Maud S. Giorgi
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Maud S. Giorgi has a total of 6 co-authors. Here are the top co-authors by number of shared publications:
Peter Vogel
University of Lausanne
2 shared publications- Philippe Christe
Department of Ecology and Evolution, University of Lausanne, UNIL-Sorge, Biophore, Lausanne, Switzerland
2 shared publications - Raphaël Arlettaz
Division of Conservation Biology, Institute of Ecology and Evolution, University of Bern, Bern, Switzerland
2 shared publications Carlo Ossola
University of Lausanne
1 shared publication- Frédéric Guillaume
Department of Organismal and Evolutionary Biology, University of Helsinki, 00014 Helsinki, Finland
1 shared publication - Sébastien Nusslé
Department of Ecology and Evolution, University of Lausanne, Biophore, 1015 Lausanne, Switzerland
1 shared publication
Latest Publications
- 9 January 2004
- Published by Springer Nature in Oecologia
- Vol. 138 (4) , 648-654
Causal mechanisms underlying host specificity in bat ectoparasites
In parasites, host specificity may result either from restricted dispersal capacity or from fixed coevolutionary host-parasite adaptations. Knowledge of those proximal mechanisms leading to particular host specificity is fundamental to understand host-parasite interactions and potential coevolution of parasites and hosts. The relative importance of these two mechanisms was quantified through infection and cross-infection experiments using mites and bats as a model. Monospecific pools of parasitic mites (Spinturnix myoti and S. andegavinus) were subjected either to individual bats belonging to their traditional, native bat host species, or to another substitute host species within the same bat genus (Myotis). The two parasite species reacted differently to these treatments. S. myoti exhibited a clear preference for, and had a higher fitness on, its native host, Myotis myotis. In contrast, S. andegavinus showed no host choice, although its fitness was higher on its native host M. daubentoni. The causal mechanisms mediating host specificity can apparently differ within closely related host-parasite systems.
- 9 January 2004
- Published by Springer Nature in Oecologia
- Vol. 138 (4) , 648-654
Causal mechanisms underlying host specificity in bat ectoparasites
In parasites, host specificity may result either from restricted dispersal capacity or from fixed coevolutionary host-parasite adaptations. Knowledge of those proximal mechanisms leading to particular host specificity is fundamental to understand host-parasite interactions and potential coevolution of parasites and hosts. The relative importance of these two mechanisms was quantified through infection and cross-infection experiments using mites and bats as a model. Monospecific pools of parasitic mites (Spinturnix myoti and S. andegavinus) were subjected either to individual bats belonging to their traditional, native bat host species, or to another substitute host species within the same bat genus (Myotis). The two parasite species reacted differently to these treatments. S. myoti exhibited a clear preference for, and had a higher fitness on, its native host, Myotis myotis. In contrast, S. andegavinus showed no host choice, although its fitness was higher on its native host M. daubentoni. The causal mechanisms mediating host specificity can apparently differ within closely related host-parasite systems.
- Open Access
- 7 October 2001
- Published by The Royal Society in Proceedings Of The Royal Society B-Biological Sciences
- Vol. 268 (1480) , 2071-2075
The energetic grooming costs imposed by a parasitic mite (Spinturnix myoti) upon its bat host (Myotis myotis)
Parasites often exert severe negative effects upon their host's fitness. Natural selection has therefore prompted the evolution of anti–parasite mechanisms such as grooming. Grooming is efficient at reducing parasitic loads in both birds and mammals, but the energetic costs it entails have not been properly quantified. We measured both the energetic metabolism and behaviour of greater mouse–eared bats submitted to three different parasite loads (no, 20 and 40 mites) during whole daily cycles. Mites greatly affected their time and energy budgets. They caused increased grooming activity, reduced the overall time devoted to resting and provoked a dramatic shortening of resting bout duration. Correspondingly, the bats' overall metabolism (oxygen consumption) increased drastically with parasite intensity and, during the course of experiments, the bats lost more weight when infested with 40 rather than 20 or no parasites. The short–term energetic constraints induced by anti–parasite grooming are probably associated with long-term detrimental effects such as a decrease in survival and overall reproductive value.
- 7 October 2001
- Published by The Royal Society in Proceedings Of The Royal Society B-Biological Sciences
- Vol. 268 (1480) , 2071-2075
Open AccessThe energetic grooming costs imposed by a parasitic mite (Spinturnix myoti) upon its bat host (Myotis myotis)
Parasites often exert severe negative effects upon their host's fitness. Natural selection has therefore prompted the evolution of anti–parasite mechanisms such as grooming. Grooming is efficient at reducing parasitic loads in both birds and mammals, but the energetic costs it entails have not been properly quantified. We measured both the energetic metabolism and behaviour of greater mouse–eared bats submitted to three different parasite loads (no, 20 and 40 mites) during whole daily cycles. Mites greatly affected their time and energy budgets. They caused increased grooming activity, reduced the overall time devoted to resting and provoked a dramatic shortening of resting bout duration. Correspondingly, the bats' overall metabolism (oxygen consumption) increased drastically with parasite intensity and, during the course of experiments, the bats lost more weight when infested with 40 rather than 20 or no parasites. The short–term energetic constraints induced by anti–parasite grooming are probably associated with long-term detrimental effects such as a decrease in survival and overall reproductive value.