Nicole Y. L. Oei
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Nicole Y. L. Oei has a total of 9 co-authors. Here are the top co-authors by number of shared publications:
Bernet M. Elzinga
Department of Clinical, Health and Neuropsychology, Institute of Psychology, Leiden University, Leiden 2Leiden Institute for Brain and Cognition, Leiden 3Department of Radiology, Leiden University Medical Center, Leiden, The Netherlands 4Department of Cognitive Psychology, Ruhr-University Bochum, Germany 5Department of Psychiatry, Leiden University Medical Center, Leiden, The Netherlands
2 shared publicationsPhilip Spinhoven
Department of Clinical, Health and Neuropsychology, Institute of Psychology, Leiden University, Leiden 2Leiden Institute for Brain and Cognition, Leiden 3Department of Radiology, Leiden University Medical Center, Leiden, The Netherlands 4Department of Cognitive Psychology, Ruhr-University Bochum, Germany 5Department of Psychiatry, Leiden University Medical Center, Leiden, The Netherlands
2 shared publicationsDiana van Heemst
Department of Gerontology and Geriatrics, Leiden University Medical Center, Leiden, The Netherlands; Netherlands Consortium of Healthy Ageing, The Netherlands
1 shared publicationIlya M. Veer
Department of Clinical, Health and Neuropsychology, Institute of Psychology, Leiden University, Leiden 2Leiden Institute for Brain and Cognition, Leiden 3Department of Radiology, Leiden University Medical Center, Leiden, The Netherlands 4Department of Cognitive Psychology, Ruhr-University Bochum, Germany 5Department of Psychiatry, Leiden University Medical Center, Leiden, The Netherlands
1 shared publicationJeroen van der Grond
Department of Radiology, Leiden University Medical Center, Leiden, The Netherlands
1 shared publicationMarieke S. Tollenaar
1 shared publication
Latest Publications
- 1 January 2014
- Published by Elsevier in Psychoneuroendocrinology
- Vol. 39, 111-120
Acute stress-induced cortisol elevations mediate reward system activity during subconscious processing of sexual stimuli
- 1 January 2014
- Published by Elsevier in Psychoneuroendocrinology
- Vol. 39, 111-120
Acute stress-induced cortisol elevations mediate reward system activity during subconscious processing of sexual stimuli
- Open Access
- 1 April 2011
- Published by Oxford University Press (OUP) in Social Cognitive and Affective Neuroscience
- Vol. 7 (4) , 403-412
Stress shifts brain activation towards ventral ‘affective’ areas during emotional distraction
Acute stress has been shown to impair working memory (WM), and to decrease prefrontal activation during WM in healthy humans. Stress also enhances amygdala responses towards emotional stimuli. Stress might thus be specifically detrimental to WM when one is distracted by emotional stimuli. Usually, emotional stimuli presented as distracters in a WM task slow down performance, while evoking more activation in ventral ‘affective’ brain areas, and a relative deactivation in dorsal ‘executive’ areas. We hypothesized that after acute social stress, this reciprocal dorsal–ventral pattern would be shifted towards greater increase of ventral ‘affective’ activation during emotional distraction, while impairing WM performance. To investigate this, 34 healthy men, randomly assigned to a social stress or control condition, performed a Sternberg WM task with emotional and neutral distracters inside an MRI scanner. Results showed that WM performance after stress tended to be slower during emotional distraction. Brain activations during emotional distraction was enhanced in ventral affective areas, while dorsal executive areas tended to show less deactivation after stress. These results suggest that acute stress shifts priority towards processing of emotionally significant stimuli, at the cost of WM performance.
- 1 April 2011
- Published by Oxford University Press (OUP) in Social Cognitive and Affective Neuroscience
- Vol. 7 (4) , 403-412
Open AccessStress shifts brain activation towards ventral ‘affective’ areas during emotional distraction
Acute stress has been shown to impair working memory (WM), and to decrease prefrontal activation during WM in healthy humans. Stress also enhances amygdala responses towards emotional stimuli. Stress might thus be specifically detrimental to WM when one is distracted by emotional stimuli. Usually, emotional stimuli presented as distracters in a WM task slow down performance, while evoking more activation in ventral ‘affective’ brain areas, and a relative deactivation in dorsal ‘executive’ areas. We hypothesized that after acute social stress, this reciprocal dorsal–ventral pattern would be shifted towards greater increase of ventral ‘affective’ activation during emotional distraction, while impairing WM performance. To investigate this, 34 healthy men, randomly assigned to a social stress or control condition, performed a Sternberg WM task with emotional and neutral distracters inside an MRI scanner. Results showed that WM performance after stress tended to be slower during emotional distraction. Brain activations during emotional distraction was enhanced in ventral affective areas, while dorsal executive areas tended to show less deactivation after stress. These results suggest that acute stress shifts priority towards processing of emotionally significant stimuli, at the cost of WM performance.
- 1 March 2010
- Published by Elsevier in Neurobiology of Learning and Memory
- Vol. 93 (3) , 388-395
Propranolol reduces emotional distraction in working memory: A partial mediating role of propranolol-induced cortisol increases?
- 1 March 2010
- Published by Elsevier in Neurobiology of Learning and Memory
- Vol. 93 (3) , 388-395
Propranolol reduces emotional distraction in working memory: A partial mediating role of propranolol-induced cortisol increases?