Chi Van Dang
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Previous Organizations / Institutes
The organizations / institutes from the scholar's other articles in the past ten years
Research Subjects
Chi Van Dang has a total of 1000 co-authors. Here are the top co-authors by number of shared publications:
- Angelo Michael De Marzo
Sidney Kimmel Comprehensive Cancer Center Johns Hopkins University, School of Medicine Baltimore MD USA
25 shared publications - Ben Z. Stanger
Department of Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA
23 shared publications Alexander Muir
Ben May Department for Cancer Research, University of Chicago, Chicago, IL, USA
22 shared publicationsYogev Sela
Abramson Cancer Center, University of Pennsylvania, Philadelphia, PA 19104
22 shared publications- Clementina Mesaros
University of Pennsylvania
21 shared publications Colin Sheehan
21 shared publications
Latest Publications
- 10 November 2026
- Published by Springer Nature in Cellular and Molecular Bioengineering
- p. 1-7
Cancer Metabolism Snapshot: Therapeutic Challenges and Dietary Opportunities
- 10 November 2026
- Published by Springer Nature in Cellular and Molecular Bioengineering
- p. 1-7
Cancer Metabolism Snapshot: Therapeutic Challenges and Dietary Opportunities
- Open Access
- 2 April 2026
- Published by Elsevier in Molecular Cell
- Vol. 86 (7) , 1260-1274.e4
The HIF-2 transcription factor mediates resistance to ferroptosis in pancreatic cancer
- 2 April 2026
- Published by Elsevier in Molecular Cell
- Vol. 86 (7) , 1260-1274.e4
Open AccessThe HIF-2 transcription factor mediates resistance to ferroptosis in pancreatic cancer
- Open Access
- 2 March 2026
- Published by American Association for Cancer Research (AACR)
FigureS1 from Androgen Receptor Drives Polyamine Synthesis, Creating a Vulnerability for Prostate Cancer
SPA increases intracellular and extracellular polyamines in prostate cancer cell lines
- 2 March 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigureS1 from Androgen Receptor Drives Polyamine Synthesis, Creating a Vulnerability for Prostate Cancer
SPA increases intracellular and extracellular polyamines in prostate cancer cell lines
- Open Access
- 2 March 2026
- Published by American Association for Cancer Research (AACR)
FigureS4 from Androgen Receptor Drives Polyamine Synthesis, Creating a Vulnerability for Prostate Cancer
AR-V7 can positively regulate ODC
- 2 March 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigureS4 from Androgen Receptor Drives Polyamine Synthesis, Creating a Vulnerability for Prostate Cancer
AR-V7 can positively regulate ODC
- Open Access
- 2 March 2026
- Published by American Association for Cancer Research (AACR)
FigureS13 from Androgen Receptor Drives Polyamine Synthesis, Creating a Vulnerability for Prostate Cancer
Correlation of metabolites and PSA
- 2 March 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigureS13 from Androgen Receptor Drives Polyamine Synthesis, Creating a Vulnerability for Prostate Cancer
Correlation of metabolites and PSA
- Open Access
- 2 March 2026
- Published by American Association for Cancer Research (AACR)
FigureS2 from Androgen Receptor Drives Polyamine Synthesis, Creating a Vulnerability for Prostate Cancer
SPA increases activity of ODC and AMD1 in prostate cancer cell lines
- 2 March 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigureS2 from Androgen Receptor Drives Polyamine Synthesis, Creating a Vulnerability for Prostate Cancer
SPA increases activity of ODC and AMD1 in prostate cancer cell lines
- Open Access
- 2 March 2026
- Published by American Association for Cancer Research (AACR)
FigureS11 from Androgen Receptor Drives Polyamine Synthesis, Creating a Vulnerability for Prostate Cancer
Inhibition of ODC increases downregulation of MYC by SPA by depleting S-adenosylmethionine
- 2 March 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigureS11 from Androgen Receptor Drives Polyamine Synthesis, Creating a Vulnerability for Prostate Cancer
Inhibition of ODC increases downregulation of MYC by SPA by depleting S-adenosylmethionine
- Open Access
- 2 March 2026
- Published by American Association for Cancer Research (AACR)
FigureS12 from Androgen Receptor Drives Polyamine Synthesis, Creating a Vulnerability for Prostate Cancer
Plasma metabolite analyses of trial participants
- 2 March 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigureS12 from Androgen Receptor Drives Polyamine Synthesis, Creating a Vulnerability for Prostate Cancer
Plasma metabolite analyses of trial participants
- Open Access
- 2 March 2026
- Published by American Association for Cancer Research (AACR)
Table S3 from Androgen Receptor Drives Polyamine Synthesis, Creating a Vulnerability for Prostate Cancer
Table S3 from Androgen Receptor Drives Polyamine Synthesis, Creating a Vulnerability for Prostate Cancer
- 2 March 2026
- Published by American Association for Cancer Research (AACR)
Open AccessTable S3 from Androgen Receptor Drives Polyamine Synthesis, Creating a Vulnerability for Prostate Cancer
Table S3 from Androgen Receptor Drives Polyamine Synthesis, Creating a Vulnerability for Prostate Cancer
- Open Access
- 2 March 2026
- Published by American Association for Cancer Research (AACR)
FigureS9 from Androgen Receptor Drives Polyamine Synthesis, Creating a Vulnerability for Prostate Cancer
DFMO decreases putrecine induced by SPA and does not increase efficacy of AR inhibition
- 2 March 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigureS9 from Androgen Receptor Drives Polyamine Synthesis, Creating a Vulnerability for Prostate Cancer
DFMO decreases putrecine induced by SPA and does not increase efficacy of AR inhibition