Britta A. Hanson
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Research Interests
Britta A. Hanson has a total of 7 co-authors. Here are the top co-authors by number of shared publications:
David E. Stallknecht
Southeastern Cooperative Wildlife Disease Study, Department of Population Health, College of Veterinary Medicine, The University of Georgia, Athens, GA 30602, USA
1 shared publication- David E. Stallknecht
Southeastern Cooperative Wildlife Disease Study, College of Veterinary Medicine, The University of Georgia, Athens, Georgia 30602, USA
1 shared publication - David E. Swayne
U.S. National Poultry Research Center, Agricultural Research Service, U.S. Department of Agriculture, Athens, GA 30605, USA
1 shared publication Dennis A. Senne
National Veterinary Services Laboratories, Veterinary Services, Animal and Plant Health Inspection Service, USDA, Ames, IA50010, USA
1 shared publication- Elizabeth W. Howerth
Department of Pathology College of Veterinary Medicine University of Georgia Athens Georgia USA
1 shared publication Justin Hurst
Texas Parks and Wildlife Department, 1120 Hodges Lane, Wharton, Texas 77488, USA
1 shared publication
Latest Publications
- 1 July 2006
- Published by Wildlife Disease Association in Journal of Wildlife Diseases
- Vol. 42 (3) , 616-624
MOLECULAR CHARACTERIZATION OF EPIZOOTIC HEMORRHAGIC DISEASE VIRUS SEROTYPE 1 ASSOCIATED WITH A 1999 EPIZOOTIC IN WHITE-TAILED DEER IN THE EASTERN UNITED STATES
During the autumn of 1999 (mid-August-late September), an outbreak of hemorrhagic disease in white-tailed deer (Odocoileus virginianus) caused by epizootic hemorrhagic disease virus serotype 1 (EHDV-1) occurred along the east coast of the United States from Georgia to New Jersey. An EHDV-1 epizootic of such magnitude had not been described in this region since 1975. To determine the genetic relatedness among the 1999 viruses, as well as among additional EHDV-1 isolates from the eastern and western United States, portions of the S10 and L2 gene segments were sequenced and compared utilizing phylogenetic analyses. Nearly all of the 1999 eastern isolates were identical in nucleotide sequence at one or both loci. Additionally, confirmed cases of EHDV-1 in white-tailed deer occurred in a south (Georgia)-to-north (New Jersey/Virginia) progression over a short period of approximately six weeks. Taken together, these results indicate that this outbreak resulted from the spread of a single viral strain. The phylograms derived from analysis of the entire sample set displayed eastern and western region-specific clusterings (topotypes), as well as an eastern versus western difference in branch lengths, which may reflect the influence of epizootic versus enzootic transmission patterns on viral genetic diversity.
- 1 July 2006
- Published by Wildlife Disease Association in Journal of Wildlife Diseases
- Vol. 42 (3) , 616-624
MOLECULAR CHARACTERIZATION OF EPIZOOTIC HEMORRHAGIC DISEASE VIRUS SEROTYPE 1 ASSOCIATED WITH A 1999 EPIZOOTIC IN WHITE-TAILED DEER IN THE EASTERN UNITED STATES
During the autumn of 1999 (mid-August-late September), an outbreak of hemorrhagic disease in white-tailed deer (Odocoileus virginianus) caused by epizootic hemorrhagic disease virus serotype 1 (EHDV-1) occurred along the east coast of the United States from Georgia to New Jersey. An EHDV-1 epizootic of such magnitude had not been described in this region since 1975. To determine the genetic relatedness among the 1999 viruses, as well as among additional EHDV-1 isolates from the eastern and western United States, portions of the S10 and L2 gene segments were sequenced and compared utilizing phylogenetic analyses. Nearly all of the 1999 eastern isolates were identical in nucleotide sequence at one or both loci. Additionally, confirmed cases of EHDV-1 in white-tailed deer occurred in a south (Georgia)-to-north (New Jersey/Virginia) progression over a short period of approximately six weeks. Taken together, these results indicate that this outbreak resulted from the spread of a single viral strain. The phylograms derived from analysis of the entire sample set displayed eastern and western region-specific clusterings (topotypes), as well as an eastern versus western difference in branch lengths, which may reflect the influence of epizootic versus enzootic transmission patterns on viral genetic diversity.
- 1 July 2005
- Published by Wildlife Disease Association in Journal of Wildlife Diseases
- Vol. 41 (3) , 624-628
Avian Influenza Viruses and Paramyxoviruses in Wintering and Resident Ducks in Texas
Cloacal swabs were collected from teal (Anas crecca, Anas cyanoptera, Anas discors), mottled duck (Anas fulvigula) and northern pintail (Anas acuta) in Brazoria County, Texas, USA, during February 2001, mottled ducks during August 2001, and blue-winged teal (A. discors) during February 2002. Prevalence of avian influenza virus (AIV) infections during each sampling period were 11, 0, and 15%, respectively. The hemagglutinin (H) subtypes H2 and H7 were detected in both years, while the H8 subtype was detected in 2001 and the H1 subtype was detected in 2002. Avian paramyxovirus type 1 (APMV-1) was isolated from 13% of mottled ducks sampled in August 2001 and 30.7% of teal in February 2002. The season of isolation of both viruses and the majority of the AIV subtypes detected in this study are not typical based on previous reports of these viruses from North American ducks.
- 1 July 2005
- Published by Wildlife Disease Association in Journal of Wildlife Diseases
- Vol. 41 (3) , 624-628
Avian Influenza Viruses and Paramyxoviruses in Wintering and Resident Ducks in Texas
Cloacal swabs were collected from teal (Anas crecca, Anas cyanoptera, Anas discors), mottled duck (Anas fulvigula) and northern pintail (Anas acuta) in Brazoria County, Texas, USA, during February 2001, mottled ducks during August 2001, and blue-winged teal (A. discors) during February 2002. Prevalence of avian influenza virus (AIV) infections during each sampling period were 11, 0, and 15%, respectively. The hemagglutinin (H) subtypes H2 and H7 were detected in both years, while the H8 subtype was detected in 2001 and the H1 subtype was detected in 2002. Avian paramyxovirus type 1 (APMV-1) was isolated from 13% of mottled ducks sampled in August 2001 and 30.7% of teal in February 2002. The season of isolation of both viruses and the majority of the AIV subtypes detected in this study are not typical based on previous reports of these viruses from North American ducks.