Dennis McGinty
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Research Interests
Dennis McGinty has a total of 8 co-authors. Here are the top co-authors by number of shared publications:
Ronald Szymusiak
Veterans Affairs Greater Los Angeles HealthCare System, 16111 Plummer Street, North Hills, CA 91343, USA
2 shared publicationsDiane Shepard
1 shared publicationEt al
1 shared publicationHui Gong
Departamento de Neurociencias, Instituto de Fisiologı́a Celular, Universidad Nacional Autónoma de México, 04510 México, D.F., Mexico
1 shared publicationMargaret N. Shouse
1 shared publicationMd.Noor Alam
Veterans Affairs Greater Los Angeles HealthCare System, 16111 Plummer Street, North Hills, CA 91343, USA
1 shared publication
Latest Publications
- 1 September 2000
- Published by Elsevier in Brain Research
- Vol. 875 (1-2) , 23-34
Discharge modulation of rat dorsal raphe neurons during sleep and waking: effects of preoptic/basal forebrain warming
- 1 September 2000
- Published by Elsevier in Brain Research
- Vol. 875 (1-2) , 23-34
Discharge modulation of rat dorsal raphe neurons during sleep and waking: effects of preoptic/basal forebrain warming
- 1 January 1990
- Published by American Psychological Association (APA) in Behavioral Neuroscience
- Vol. 104 (1) , 217-225
Effects of systemic atropine sulfate administration on the frequency content of the cat sensorimotor EEG during sleep and waking.
Sensorimotor electroencephalogram (EEG) frequencies in cats were evaluated with power spectral analysis before and after 3 doses of atropine sulfate. All doses of atropine tested caused enhanced EEG slow waves (0-7 Hz) and spindles (8-15 Hz) during waking immobility, and postdrug frequency profiles during slow-wave sleep and waking immobility were identical. With 0.75 mg/kg atropine, movement (head movement, locomotion) resulted in EEG desynchronization and reduced power in all frequencies less than 24 Hz. After 1.5 or 3.0 mg/kg atropine, power in low frequencies remained elevated during movement, but power in spindle frequencies was significantly reduced compared with other states. During active REM sleep after 1.5 mg/kg atropine, power in spindle frequencies was significantly lower than that during quiet REM sleep. These results indicate that the sensorimotor cortical EEG in cats is under the control of multiple systems. At least 1 of these systems is active during movement, and its actions are resistant to muscarinic receptor blockade.
- 1 January 1990
- Published by American Psychological Association (APA) in Behavioral Neuroscience
- Vol. 104 (1) , 217-225
Effects of systemic atropine sulfate administration on the frequency content of the cat sensorimotor EEG during sleep and waking.
Sensorimotor electroencephalogram (EEG) frequencies in cats were evaluated with power spectral analysis before and after 3 doses of atropine sulfate. All doses of atropine tested caused enhanced EEG slow waves (0-7 Hz) and spindles (8-15 Hz) during waking immobility, and postdrug frequency profiles during slow-wave sleep and waking immobility were identical. With 0.75 mg/kg atropine, movement (head movement, locomotion) resulted in EEG desynchronization and reduced power in all frequencies less than 24 Hz. After 1.5 or 3.0 mg/kg atropine, power in low frequencies remained elevated during movement, but power in spindle frequencies was significantly reduced compared with other states. During active REM sleep after 1.5 mg/kg atropine, power in spindle frequencies was significantly lower than that during quiet REM sleep. These results indicate that the sensorimotor cortical EEG in cats is under the control of multiple systems. At least 1 of these systems is active during movement, and its actions are resistant to muscarinic receptor blockade.