Anil K. Malhotra
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Previous Organizations / Institutes
The organizations / institutes from the scholar's other articles in the past ten years
Research Subjects
Anil K. Malhotra has a total of 30 co-authors. Here are the top co-authors by number of shared publications:
Deepak K. Sarpal
From the Department of Psychiatry, Zucker Hillside Hospital, North Shore-LIJ Health System, Glen Oaks, N.Y.; the Center for Psychiatric Neuroscience, Feinstein Institute for Medical Research, Manhasset, N.Y.; the Department of Psychiatry, Hofstra North Shore-LIJ School of Medicine, Hempstead, N.Y.; and the Department of Mathematics, Hofstra University, Hempstead, N.Y.
2 shared publicationsDelbert G. Robinson
From the Department of Psychiatry, Zucker Hillside Hospital, North Shore-LIJ Health System, Glen Oaks, N.Y.; the Center for Psychiatric Neuroscience, Feinstein Institute for Medical Research, Manhasset, N.Y.; the Department of Psychiatry, Hofstra North Shore-LIJ School of Medicine, Hempstead, N.Y.; and the Department of Mathematics, Hofstra University, Hempstead, N.Y.
2 shared publicationsJohn M. Kane
From the Department of Psychiatry, Zucker Hillside Hospital, North Shore-LIJ Health System, Glen Oaks, N.Y.; the Center for Psychiatric Neuroscience, Feinstein Institute for Medical Research, Manhasset, N.Y.; the Department of Psychiatry, Hofstra North Shore-LIJ School of Medicine, Hempstead, N.Y.; and the Department of Mathematics, Hofstra University, Hempstead, N.Y.
2 shared publicationsJuan A. Gallego
From the Department of Psychiatry, Zucker Hillside Hospital, North Shore-LIJ Health System, Glen Oaks, N.Y.; the Center for Psychiatric Neuroscience, Feinstein Institute for Medical Research, Manhasset, N.Y.; the Department of Psychiatry, Hofstra North Shore-LIJ School of Medicine, Hempstead, N.Y.; and the Department of Mathematics, Hofstra University, Hempstead, N.Y.
2 shared publications- Katherine H. Karlsgodt
University of California, Los Angeles
2 shared publications - Miklos Argyelan
Feinstein Institutes for Medical Research, Institute of Behavioral Science, Manhasset, USA
2 shared publications
Latest Publications
- 1 February 2017
- Published by Elsevier in Neuroscience
- Vol. 343, 423-433
Polyunsaturated fatty acid biostatus, phospholipase A2 activity and brain white matter microstructure across adolescence
- 1 February 2017
- Published by Elsevier in Neuroscience
- Vol. 343, 423-433
Polyunsaturated fatty acid biostatus, phospholipase A2 activity and brain white matter microstructure across adolescence
- 1 January 2016
- Published by American Psychiatric Association Publishing in American Journal of Psychiatry
- Vol. 173 (1) , 69-77
Baseline Striatal Functional Connectivity as a Predictor of Response to Antipsychotic Drug Treatment
Objective:Clinical response to antipsychotic drug treatment is highly variable, yet prognostic biomarkers are lacking. The authors recently demonstrated that successful antipsychotic drug treatment...
- 1 January 2016
- Published by American Psychiatric Association Publishing in American Journal of Psychiatry
- Vol. 173 (1) , 69-77
Baseline Striatal Functional Connectivity as a Predictor of Response to Antipsychotic Drug Treatment
Objective:Clinical response to antipsychotic drug treatment is highly variable, yet prognostic biomarkers are lacking. The authors recently demonstrated that successful antipsychotic drug treatment...
- Open Access
- 2 September 2015
- Published by Oxford University Press (OUP) in Schizophrenia Bulletin
- Vol. 41 (6) , 1224-1226
Dissecting the Heterogeneity of Treatment Response in First-Episode Schizophrenia
The Mental Health Centers for Intervention Development and Applied Research (CIDAR) program prioritized research to provide an evidence base for biomarker development. At the Zucker Hillside Hospital (ZHH), our CIDAR grant supported research on a comprehensive investigation of treatment response and outcome in first episode schizophrenia. Results provide evidence that baseline neuroimaging, neurocognitive, and genetic measures are significantly associated with clinical response to treatment, and that our currently available interventions can effectively treat aspects of psychotic illness, as well as potentially reduce comorbidity associated with illness. Future research may include combining modalities to more robustly predict response and identify treatment targets, as well as to further develop more effective intervention strategies for these devastating and disabling disorders.
- 2 September 2015
- Published by Oxford University Press (OUP) in Schizophrenia Bulletin
- Vol. 41 (6) , 1224-1226
Open AccessDissecting the Heterogeneity of Treatment Response in First-Episode Schizophrenia
The Mental Health Centers for Intervention Development and Applied Research (CIDAR) program prioritized research to provide an evidence base for biomarker development. At the Zucker Hillside Hospital (ZHH), our CIDAR grant supported research on a comprehensive investigation of treatment response and outcome in first episode schizophrenia. Results provide evidence that baseline neuroimaging, neurocognitive, and genetic measures are significantly associated with clinical response to treatment, and that our currently available interventions can effectively treat aspects of psychotic illness, as well as potentially reduce comorbidity associated with illness. Future research may include combining modalities to more robustly predict response and identify treatment targets, as well as to further develop more effective intervention strategies for these devastating and disabling disorders.
- Top Cited PapersOpen Access
- 1 January 2015
- Published by American Medical Association (AMA) in JAMA Psychiatry
- Vol. 72 (1) , 5-13
Antipsychotic Treatment and Functional Connectivity of the Striatum in First-Episode Schizophrenia
Disruptions in corticostriatal circuitry have been implicated in the pathophysiology of schizophrenia. Early proposals linked schizophrenia with decreases in dopamine in the prefrontal cortex and excessive dopamine in the striatum.1,2 Although elevated striatal dopamine has been shown in patients with schizophrenia,3 their unaffected relatives,4 and individuals who are at risk for developing psychotic symptoms,5 corticostriatal relationships have also been demonstrated to play a central role in psychosis. Early positron emission tomography work found that the severity of psychotic symptoms correlated with abnormal patterns of blood flow in limbic and prefrontal cortical regions.6 Studies in schizophrenia using functional magnetic resonance imaging (fMRI) have reported abnormal corticostriatal activation during reward7,8 and executive processing.9,10 Evidence from functional connectivity and multimodal studies have shown altered corticostriatal circuitry in patients with chronic schizophrenia11,12 and in patients with prodromal psychotic symptoms.13,14 A previous family-based study suggested that altered functional connectivity between striatum and cortical regions may represent a risk phenotype in patients with first-episode psychosis (FEP) and their relatives.15
- 1 January 2015
- Published by American Medical Association (AMA) in JAMA Psychiatry
- Vol. 72 (1) , 5-13
Top Cited PapersOpen AccessAntipsychotic Treatment and Functional Connectivity of the Striatum in First-Episode Schizophrenia
Disruptions in corticostriatal circuitry have been implicated in the pathophysiology of schizophrenia. Early proposals linked schizophrenia with decreases in dopamine in the prefrontal cortex and excessive dopamine in the striatum.1,2 Although elevated striatal dopamine has been shown in patients with schizophrenia,3 their unaffected relatives,4 and individuals who are at risk for developing psychotic symptoms,5 corticostriatal relationships have also been demonstrated to play a central role in psychosis. Early positron emission tomography work found that the severity of psychotic symptoms correlated with abnormal patterns of blood flow in limbic and prefrontal cortical regions.6 Studies in schizophrenia using functional magnetic resonance imaging (fMRI) have reported abnormal corticostriatal activation during reward7,8 and executive processing.9,10 Evidence from functional connectivity and multimodal studies have shown altered corticostriatal circuitry in patients with chronic schizophrenia11,12 and in patients with prodromal psychotic symptoms.13,14 A previous family-based study suggested that altered functional connectivity between striatum and cortical regions may represent a risk phenotype in patients with first-episode psychosis (FEP) and their relatives.15
- 1 June 2009
- Published by Elsevier in Schizophrenia Research
- Vol. 111 (1-3) , 167-173
Acute dopamine depletion with branched chain amino acids decreases auditory top-down event-related potentials in healthy subjects
- 1 June 2009
- Published by Elsevier in Schizophrenia Research
- Vol. 111 (1-3) , 167-173
Acute dopamine depletion with branched chain amino acids decreases auditory top-down event-related potentials in healthy subjects
- 1 July 2006
- Published by Elsevier in General Hospital Psychiatry
- Vol. 28 (4) , 357-358
Interferon-induced obsessive-compulsive disorder
- 1 July 2006
- Published by Elsevier in General Hospital Psychiatry
- Vol. 28 (4) , 357-358
Interferon-induced obsessive-compulsive disorder