Ramses Forsyth
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Previous Organizations / Institutes
The organizations / institutes from the scholar's other articles in the past ten years
Ramses Forsyth has a total of 342 co-authors. Here are the top co-authors by number of shared publications:
- Guillaume E. Courtoy
Laboratory of Experimental Pathology (EXPA), Vrije Universiteit Brussel, Brussels, Belgium
18 shared publications - Wim Waelput
Laboratory of Experimental Pathology (EXPA), Vrije Universiteit Brussel, Brussels, Belgium
18 shared publications - Aleksandar Murgaski
King’s College London 3 Centre for Inflammation Biology and Cancer Immunology, Cancer Research UK City of London Centre, School of Immunology and Microbial Sciences London, United Kingdom
17 shared publications - Ayla Debraekeleer
Lab of Cellular and Molecular Immunology, Brussels Center for Immunology, Vrije Universiteit Brussel, Brussels, Belgium
17 shared publications - Bruno Dombrecht
13VIB Discovery Sciences, VIB, Ghent, Belgium
17 shared publications - Catelijne Stortelers
13VIB Discovery Sciences, VIB, Ghent, Belgium
17 shared publications
Latest Publications
- Open Access
- 10 August 2026
- Published by Springer Nature in Breast Cancer Research
- Vol. 28 (1)
Micro-CT radiomics reveals the microcalcification-surrounding tissue interface as the diagnostic epicenter of breast microcalcifications
- 10 August 2026
- Published by Springer Nature in Breast Cancer Research
- Vol. 28 (1)
Open AccessMicro-CT radiomics reveals the microcalcification-surrounding tissue interface as the diagnostic epicenter of breast microcalcifications
- Open Access
- 1 December 2025
- Published by American Association for Cancer Research (AACR)
Figure S9 from Depleting IL1R2+ Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
Similarity between mouse/human tiTregs and the activation state of tiTreg subsets in E0771 model
- 1 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S9 from Depleting IL1R2+ Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
Similarity between mouse/human tiTregs and the activation state of tiTreg subsets in E0771 model
- Open Access
- 1 December 2025
- Published by American Association for Cancer Research (AACR)
Figure S11 from Depleting IL1R2+ Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
IL1R2 is functionally redundant on tiTregs
- 1 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S11 from Depleting IL1R2+ Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
IL1R2 is functionally redundant on tiTregs
- Open Access
- 1 December 2025
- Published by American Association for Cancer Research (AACR)
Figure S4 from Depleting IL1R2+ Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
Transcriptomic analysis of the different cell populations infiltrating human TNBC and CRC tumors
- 1 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S4 from Depleting IL1R2+ Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
Transcriptomic analysis of the different cell populations infiltrating human TNBC and CRC tumors
- Open Access
- 1 December 2025
- Published by American Association for Cancer Research (AACR)
Figure S6 from Depleting IL1R2+ Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
TiTreg subsets in the MC38 model
- 1 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S6 from Depleting IL1R2+ Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
TiTreg subsets in the MC38 model
- Open Access
- 1 December 2025
- Published by American Association for Cancer Research (AACR)
Figure S10 from Depleting IL1R2+ Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
Prediction of the active regulons in the different tiTreg clusters as determined via SCENIC
- 1 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S10 from Depleting IL1R2+ Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
Prediction of the active regulons in the different tiTreg clusters as determined via SCENIC
- Open Access
- 1 December 2025
- Published by American Association for Cancer Research (AACR)
Figure S14 from Depleting IL1R2+ Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
Some TME immune components of E0771 and MC38 tumors when treated with anti-IL1R2 Nb-Fc monotherapy
- 1 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S14 from Depleting IL1R2+ Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
Some TME immune components of E0771 and MC38 tumors when treated with anti-IL1R2 Nb-Fc monotherapy
- Open Access
- 1 December 2025
- Published by American Association for Cancer Research (AACR)
Figure S1 from Depleting IL1R2+ Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
Representative gating strategy for the different lymphoid and myeloid cell subsets infiltrating the TME
- 1 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S1 from Depleting IL1R2+ Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
Representative gating strategy for the different lymphoid and myeloid cell subsets infiltrating the TME
- Open Access
- 1 December 2025
- Published by American Association for Cancer Research (AACR)
Figure S5 from Depleting IL1R2+ Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
TiTreg subsets in the E0771 model
- 1 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S5 from Depleting IL1R2+ Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
TiTreg subsets in the E0771 model
- Open Access
- 1 December 2025
- Published by American Association for Cancer Research (AACR)
Figure S13 from Depleting IL1R2+ Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
Results of the monotherapy with different anti-IL1R2 Nb-Fc constructs in E0771 and MC38 tumor-bearing mice
- 1 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S13 from Depleting IL1R2+ Tumor-Infiltrating Regulatory T Cells with an ADCC-Prone Nanobody Construct Boosts the Efficacy of Anti–PD-1 Immunotherapy
Results of the monotherapy with different anti-IL1R2 Nb-Fc constructs in E0771 and MC38 tumor-bearing mice