Nobuharu Hanaoka
Research Subjects
Research Interests
Nobuharu Hanaoka has a total of 7 co-authors. Here are the top co-authors by number of shared publications:
- Akira Takasu
Department of Emergency and Critical Care Medicine, Osaka Medical and Pharmaceutical University, 2-7 Daigaku-machi, Takatsuki City 596-8686, Osaka, Japan
1 shared publication - Kanna Ota
Osaka Healthcare Clinic, Osaka City 531-0074, Osaka, Japan
1 shared publication - Kohei Taniguchi
Department of General and Gastroenterological Surgery Osaka Medical and Pharmaceutical University Takatsuki Osaka Japan
1 shared publication - Koshi Ota
Osaka Medical and Pharmaceutical University
1 shared publication - Ryo Iida
Shunjukai Shiroyama Hosp, Gastroenterol Ctr, Dept Surg, 2-8-1 Habikino, Osaka 5830872, Japan
1 shared publication - Satoshi Fumimoto
Osaka Med Coll, Dept Thorac & Cardiovasc Surg, 2-7 Daigaku Cho, Takatsuki, Osaka 5698686, Japan
1 shared publication
Latest Publications
- Open Access
- 7 October 2018
- Published by Springer Nature in Journal of Medical Case Reports
- Vol. 12 (1) , 291
Massive hemothorax due to two bleeding sources with minor injury mechanism: a case report
Massive hemothorax resulting from a minor injury mechanism is considered to be rare particularly when the diaphragm is injured. We report a case of massive hemothorax with bleeding from the intercostal artery and diaphragmatic damage caused by minor blunt trauma. An 83-year-old Japanese man was transported to our hospital 3 hours after falling out of bed. Computed tomography revealed hemothorax and multiple rib fractures. He underwent fluid resuscitation and a tube thoracostomy, but he became hemodynamically unstable. Contrast-enhanced computed tomography revealed worsening hemothorax with contrast extravasation 4 hours after arrival at the hospital. Emergency angiography indicated hemorrhage in the area supplied by the tenth intercostal artery. Transcatheter arterial embolization stabilized his vital signs for a short period. However, further hemodynamic stabilization required a thoracotomy, which revealed diaphragmatic trauma, which was removed and sutured before fixing his fractured ribs. His postoperative course was uneventful, and he was transferred to another hospital for rehabilitation without complications on hospital day 29. Minor mechanisms of blunt trauma can cause rib fractures and massive hemothorax. Traumatic diaphragm injury should be considered a differential diagnosis if hemodynamic instability persists after transcatheter arterial embolization in patients with lower level rib fractures.
- 7 October 2018
- Published by Springer Nature in Journal of Medical Case Reports
- Vol. 12 (1) , 291
Open AccessMassive hemothorax due to two bleeding sources with minor injury mechanism: a case report
Massive hemothorax resulting from a minor injury mechanism is considered to be rare particularly when the diaphragm is injured. We report a case of massive hemothorax with bleeding from the intercostal artery and diaphragmatic damage caused by minor blunt trauma. An 83-year-old Japanese man was transported to our hospital 3 hours after falling out of bed. Computed tomography revealed hemothorax and multiple rib fractures. He underwent fluid resuscitation and a tube thoracostomy, but he became hemodynamically unstable. Contrast-enhanced computed tomography revealed worsening hemothorax with contrast extravasation 4 hours after arrival at the hospital. Emergency angiography indicated hemorrhage in the area supplied by the tenth intercostal artery. Transcatheter arterial embolization stabilized his vital signs for a short period. However, further hemodynamic stabilization required a thoracotomy, which revealed diaphragmatic trauma, which was removed and sutured before fixing his fractured ribs. His postoperative course was uneventful, and he was transferred to another hospital for rehabilitation without complications on hospital day 29. Minor mechanisms of blunt trauma can cause rib fractures and massive hemothorax. Traumatic diaphragm injury should be considered a differential diagnosis if hemodynamic instability persists after transcatheter arterial embolization in patients with lower level rib fractures.