Motohiro Kato
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Previous Organizations / Institutes
The organizations / institutes from the scholar's other articles in the past ten years
Research Subjects
Motohiro Kato has a total of 1000 co-authors. Here are the top co-authors by number of shared publications:
- Daisuke Tomizawa
Division of Leukemia and Lymphoma, Children's Cancer Center National Center for Child Health and Development Tokyo Japan
105 shared publications - Junko Takita
Department of Pediatrics, Kyoto University Graduate School of Medicine, Kyoto, Japan
93 shared publications - Takao Deguchi
Children's Cancer Center, National Center for Child Health and Development, Tokyo, Japan
85 shared publications - Masatoshi Takagi
Department of Pediatrics and Developmental Biology Institute of Science Tokyo Tokyo Japan
81 shared publications - Jun J. Yang
Department of Pharmacy and Pharmaceutical Sciences St. Jude Children's Research Hospital Memphis Tennessee USA
80 shared publications - Atsushi Manabe
Department of Pediatrics Hokkaido University Graduate School of Medicine Sapporo Japan
78 shared publications
Latest Publications
- Open Access
- 28 October 2026
- Published by Wiley in Pediatric Blood & Cancer
Haploidentical Hematopoietic Cell Transplantation for Infants With X‐Linked Severe Combined Immunodeficiency Using Post‐Transplant Cyclophosphamide
- 28 October 2026
- Published by Wiley in Pediatric Blood & Cancer
Open AccessHaploidentical Hematopoietic Cell Transplantation for Infants With X‐Linked Severe Combined Immunodeficiency Using Post‐Transplant Cyclophosphamide
- 27 July 2026
- Published by Springer Nature in Leukemia
- Vol. 40 (10) , 2346-2349
Revisiting classical genetic subtypes reveals subtype-specific relapse patterns and MRD prognostic relevance in pediatric acute lymphoblastic leukemia
- 27 July 2026
- Published by Springer Nature in Leukemia
- Vol. 40 (10) , 2346-2349
Revisiting classical genetic subtypes reveals subtype-specific relapse patterns and MRD prognostic relevance in pediatric acute lymphoblastic leukemia
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Table S1 from Biologic and Clinical Analysis of Childhood Gamma Delta T-ALL Identifies LMO2/STAG2 Rearrangements as Extremely High Risk
A multi-national consortium of γδ T-ALL study.
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S1 from Biologic and Clinical Analysis of Childhood Gamma Delta T-ALL Identifies LMO2/STAG2 Rearrangements as Extremely High Risk
A multi-national consortium of γδ T-ALL study.
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Table S16 from Biologic and Clinical Analysis of Childhood Gamma Delta T-ALL Identifies LMO2/STAG2 Rearrangements as Extremely High Risk
Differentially expressed genes (adjusted P <0.01 ) between PF382-STAG2KO and PF382-WT.
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S16 from Biologic and Clinical Analysis of Childhood Gamma Delta T-ALL Identifies LMO2/STAG2 Rearrangements as Extremely High Risk
Differentially expressed genes (adjusted P <0.01 ) between PF382-STAG2KO and PF382-WT.
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Table S2 from Biologic and Clinical Analysis of Childhood Gamma Delta T-ALL Identifies LMO2/STAG2 Rearrangements as Extremely High Risk
Comparison of clinical characteristics and outcomes between gamma delta T-ALL and non-gamma delta T-ALL.
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S2 from Biologic and Clinical Analysis of Childhood Gamma Delta T-ALL Identifies LMO2/STAG2 Rearrangements as Extremely High Risk
Comparison of clinical characteristics and outcomes between gamma delta T-ALL and non-gamma delta T-ALL.
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S4 from Biologic and Clinical Analysis of Childhood Gamma Delta T-ALL Identifies LMO2/STAG2 Rearrangements as Extremely High Risk
The genomic features of “Chromosome gains” and “TLX3” subtypes.
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S4 from Biologic and Clinical Analysis of Childhood Gamma Delta T-ALL Identifies LMO2/STAG2 Rearrangements as Extremely High Risk
The genomic features of “Chromosome gains” and “TLX3” subtypes.
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S2 from Biologic and Clinical Analysis of Childhood Gamma Delta T-ALL Identifies LMO2/STAG2 Rearrangements as Extremely High Risk
The gene expression profiles of γδ T-ALL.
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S2 from Biologic and Clinical Analysis of Childhood Gamma Delta T-ALL Identifies LMO2/STAG2 Rearrangements as Extremely High Risk
The gene expression profiles of γδ T-ALL.
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Table S21 from Biologic and Clinical Analysis of Childhood Gamma Delta T-ALL Identifies LMO2/STAG2 Rearrangements as Extremely High Risk
Compounds used in single-dose (10uM) drug screening.
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S21 from Biologic and Clinical Analysis of Childhood Gamma Delta T-ALL Identifies LMO2/STAG2 Rearrangements as Extremely High Risk
Compounds used in single-dose (10uM) drug screening.
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Table S12 from Biologic and Clinical Analysis of Childhood Gamma Delta T-ALL Identifies LMO2/STAG2 Rearrangements as Extremely High Risk
Differentially expressed genes (adjusted P <0.01 ) in STAG2/LMO2 subtype compared to other T-ALL cases.
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S12 from Biologic and Clinical Analysis of Childhood Gamma Delta T-ALL Identifies LMO2/STAG2 Rearrangements as Extremely High Risk
Differentially expressed genes (adjusted P <0.01 ) in STAG2/LMO2 subtype compared to other T-ALL cases.