James Patrick Allison
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Previous Organizations / Institutes
The organizations / institutes from the scholar's other articles in the past ten years
Research Subjects
James Patrick Allison has a total of 1000 co-authors. Here are the top co-authors by number of shared publications:
- Padmanee Sharma
Department of Genitourinary Medical Oncology, The University of Texas MD Anderson Cancer Center, Houston, TX, USA
210 shared publications - Shalini S. Yadav
Immunotherapy Platform, University of MD Anderson Cancer Center, Houston, Texas. 7
161 shared publications - Sumit K. Subudhi
Department of Genitourinary Medical Oncology, The University of Texas MD Anderson Cancer Center, Houston, TX, USA
143 shared publications - Sreyashi Basu
James P. Allison Institute, The University of Texas MD Anderson Cancer Center, Houston, USA
132 shared publications - Bilal A. Siddiqui
Department of Genitourinary Medical Oncology, The University of Texas MD Anderson Cancer Center, Houston, USA
110 shared publications - Linghua Wang
Genomic Medicine, The University of Texas MD Anderson Cancer Center, Houston, Texas, USA
110 shared publications
Latest Publications
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure 4 from Pancreatic Epithelial IL17/IL17RA Signaling Drives B7-H4 Expression to Promote Tumorigenesis
Supplementary Figure 4. Immunoprofiling of KC mice receiving Wild-Type (WT) Bone Marrow (BM) or Bone Marrow from IL-17RA Knock-Out (KO) Mice.
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure 4 from Pancreatic Epithelial IL17/IL17RA Signaling Drives B7-H4 Expression to Promote Tumorigenesis
Supplementary Figure 4. Immunoprofiling of KC mice receiving Wild-Type (WT) Bone Marrow (BM) or Bone Marrow from IL-17RA Knock-Out (KO) Mice.
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure 7 from Pancreatic Epithelial IL17/IL17RA Signaling Drives B7-H4 Expression to Promote Tumorigenesis
Supplementary Figure 7: Identification of IL-17-producing cell types
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure 7 from Pancreatic Epithelial IL17/IL17RA Signaling Drives B7-H4 Expression to Promote Tumorigenesis
Supplementary Figure 7: Identification of IL-17-producing cell types
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure 16 from Molecular Pathways and Cellular Subsets Associated with Adverse Clinical Outcomes in Overlapping Immune-Related Myocarditis and Myositis
Cell-cell communication analysis from fibroblasts, endothelial cells, and myeloid cells to cytotoxic T cells in myocarditis
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure 16 from Molecular Pathways and Cellular Subsets Associated with Adverse Clinical Outcomes in Overlapping Immune-Related Myocarditis and Myositis
Cell-cell communication analysis from fibroblasts, endothelial cells, and myeloid cells to cytotoxic T cells in myocarditis
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure 10 from Molecular Pathways and Cellular Subsets Associated with Adverse Clinical Outcomes in Overlapping Immune-Related Myocarditis and Myositis
Comparison of myeloid cell subclusters in skeletal muscle with previously reported subsets
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure 10 from Molecular Pathways and Cellular Subsets Associated with Adverse Clinical Outcomes in Overlapping Immune-Related Myocarditis and Myositis
Comparison of myeloid cell subclusters in skeletal muscle with previously reported subsets
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure 11 from Pancreatic Epithelial IL17/IL17RA Signaling Drives B7-H4 Expression to Promote Tumorigenesis
Supplementary Figure 11: B7-H4 is involved in PanINs progression
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure 11 from Pancreatic Epithelial IL17/IL17RA Signaling Drives B7-H4 Expression to Promote Tumorigenesis
Supplementary Figure 11: B7-H4 is involved in PanINs progression
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure 8 from Molecular Pathways and Cellular Subsets Associated with Adverse Clinical Outcomes in Overlapping Immune-Related Myocarditis and Myositis
Single cell TCR sequencing (scRNASeq) from patient cardiac and skeletal muscle tissue
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure 8 from Molecular Pathways and Cellular Subsets Associated with Adverse Clinical Outcomes in Overlapping Immune-Related Myocarditis and Myositis
Single cell TCR sequencing (scRNASeq) from patient cardiac and skeletal muscle tissue
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure 3 from Pancreatic Epithelial IL17/IL17RA Signaling Drives B7-H4 Expression to Promote Tumorigenesis
Supplementary Figure 3. IL-17RA deletion results in an increase in apoptosis, but not proliferation at 30 week time point.
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure 3 from Pancreatic Epithelial IL17/IL17RA Signaling Drives B7-H4 Expression to Promote Tumorigenesis
Supplementary Figure 3. IL-17RA deletion results in an increase in apoptosis, but not proliferation at 30 week time point.
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Table 1 from Molecular Pathways and Cellular Subsets Associated with Adverse Clinical Outcomes in Overlapping Immune-Related Myocarditis and Myositis
Baseline Clinical Characteristics
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table 1 from Molecular Pathways and Cellular Subsets Associated with Adverse Clinical Outcomes in Overlapping Immune-Related Myocarditis and Myositis
Baseline Clinical Characteristics
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure 11 from Molecular Pathways and Cellular Subsets Associated with Adverse Clinical Outcomes in Overlapping Immune-Related Myocarditis and Myositis
Complement activation associated with serum autoantibodies and tissue IgG deposition in ICT-induced myocarditis and myositis
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure 11 from Molecular Pathways and Cellular Subsets Associated with Adverse Clinical Outcomes in Overlapping Immune-Related Myocarditis and Myositis
Complement activation associated with serum autoantibodies and tissue IgG deposition in ICT-induced myocarditis and myositis
- Open Access
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure 1 from Molecular Pathways and Cellular Subsets Associated with Adverse Clinical Outcomes in Overlapping Immune-Related Myocarditis and Myositis
Clinical characteristics of patients with confirmed toxicities
- 17 June 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure 1 from Molecular Pathways and Cellular Subsets Associated with Adverse Clinical Outcomes in Overlapping Immune-Related Myocarditis and Myositis
Clinical characteristics of patients with confirmed toxicities