Alan H. Jobe
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Research Interests
Alan H. Jobe has a total of 28 co-authors. Here are the top co-authors by number of shared publications:
- Anna M Dusick1 shared publication
- Betty R. Vohr
Department of Pediatrics, Women & Infants Hospital, Brown University, Providence, RI, USA
1 shared publication - Carol E. Nicholson
National Institute of Child Health and Human Development, 6100 Executive Boulevard, Room 2A03 MSC 7510, Bethesda, MD 20892, USA
1 shared publication - Deborah Hirtz
National Institutes of Health, Bethesda, MD, USA
1 shared publication - Debra Birenbaum1 shared publication
Eduardo Bancalari
Children's Hospital Medical Center, Division of Pulmonary Biology, Cincinnati, Ohio; and Department of Pediatrics, University of Miami, Miami, Florida
1 shared publication
Latest Publications
- Open Access
- 1 November 2004
- Published by American Academy of Pediatrics (AAP) in Pediatrics
- Vol. 114 (Supplement)
Conference Participants
- 1 November 2004
- Published by American Academy of Pediatrics (AAP) in Pediatrics
- Vol. 114 (Supplement)
Open AccessConference Participants
- Top Cited Papers
- 1 June 2001
- Published by Oxford University Press (OUP) in American Journal of Respiratory and Critical Care Medicine
- Vol. 163 (7) , 1723-1729
Bronchopulmonary Dysplasia
Bronchopulmonary dysplasia is a chronic lung disease that affects premature babies and contributes to their morbidity and mortality. Improved survival of very immature infants has led to increased numbers of infants with this disorder. This increase puts a heavy burden on health resources since these infants need frequent re-admission to hospital in the first 2 years after birth and, even as adolescents, have lung-function abnormalities and persistent respiratory symptoms. Unlike the original description of the disease in 1967, premature infants can develop chronic oxygen dependency without severe, acute respiratory distress; this “new bronchopulmonary dysplasia” could be the result of impaired postnatal lung growth. Whether such infants subsequently have catch-up lung growth, especially if given corticosteroids postnatally, is unknown. No safe and effective preventive therapy has been identified, but promising new treatments directed either at reducing lung injury or improving lung growth are under study.
- 1 June 2001
- Published by Oxford University Press (OUP) in American Journal of Respiratory and Critical Care Medicine
- Vol. 163 (7) , 1723-1729
Top Cited PapersBronchopulmonary Dysplasia
Bronchopulmonary dysplasia is a chronic lung disease that affects premature babies and contributes to their morbidity and mortality. Improved survival of very immature infants has led to increased numbers of infants with this disorder. This increase puts a heavy burden on health resources since these infants need frequent re-admission to hospital in the first 2 years after birth and, even as adolescents, have lung-function abnormalities and persistent respiratory symptoms. Unlike the original description of the disease in 1967, premature infants can develop chronic oxygen dependency without severe, acute respiratory distress; this “new bronchopulmonary dysplasia” could be the result of impaired postnatal lung growth. Whether such infants subsequently have catch-up lung growth, especially if given corticosteroids postnatally, is unknown. No safe and effective preventive therapy has been identified, but promising new treatments directed either at reducing lung injury or improving lung growth are under study.