Andreas Seeber
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Research Interests
Andreas Seeber has a total of 313 co-authors. Here are the top co-authors by number of shared publications:
- Moh'D Khushman
Departments of Medical Oncology, Washington University in St. Louis/Siteman Cancer Center, St. Louis, Missouri, USA
78 shared publications - Upender Manne
Department of Pathology, University of Alabama at Birmingham, Birmingham, AL, USA; O'Neal Comprehensive Cancer Center, University of Alabama at Birmingham, Birmingham, AL, USA
70 shared publications - Amith Varambally
Vestavia Hills High School, Vestavia Hills, Alabama 35216, USA
68 shared publications - Darshan Shimoga Chandrashekar
Department of Pathology, University of Alabama at Birmingham, Birmingham, AL
68 shared publications - Farrukh Afaq
Department of Dermatology, University of Alabama at Birmingham, Birmingham, AL, USA
68 shared publications - Hyung-Gyoon Kim
Department of Pathology, University of Alabama at Birmingham, Birmingham, Alabama
68 shared publications
Latest Publications
- Open Access
- 27 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Figure 8 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis
S8. EZH2 and BZW2 correlation in CRC cells.
- 27 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure 8 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis
S8. EZH2 and BZW2 correlation in CRC cells.
- Open Access
- 27 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Data from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis
Supplementary figure legends 1-8, and supplementary tables 1-5
- 27 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Data from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis
Supplementary figure legends 1-8, and supplementary tables 1-5
- Open Access
- 27 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Figure 2 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis
Supplementary Figure 2 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis
- 27 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure 2 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis
Supplementary Figure 2 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis
- Open Access
- 27 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Figure 6 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis
S6. MYC regulates BZW2 in CRC cells.
- 27 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure 6 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis
S6. MYC regulates BZW2 in CRC cells.
- Open Access
- 27 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Figure 1 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis
S1. BZW2 mRNA overexpression in CRC.
- 27 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure 1 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis
S1. BZW2 mRNA overexpression in CRC.
- Open Access
- 27 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Figure 4 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis
S4. BZW2 is involved in CRC tumor growth.
- 27 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure 4 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis
S4. BZW2 is involved in CRC tumor growth.
- Open Access
- 27 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Figure 5 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis
S5. MALAT1 and NEAT1 are downstream target genes of BZW2 in CRC.
- 27 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure 5 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis
S5. MALAT1 and NEAT1 are downstream target genes of BZW2 in CRC.
- Open Access
- 27 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Figure 7 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis
S7. miR-98-5p regulates BZW2 in CRC cells.
- 27 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure 7 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis
S7. miR-98-5p regulates BZW2 in CRC cells.
- Open Access
- 27 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Figure 3 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis
S3. BZW2-knockdown reduces cell invasion.
- 27 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure 3 from BZW2 Inhibition Reduces Colorectal Cancer Growth and Metastasis
S3. BZW2-knockdown reduces cell invasion.
- Open Access
- 3 June 2025
- Published by American Association for Cancer Research (AACR)
Supplemental Figure S2 from Large-Scale Multiomic Analysis Identifies Anatomic Differences and Immunogenic Potential in Subtypes of Leiomyosarcoma
Differential expression of HIF1A and ESR1 transcripts across LMS subtypes
- 3 June 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplemental Figure S2 from Large-Scale Multiomic Analysis Identifies Anatomic Differences and Immunogenic Potential in Subtypes of Leiomyosarcoma
Differential expression of HIF1A and ESR1 transcripts across LMS subtypes