Errol C. Friedberg
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Previous Organizations / Institutes
The organizations / institutes from the scholar's other articles in the past ten years
Research Subjects
Errol C. Friedberg has a total of 60 co-authors. Here are the top co-authors by number of shared publications:
- Lisiane Borges Meira
Department of Clinical and Experimental Medicine, University of Surrey, Daphne Jackson Road, Guildford GU2 7WG, UK
6 shared publications William J. Feaver
Laboratory of Molecular Pathology, Department of Pathology, University of Texas Southwestern Medical Center, Dallas, TX 75390-9072
5 shared publicationsDavid L Cheo
Laboratory of Molecular Pathology, Department of Pathology, Southwestern Medical Center, University of Texas, Dallas, TX 75235, USA
3 shared publicationsAntonio M Reis
Laboratory of Molecular Pathology, Department of Pathology, Southwestern Medical Center, University of Texas, Dallas, TX 75235, USA
2 shared publicationsCaixia Guo
Laboratory of Molecular Pathology, Department of Pathology, University of Texas Southwestern Medical Center, Dallas, TX 75390-9072, USA
2 shared publicationsDennis K Burns
Laboratory of Molecular Pathology, Department of Pathology, Southwestern Medical Center, University of Texas, Dallas, TX 75235, USA
2 shared publications
Latest Publications
- 1 January 2016
- Published by Springer Nature
- p. 4017-4018
Repair of DNA
- 20 November 2015
- Published by Springer Nature
- p. 1-2
Repair of DNA
- 1 January 2011
- Published by Springer Nature
- p. 3253-3254
Repair of DNA
- 1 December 2006
- Published by Taylor & Francis in Molecular and Cellular Biology
- Vol. 26 (23) , 8892-8900
Ubiquitin-Binding Motifs in REV1 Protein Are Required for Its Role in the Tolerance of DNA Damage
REV1 protein is a eukaryotic member of the Y family of DNA polymerases involved in the tolerance of DNA damage by replicative bypass. The precise role(s) of REV1 in this process is not known. Here we show, by using the yeast two-hybrid assay and the glutathione S-transferase pull-down assay, that mouse REV1 can physically interact with ubiquitin. The association of REV1 with ubiquitin requires the ubiquitin-binding motifs (UBMs) located at the C terminus of REV1. The UBMs also mediate the enhanced association between monoubiquitylated PCNA and REV1. In cells exposed to UV radiation, the association of REV1 with replication foci is dependent on functional UBMs. The UBMs of REV1 are shown to contribute to DNA damage tolerance and damage-induced mutagenesis in vivo.
- 1 December 2006
- Published by Taylor & Francis in Molecular and Cellular Biology
- Vol. 26 (23) , 8892-8900
Ubiquitin-Binding Motifs in REV1 Protein Are Required for Its Role in the Tolerance of DNA Damage
REV1 protein is a eukaryotic member of the Y family of DNA polymerases involved in the tolerance of DNA damage by replicative bypass. The precise role(s) of REV1 in this process is not known. Here we show, by using the yeast two-hybrid assay and the glutathione S-transferase pull-down assay, that mouse REV1 can physically interact with ubiquitin. The association of REV1 with ubiquitin requires the ubiquitin-binding motifs (UBMs) located at the C terminus of REV1. The UBMs also mediate the enhanced association between monoubiquitylated PCNA and REV1. In cells exposed to UV radiation, the association of REV1 with replication foci is dependent on functional UBMs. The UBMs of REV1 are shown to contribute to DNA damage tolerance and damage-induced mutagenesis in vivo.
- 7 March 2006
- Published by Elsevier in DNA Repair
- Vol. 5 (3) , 291-293
Fraud in science—Reflections on some whys and wherefores
- 7 March 2006
- Published by Elsevier in DNA Repair
- Vol. 5 (3) , 291-293
Fraud in science—Reflections on some whys and wherefores
- 28 September 2005
- Published by Elsevier in DNA Repair
- Vol. 4 (10) , 1066-1067
Journal impact factor, 2004
- 28 September 2005
- Published by Elsevier in DNA Repair
- Vol. 4 (10) , 1066-1067
Journal impact factor, 2004
- 1 September 2005
- Published by Elsevier in Mutation Research - Fundamental and Molecular Mechanisms of Mutagenesis
- Vol. 577 (1-2) , 9-13
Reminiscences of a long-time colleague and friend, Philip C. Hanawalt
- 1 September 2005
- Published by Elsevier in Mutation Research - Fundamental and Molecular Mechanisms of Mutagenesis
- Vol. 577 (1-2) , 9-13
Reminiscences of a long-time colleague and friend, Philip C. Hanawalt
- 1 May 2005
- Published by Elsevier in Molecular Cell
- Vol. 18 (5) , 499-505
Trading Places: How Do DNA Polymerases Switch during Translesion DNA Synthesis?
- 1 May 2005
- Published by Elsevier in Molecular Cell
- Vol. 18 (5) , 499-505
Trading Places: How Do DNA Polymerases Switch during Translesion DNA Synthesis?
- 4 April 2005
- Published by Elsevier in DNA Repair
- Vol. 4 (4) , 407-408
Erling Christen Seeberg (1946–2004)
- 4 April 2005
- Published by Elsevier in DNA Repair
- Vol. 4 (4) , 407-408
Erling Christen Seeberg (1946–2004)