Sruti Shiva
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Sruti Shiva has a total of 431 co-authors. Here are the top co-authors by number of shared publications:
- Abdul Yassin-Kassab
Rush University Medical Center
28 shared publications - Avani Gopalkrishnan
Department of Radiation Oncology, University of Pittsburgh School of Medicine, Pittsburgh, Pennsylvania
28 shared publications - Chareeni Kurukulasuriya
Department of Otolaryngology–Head and Neck Surgery, New York University Grossman School of Medicine, New York, New York
28 shared publications - Heath Devin Skinner
UPMC Hillman Cancer Center, Pittsburgh, Pennsylvania
28 shared publications - Kirill Kiselyov
Department of Biological Sciences, University of Pittsburgh, Pittsburgh PA 15260, USA
28 shared publications - Nathaniel Wang
UPMC Hillman Cancer Center, University of Pittsburgh Medical Center, Pittsburgh, Pennsylvania
28 shared publications
Latest Publications
- 18 June 2026
- Published by American Society of Hematology in Blood
- Vol. 147 (25) , 2998-2999
Platelets under pressure: a sticky outside-in problem
- 18 June 2026
- Published by American Society of Hematology in Blood
- Vol. 147 (25) , 2998-2999
Platelets under pressure: a sticky outside-in problem
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Table ST2 from Lysosomal–Mitochondrial Interaction Promotes Tumor Growth in Squamous Cell Carcinoma of the Head and Neck
Supplementary Table ST2. Coevolution of vertebrate lysosomal proteins with mitochondrial inner membrane proteins
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table ST2 from Lysosomal–Mitochondrial Interaction Promotes Tumor Growth in Squamous Cell Carcinoma of the Head and Neck
Supplementary Table ST2. Coevolution of vertebrate lysosomal proteins with mitochondrial inner membrane proteins
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S2 from Lysosomal–Mitochondrial Interaction Promotes Tumor Growth in Squamous Cell Carcinoma of the Head and Neck
Supplementary Figure S2. Mitochondrial function is diminished in TMEM16A knock-down model of HN30
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S2 from Lysosomal–Mitochondrial Interaction Promotes Tumor Growth in Squamous Cell Carcinoma of the Head and Neck
Supplementary Figure S2. Mitochondrial function is diminished in TMEM16A knock-down model of HN30
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Table ST3 from Lysosomal–Mitochondrial Interaction Promotes Tumor Growth in Squamous Cell Carcinoma of the Head and Neck
Supplementary Table ST3. Lysosomal and mitochondrial components that show significant coevolution (at ERC R ≥ 0.3) with 10% of the corresponding organellar proteome and R ≥ 0.3 correlation with TMEM16A expression in upper aerodigestive cancer cell lines
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table ST3 from Lysosomal–Mitochondrial Interaction Promotes Tumor Growth in Squamous Cell Carcinoma of the Head and Neck
Supplementary Table ST3. Lysosomal and mitochondrial components that show significant coevolution (at ERC R ≥ 0.3) with 10% of the corresponding organellar proteome and R ≥ 0.3 correlation with TMEM16A expression in upper aerodigestive cancer cell lines
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S5 from Lysosomal–Mitochondrial Interaction Promotes Tumor Growth in Squamous Cell Carcinoma of the Head and Neck
Supplementary Figure S5. OXPHOS inhibition is detrimental to in vivo growth in high-TMEM16A cells
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S5 from Lysosomal–Mitochondrial Interaction Promotes Tumor Growth in Squamous Cell Carcinoma of the Head and Neck
Supplementary Figure S5. OXPHOS inhibition is detrimental to in vivo growth in high-TMEM16A cells
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S1 from Lysosomal–Mitochondrial Interaction Promotes Tumor Growth in Squamous Cell Carcinoma of the Head and Neck
Supplementary Figure S1. Lysosome-mitochondria interaction in TMEM16A cells is independent of mitophagy
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S1 from Lysosomal–Mitochondrial Interaction Promotes Tumor Growth in Squamous Cell Carcinoma of the Head and Neck
Supplementary Figure S1. Lysosome-mitochondria interaction in TMEM16A cells is independent of mitophagy
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S4 from Lysosomal–Mitochondrial Interaction Promotes Tumor Growth in Squamous Cell Carcinoma of the Head and Neck
Supplementary Figure S4. Transient knockdown of lysosomal genes in OSC19-VC and TMEM16A cells
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S4 from Lysosomal–Mitochondrial Interaction Promotes Tumor Growth in Squamous Cell Carcinoma of the Head and Neck
Supplementary Figure S4. Transient knockdown of lysosomal genes in OSC19-VC and TMEM16A cells
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S7 from Lysosomal–Mitochondrial Interaction Promotes Tumor Growth in Squamous Cell Carcinoma of the Head and Neck
Supplementary Figure S7. Over expression of constitutively active PI3KCA or EGFR does not promote lysosomal or mitochondrial biogenesis
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S7 from Lysosomal–Mitochondrial Interaction Promotes Tumor Growth in Squamous Cell Carcinoma of the Head and Neck
Supplementary Figure S7. Over expression of constitutively active PI3KCA or EGFR does not promote lysosomal or mitochondrial biogenesis
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Table ST4 from Lysosomal–Mitochondrial Interaction Promotes Tumor Growth in Squamous Cell Carcinoma of the Head and Neck
Supplementary Table ST4. Transient knockdown of lysosomal genes in OSC19-VC and TMEM16A cells
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table ST4 from Lysosomal–Mitochondrial Interaction Promotes Tumor Growth in Squamous Cell Carcinoma of the Head and Neck
Supplementary Table ST4. Transient knockdown of lysosomal genes in OSC19-VC and TMEM16A cells
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Table ST6 from Lysosomal–Mitochondrial Interaction Promotes Tumor Growth in Squamous Cell Carcinoma of the Head and Neck
Supplementary Table ST6. qPCR primers used in the study
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table ST6 from Lysosomal–Mitochondrial Interaction Promotes Tumor Growth in Squamous Cell Carcinoma of the Head and Neck
Supplementary Table ST6. qPCR primers used in the study