Alfred Dieudonné Kinana
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Alfred Dieudonné Kinana has a total of 11 co-authors. Here are the top co-authors by number of shared publications:
Benoit Garin
3 shared publicationsCheikh Saad-Bouh Boye
3 shared publicationsEric Cardinale
3 shared publicationsFatou Tall
3 shared publicationsIbrahim Bahsoun
3 shared publicationsJean-David Perrier-Gros-Claude
3 shared publications
Latest Publications
- Open Access
- 3 July 2009
- Published by Oxford University Press (OUP) in Journal of Antimicrobial Chemotherapy
- Vol. 64 (3) , 650-652
Contribution of efflux to antibiotic resistance in Campylobacter isolated from poultry in Senegal
A. D. K. thanks the BSAC for his Overseas Scholarship.
- 3 July 2009
- Published by Oxford University Press (OUP) in Journal of Antimicrobial Chemotherapy
- Vol. 64 (3) , 650-652
Open AccessContribution of efflux to antibiotic resistance in Campylobacter isolated from poultry in Senegal
A. D. K. thanks the BSAC for his Overseas Scholarship.
- 1 April 2007
- Published by Elsevier in International Journal of Antimicrobial Agents
- Vol. 29 (4) , 397-401
Analysis of topoisomerase mutations in fluoroquinolone-resistant and -susceptible Campylobacter jejuni strains isolated in Senegal
- 1 April 2007
- Published by Elsevier in International Journal of Antimicrobial Agents
- Vol. 29 (4) , 397-401
Analysis of topoisomerase mutations in fluoroquinolone-resistant and -susceptible Campylobacter jejuni strains isolated in Senegal
- 1 March 2007
- Published by Elsevier in Research in Microbiology
- Vol. 158 (2) , 138-142
Campylobacter coli isolates derived from chickens in Senegal: Diversity, genetic exchange with Campylobacter jejuni and quinolone resistance
- 1 March 2007
- Published by Elsevier in Research in Microbiology
- Vol. 158 (2) , 138-142
Campylobacter coli isolates derived from chickens in Senegal: Diversity, genetic exchange with Campylobacter jejuni and quinolone resistance
- 1 May 2006
- Published by American Society for Microbiology in Applied and Environmental Microbiology
- Vol. 72 (5) , 3309-13
Genetic Diversity and Quinolone Resistance in Campylobacter jejuni Isolates from Poultry in Senegal
We used the multilocus sequence typing (MLST) method to evaluate the genetic diversity of 46 Campylobacter jejuni isolates from chickens and to determine the link between quinolone resistance and sequence type (ST). There were a total of 16 ST genotypes, and the majority of them belonged to seven clonal complexes previously identified by using isolates from human disease. The ST-353 complex was the most common complex, whereas the ST-21, ST-42, ST-52, and ST-257 complexes were less well represented. The resistance phenotype varied for each ST, and the Thr-86-Ile substitution in the GyrA protein was the predominant mechanism of resistance to quinolone. Nine of the 14 isolates having the Thr-86-Ile substitution belonged to the ST-353 complex. MLST showed that the emergence of quinolone resistance is not related to the diffusion of a unique clone and that there is no link between ST genotype and quinolone resistance. Based on silent mutations, different variants of the gyrA gene were shown to exist for the same ST. These data provide useful information for understanding the epidemiology of C. jejuni in Senegal.
- 1 May 2006
- Published by American Society for Microbiology in Applied and Environmental Microbiology
- Vol. 72 (5) , 3309-13
Genetic Diversity and Quinolone Resistance in Campylobacter jejuni Isolates from Poultry in Senegal
We used the multilocus sequence typing (MLST) method to evaluate the genetic diversity of 46 Campylobacter jejuni isolates from chickens and to determine the link between quinolone resistance and sequence type (ST). There were a total of 16 ST genotypes, and the majority of them belonged to seven clonal complexes previously identified by using isolates from human disease. The ST-353 complex was the most common complex, whereas the ST-21, ST-42, ST-52, and ST-257 complexes were less well represented. The resistance phenotype varied for each ST, and the Thr-86-Ile substitution in the GyrA protein was the predominant mechanism of resistance to quinolone. Nine of the 14 isolates having the Thr-86-Ile substitution belonged to the ST-353 complex. MLST showed that the emergence of quinolone resistance is not related to the diffusion of a unique clone and that there is no link between ST genotype and quinolone resistance. Based on silent mutations, different variants of the gyrA gene were shown to exist for the same ST. These data provide useful information for understanding the epidemiology of C. jejuni in Senegal.