Alessandro Carugo
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Alessandro Carugo has a total of 639 co-authors. Here are the top co-authors by number of shared publications:
- Giulio Francesco Draetta
Department of Genomic Medicine, The University of Texas MD Anderson Cancer Center, Houston, USA
42 shared publications - Timothy P. Heffernan
TRACTION platform, The University of Texas MD Anderson Cancer Center, Houston, USA
39 shared publications Christopher A. Bristow
TRACTION platform, The University of Texas MD Anderson Cancer Center, Houston, USA
33 shared publications- Giannicola Genovese
Department of Genomic Medicine, The University of Texas MD Anderson Cancer Center, Houston, Texas
33 shared publications Andrea Viale
Department of Genomic Medicine, The University of Texas MD Anderson Cancer Center, Houston, USA
31 shared publicationsTimothy P. Heffernan
29 shared publications
Latest Publications
- Open Access
- 15 June 2026
- Published by American Association for Cancer Research (AACR)
Table S2 from DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer
Geneset Enrichment analysis of human PDAC cells silenced for WDR5 or DPY30 compared to controls.
- 15 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessTable S2 from DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer
Geneset Enrichment analysis of human PDAC cells silenced for WDR5 or DPY30 compared to controls.
- Open Access
- 15 June 2026
- Published by American Association for Cancer Research (AACR)
Table S4 from DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer
Geneset Enrichment Analysis comparing KP-/-C Dpy30-KO tumors versus control tumors grown in NSG and C57BL/6 mice.
- 15 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessTable S4 from DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer
Geneset Enrichment Analysis comparing KP-/-C Dpy30-KO tumors versus control tumors grown in NSG and C57BL/6 mice.
- Open Access
- 15 June 2026
- Published by American Association for Cancer Research (AACR)
Figure S5 from DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer
DPY30 loss impairs the stability of reversed forks.
- 15 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S5 from DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer
DPY30 loss impairs the stability of reversed forks.
- Open Access
- 15 June 2026
- Published by American Association for Cancer Research (AACR)
Table S1 from DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer
WDR5-dependent H3K4me3 deposition at regulatory regions proximal to gene transcriptional start sites (TSS).
- 15 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessTable S1 from DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer
WDR5-dependent H3K4me3 deposition at regulatory regions proximal to gene transcriptional start sites (TSS).
- Open Access
- 15 June 2026
- Published by American Association for Cancer Research (AACR)
Figure S2 from DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer
DPY30 loss does not affect tumor growth in vivo.
- 15 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S2 from DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer
DPY30 loss does not affect tumor growth in vivo.
- Open Access
- 15 June 2026
- Published by American Association for Cancer Research (AACR)
Table S3 from DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer
DPY30 interactors across different PDAC cell lines.
- 15 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessTable S3 from DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer
DPY30 interactors across different PDAC cell lines.
- Open Access
- 15 June 2026
- Published by American Association for Cancer Research (AACR)
Figure S1 from DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer
DPY30 loss does not impair cell proliferation.
- 15 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S1 from DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer
DPY30 loss does not impair cell proliferation.
- Open Access
- 15 June 2026
- Published by American Association for Cancer Research (AACR)
Figure S3 from DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer
DPY30 loss does not alter H3K4me3 deposition.
- 15 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S3 from DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer
DPY30 loss does not alter H3K4me3 deposition.
- Open Access
- 15 June 2026
- Published by American Association for Cancer Research (AACR)
Figure S4 from DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer
The WRAD core interacts with the replisome machinery.
- 15 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S4 from DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer
The WRAD core interacts with the replisome machinery.
- Open Access
- 15 June 2026
- Published by American Association for Cancer Research (AACR)
Figure S7 from DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer
DPY30 loss potently recruits intra-tumor CD8+ T cell.
- 15 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S7 from DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer
DPY30 loss potently recruits intra-tumor CD8+ T cell.