributes to defective interfollicular epidermal differentiation in hyperproliferative murine skin
Open Access
- 20 November 2015
- journal article
- Published by Oxford University Press (OUP) in British Journal of Dermatology
- Vol. 174 (1) , 204-208
Abstract
No abstract availableKeywords
Funding Information
- Medical Research Council
- Wellcome Trust
- Cancer Research UK
- Seventh Framework Programme
- University of Cambridge
- Monash University
This publication has 15 references indexed in Scilit:
- E-box-independent regulation of transcription and differentiation by MYCNature Cell Biology, 2011
- Psoriasis: what we have learned from mouse modelsNature Reviews Rheumatology, 2010
- Dose and context dependent effects of Myc on epidermal stem cell proliferation and differentiationEMBO Molecular Medicine, 2009
- Expression of p53 protein in psoriasis.2005
- Peroxisome-Proliferator-Activated Receptor (PPAR)-γ Activation Stimulates Keratinocyte DifferentiationJournal of Investigative Dermatology, 2004
- Peroxisome Proliferator-Activated Receptor (PPAR)-β/δ Stimulates Differentiation and Lipid Accumulation in KeratinocytesJournal of Investigative Dermatology, 2004
- Physiological and retinoid‐induced proliferations of epidermis basal keratinocytes are differently controlledThe EMBO Journal, 2002
- Critical roles of PPARβ/δ in keratinocyte response to inflammationGenes & Development, 2001
- c-Myc activation in transgenic mouse epidermis results in mobilization of stem cells and differentiation of their progenyCurrent Biology, 2001
- Paradoxical tumor inhibitory effect of p53 loss in transgenic mice expressing epidermal-targeted v-rasHa, v-fos, or human transforming growth factor alpha.1996