LXLiu Xuan

Liu Xuan

*This is an algorithmically generated author record based on publicly available information.

Current Organizations / Institutes

The organizations/institutes from the scholar's latest article


Liu Xuan has a total of 14 co-authors. Here are the top co-authors by number of shared publications:

  • Adrian Liau

    Indiana University School of Medicine, 410 W. 10th Street, HS 100, Indianapolis, IN 46202, USA

    1 shared publication
  • Biao Cai

    Anhui Academy of Medical Sciences, No. 1 Yonghong Road, Luyang District, Hefei, Anhui 230061, People's Republic of China

    1 shared publication
  • Dong-Qing Ye

    Affiliations Department of Epidemiology and Biostatistics, School of Public Health, Anhui Medical University, Hefei, Anhui, China, Inflammation and Immune Mediated Diseases Laboratory of Anhui Province, Hefei, Anhui, China, School of Public Health, Anhui University of Science and Technology, Hefei, Anhui, China

    1 shared publication
  • Fen Huang

    Department of Epidemiology and Health Statistics, Public Health of Anhui Medical University, Hefei, China

    1 shared publication
  • Fuyang Ge

    Anhui Academy of Medical Sciences, No. 1 Yonghong Road, Luyang District, Hefei, Anhui 230061, People's Republic of China

    1 shared publication
  • Jing Li

    Anhui Academy of Medical Sciences, No. 1 Yonghong Road, Luyang District, Hefei, Anhui 230061, People's Republic of China

    1 shared publication

Latest Publications

  • Open Access

    Hepatitis B virus infections and risk factors among the general population in Anhui Province, China: an epidemiological study

    by Xiaoqing Li, Yingjun Zheng, Adrian Liau, Biao Cai, Dongqing Ye, Feng Huang, Xiaorong Sheng, Fuyang Ge, Liu Xuan, Shun Li, et al.

    Hepatitis B is one of the most common infectious diseases in China. The aim of this study was to determine the prevalence of hepatitis B surface antigen (HBsAg) among the general population and the risk factors associated with HBV infection in Anhui province, China.


    • 1 September 2009
      • Vol. 34  (17) , 2232-5

    [Effect of dachaihutang on expression of carnitine patmitoryl transferase-1 in vascular smooth muscle layer of atherosclerotic rabbits].

    by Liu Xuan, Xiaoqing Li, Xiaorong Sheng, Mingsheng Xu, Xun Che

    To investigate the therapeutic effect of Dachaihutang on the development of atherosclerosis (AS) in rabbits and its possible mechanism by detecting the expression level of carnitine patmitoyl transferase-1 (CPT-1) in vascular smooth muscle layer of atherosclerotic rabbits, and search the new way and evidence for AS cures. Thirty six male New Zealand white rabbits were divided randomly into control group, model control group, simvastatin group and Chinese traditional medicine dachaihutang group. After 9 weeks and 20 weeks of treatment, serum total cholesterol (TC) and triglyceride (TG) and low-density lipoprotein (LDL) levels were examined. At the end of 25 th weeks, histological changes in ascending aorta were studied by HE staining and histomorphometric analysis. The gene expression of CPT-1 in vascular smooth muscle layer of thoracic aorta was detected by semi-quantitative reverse transcription-polymerase chain reaction (RT-PCR). Compared with model control group, in dachaihutang group serum TC and TG and LDL levels attenuated. Pathomorphology indicated that intima and media (I + M) became thinned, and the ratios of the thickness of intima to media(I/M) and the area of intima to media (SI/SM) were decreased (P < 0.05). Aortic intimal proliferation in Dachaihutang group was associated with a marked increase in CPT-1 expression in vascular smooth muscle layer of thoracic aorta. Compared to simvastatin group, except TG value, other values were higher in Dachaihutang group, however, there were no significant differences between the two groups. These findings suggest that early treatment with Dachaihutang not only induces a significant regression of arterial lesions of high cholesterol diet rabbits, but also has a crucial inhibited genesis and development of atherosclerosis effect by up-regulating CPT-1 expression in vascular smooth muscle layer.