Valentina Cecchetti
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Valentina Cecchetti has a total of 28 co-authors. Here are the top co-authors by number of shared publications:
Andrea Minisci
1 shared publication- Andrea Torreggiani
Department of Soil, Plant, and Food Sciences, University of Bari, 70125 Bari, Italy
1 shared publication - Angelo De Paolis
Istituto di Scienze delle Produzioni Alimentari (ISPA), Consiglio Nazionale delle Ricerche (CNR), Via Monteroni, 73100 Lecce, Italy
1 shared publication - Carlo Giuseppe Rizzello
Department of Environmental Biology, “Sapienza” University of Rome, 00185 Rome, Italy
1 shared publication - Diego Piacentini
Department of Environmental Biology, Sapienza University of Rome, 00185 Rome, Italy
1 shared publication - Erica Pontonio
Department of the Soil, Plant and Food Science (DiSSPA), University of Bari Aldo Moro, Via Amendola, 165/a, 70126 Bari, Italy
1 shared publication
Latest Publications
- Open Access
- 4 March 2023
- Published by Elsevier in Environmental and Experimental Botany
- Vol. 209
Brassinosteroids interact with nitric oxide in the response of rice root systems to arsenic stress
- 4 March 2023
- Published by Elsevier in Environmental and Experimental Botany
- Vol. 209
Open AccessBrassinosteroids interact with nitric oxide in the response of rice root systems to arsenic stress
- 7 March 2018
- Published by Oxford University Press (OUP) in The Plant Cell
- Vol. 30 (3) , 620-637
A Newly Identified Flower-Specific Splice Variant of AUXIN RESPONSE FACTOR8 Regulates Stamen Elongation and Endothecium Lignification in Arabidopsis
In addition to the full-length transcript ARF8.1, a splice variant (ARF8.2) of the auxin response factor gene ARF8 has been reported. Here, we identified an intron-retaining variant of ARF8.2, ARF8.4, whose translated product is imported into the nucleus and has tissue-specific localization in Arabidopsis thaliana. By inducibly expressing each variant in arf8-7 flowers, we show that ARF8.4 fully complements the short-stamen phenotype of the mutant and restores the expression of AUX/IAA19, encoding a key regulator of stamen elongation. By contrast, the expression of ARF8.2 and ARF8.1 had minor or no effects on arf8-7 stamen elongation and AUX/IAA19 expression. Coexpression of ARF8.2 and ARF8.4 in both the wild type and arf8-7 caused premature anther dehiscence: We show that ARF8.2 is responsible for increased expression of the jasmonic acid biosynthetic gene DAD1 and that ARF8.4 is responsible for premature endothecium lignification due to precocious expression of transcription factor gene MYB26. Finally, we show that ARF8.4 binds to specific auxin-related sequences in both the AUX/IAA19 and MYB26 promoters and activates their transcription more efficiently than ARF8.2. Our data suggest that ARF8.4 is a tissue-specific functional splice variant that controls filament elongation and endothecium lignification by directly regulating key genes involved in these processes.
- 7 March 2018
- Published by Oxford University Press (OUP) in The Plant Cell
- Vol. 30 (3) , 620-637
A Newly Identified Flower-Specific Splice Variant of AUXIN RESPONSE FACTOR8 Regulates Stamen Elongation and Endothecium Lignification in Arabidopsis
In addition to the full-length transcript ARF8.1, a splice variant (ARF8.2) of the auxin response factor gene ARF8 has been reported. Here, we identified an intron-retaining variant of ARF8.2, ARF8.4, whose translated product is imported into the nucleus and has tissue-specific localization in Arabidopsis thaliana. By inducibly expressing each variant in arf8-7 flowers, we show that ARF8.4 fully complements the short-stamen phenotype of the mutant and restores the expression of AUX/IAA19, encoding a key regulator of stamen elongation. By contrast, the expression of ARF8.2 and ARF8.1 had minor or no effects on arf8-7 stamen elongation and AUX/IAA19 expression. Coexpression of ARF8.2 and ARF8.4 in both the wild type and arf8-7 caused premature anther dehiscence: We show that ARF8.2 is responsible for increased expression of the jasmonic acid biosynthetic gene DAD1 and that ARF8.4 is responsible for premature endothecium lignification due to precocious expression of transcription factor gene MYB26. Finally, we show that ARF8.4 binds to specific auxin-related sequences in both the AUX/IAA19 and MYB26 promoters and activates their transcription more efficiently than ARF8.2. Our data suggest that ARF8.4 is a tissue-specific functional splice variant that controls filament elongation and endothecium lignification by directly regulating key genes involved in these processes.