John McGrath
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Research Interests
John McGrath has a total of 2 co-authors. Here are the top co-authors by number of shared publications:
Alan Brown
College of Physicians and Surgeons of Columbia University, New York State Psychiatric Institute, New York, NY
1 shared publicationDavid St Clair
Institute of Medical Sciences, University of Aberdeen, Foresterhill, Aberdeen, UK
1 shared publication
Latest Publications
- Open Access
- 25 October 2010
- Published by Oxford University Press (OUP) in Schizophrenia Bulletin
- Vol. 37 (2) , 272-283
Prevention and Schizophrenia--The Role of Dietary Factors
Adequate prenatal nutrition is essential for optimal brain development. There is a growing body of evidence from epidemiology linking exposure to nutritional deprivation and increased risk of schizophrenia. Based on studies from the Netherlands and China, those exposed to macronutrient deficiencies during famine have an increased risk of schizophrenia. With respect to micronutrients, we focus on 3 candidates where there is biological plausibility for a role in this disorder and at least 1 study of an association with schizophrenia. These nutrients include vitamin D, folic acid, and iron. While the current evidence is incomplete, we discuss the potential implications of these findings for the prevention of schizophrenia. We argue that schizophrenia can draw inspiration from public health interventions related to prenatal nutrition and other outcomes and speculate on relevant factors that bear on the nature, risks, impact, and logistics of various nutritional strategies that may be employed to prevent this disorder.
- 25 October 2010
- Published by Oxford University Press (OUP) in Schizophrenia Bulletin
- Vol. 37 (2) , 272-283
Open AccessPrevention and Schizophrenia--The Role of Dietary Factors
Adequate prenatal nutrition is essential for optimal brain development. There is a growing body of evidence from epidemiology linking exposure to nutritional deprivation and increased risk of schizophrenia. Based on studies from the Netherlands and China, those exposed to macronutrient deficiencies during famine have an increased risk of schizophrenia. With respect to micronutrients, we focus on 3 candidates where there is biological plausibility for a role in this disorder and at least 1 study of an association with schizophrenia. These nutrients include vitamin D, folic acid, and iron. While the current evidence is incomplete, we discuss the potential implications of these findings for the prevention of schizophrenia. We argue that schizophrenia can draw inspiration from public health interventions related to prenatal nutrition and other outcomes and speculate on relevant factors that bear on the nature, risks, impact, and logistics of various nutritional strategies that may be employed to prevent this disorder.