Jeremy Steven Frieling
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Previous Organizations / Institutes
The organizations / institutes from the scholar's other articles in the past ten years
Jeremy Steven Frieling has a total of 105 co-authors. Here are the top co-authors by number of shared publications:
- Conor C. Lynch
Department of Tumor Microenvironment and Metastasis, H. Lee Moffitt Cancer Center and Research Institute, Tampa, Florida
27 shared publications - Ryan T. Bishop
H. Lee Moffitt Cancer Center and Research Institute 6 Department of Tumor Microenvironment and Metastasis Tampa, Florida
17 shared publications - Tao Li
Department of Tumor Microenvironment & Metastasis, H. Lee Moffitt Cancer Center and Research Institute, Tampa, USA
16 shared publications - Baasil Malik
Department of Tumor Microenvironment and Metastasis, H. Lee Moffitt Cancer Center and Research Institute, Tampa, Florida
15 shared publications - Bishoy Wadie
Genome Biology Unit, European Molecular Biology Laboratory (EMBL), Heidelberg, Germany
15 shared publications - Haley du Bois
Department of Tumor Microenvironment and Metastasis, H. Lee Moffitt Cancer Center and Research Institute, Tampa, Florida
15 shared publications
Latest Publications
- Open Access
- 2 February 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Movie S2 from PRDM16 Regulates Prostate Cancer Cell Dormancy and Prevents Bone Metastatic Outgrowth
PRDM16 stalls cell division upon reawakening
- 2 February 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Movie S2 from PRDM16 Regulates Prostate Cancer Cell Dormancy and Prevents Bone Metastatic Outgrowth
PRDM16 stalls cell division upon reawakening
- Open Access
- 2 February 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Table S3 from PRDM16 Regulates Prostate Cancer Cell Dormancy and Prevents Bone Metastatic Outgrowth
Average normalized values (against Actin or GAPDH) for all protein bands included in immunoblots across this study based on densitometric analysis
- 2 February 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S3 from PRDM16 Regulates Prostate Cancer Cell Dormancy and Prevents Bone Metastatic Outgrowth
Average normalized values (against Actin or GAPDH) for all protein bands included in immunoblots across this study based on densitometric analysis
- Open Access
- 2 February 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Table S2 from PRDM16 Regulates Prostate Cancer Cell Dormancy and Prevents Bone Metastatic Outgrowth
QPCR primers used for gene expression or ChIP analysis
- 2 February 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S2 from PRDM16 Regulates Prostate Cancer Cell Dormancy and Prevents Bone Metastatic Outgrowth
QPCR primers used for gene expression or ChIP analysis
- Open Access
- 2 February 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Movie S1 from PRDM16 Regulates Prostate Cancer Cell Dormancy and Prevents Bone Metastatic Outgrowth
Entry into and exit from dormancy
- 2 February 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Movie S1 from PRDM16 Regulates Prostate Cancer Cell Dormancy and Prevents Bone Metastatic Outgrowth
Entry into and exit from dormancy
- Open Access
- 2 February 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Table S1 from PRDM16 Regulates Prostate Cancer Cell Dormancy and Prevents Bone Metastatic Outgrowth
- 2 February 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S1 from PRDM16 Regulates Prostate Cancer Cell Dormancy and Prevents Bone Metastatic Outgrowth
- Open Access
- 2 February 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S2 from PRDM16 Regulates Prostate Cancer Cell Dormancy and Prevents Bone Metastatic Outgrowth
PRDM16 expression is influenced by known dormancy regulators
- 2 February 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S2 from PRDM16 Regulates Prostate Cancer Cell Dormancy and Prevents Bone Metastatic Outgrowth
PRDM16 expression is influenced by known dormancy regulators
- Open Access
- 2 February 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S1 from PRDM16 Regulates Prostate Cancer Cell Dormancy and Prevents Bone Metastatic Outgrowth
- 2 February 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S1 from PRDM16 Regulates Prostate Cancer Cell Dormancy and Prevents Bone Metastatic Outgrowth
- Open Access
- 2 February 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S4 from PRDM16 Regulates Prostate Cancer Cell Dormancy and Prevents Bone Metastatic Outgrowth
PRDM16 promotes PCa dormancy initiation and survival
- 2 February 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S4 from PRDM16 Regulates Prostate Cancer Cell Dormancy and Prevents Bone Metastatic Outgrowth
PRDM16 promotes PCa dormancy initiation and survival
- Open Access
- 2 February 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S5 from PRDM16 Regulates Prostate Cancer Cell Dormancy and Prevents Bone Metastatic Outgrowth
PRDM16 promotes the survival of dormant DTCs in the bone microenvironment
- 2 February 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S5 from PRDM16 Regulates Prostate Cancer Cell Dormancy and Prevents Bone Metastatic Outgrowth
PRDM16 promotes the survival of dormant DTCs in the bone microenvironment
- Open Access
- 2 February 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S7 from PRDM16 Regulates Prostate Cancer Cell Dormancy and Prevents Bone Metastatic Outgrowth
Schematic model of PRDM16 mediated PCa dormancy
- 2 February 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S7 from PRDM16 Regulates Prostate Cancer Cell Dormancy and Prevents Bone Metastatic Outgrowth
Schematic model of PRDM16 mediated PCa dormancy