MUMohammad N. Uddin
Mohammad N. Uddin
*This is an algorithmically generated author record based on publicly available information.
Research Subjects
Research Interests
Mohammad N. Uddin has a total of 25 co-authors. Here are the top co-authors by number of shared publications:
- Thomas J. Kuehl
Baylor Scott & White/Texas A&M HSC-COM, Temple, TX
5 shared publications Steven R. Allen
4 shared publicationsDean Leonard
3 shared publications- David C. Zawieja
Department of Medical Physiology, Texas A&M Health Science Center, Temple, TX 76504
2 shared publications - Lena Perger
Department of Surgery, Texas A&M College of Medicine, McLane's Children's Hospital at Scott & White , Temple, Texas
2 shared publications - Madhava R. Beeram
Department of Pediatrics, Baylor Scott & White Health, Temple, USA
2 shared publications
Latest Publications
- 1 January 2017
- Published by Elsevier in American Journal of Obstetrics and Gynecology
- Vol. 216 (1) , S469-S470
818: A single-chain derivative of the relaxin hormone (b7-33) protects cytotrophoblasts from hyperglycemia-induced preeclampsia phenotype and induces the survival pathway
- 1 January 2017
- Published by Elsevier in American Journal of Obstetrics and Gynecology
- Vol. 216 (1) , S469-S470
818: A single-chain derivative of the relaxin hormone (b7-33) protects cytotrophoblasts from hyperglycemia-induced preeclampsia phenotype and induces the survival pathway
- 1 January 2015
- Published by Elsevier in Pregnancy Hypertension
- Vol. 5 (1) , 138
[277-POS]
- 1 January 2015
- Published by Elsevier in Pregnancy Hypertension
- Vol. 5 (1) , 133
[266-POS]
- 1 January 2015
- Published by Elsevier in Pregnancy Hypertension
- Vol. 5 (1) , 113-114
[225-POS]
- 1 August 2012
- Published by Elsevier in Translational Research
- Vol. 160 (2) , 99-113
Pathogenesis of pre-eclampsia: marinobufagenin and angiogenic imbalance as biomarkers of the syndrome
- 1 August 2012
- Published by Elsevier in Translational Research
- Vol. 160 (2) , 99-113
Pathogenesis of pre-eclampsia: marinobufagenin and angiogenic imbalance as biomarkers of the syndrome