Christian Max Schmidt
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Previous Organizations / Institutes
The organizations / institutes from the scholar's other articles in the past ten years
Research Subjects
Christian Max Schmidt has a total of 503 co-authors. Here are the top co-authors by number of shared publications:
- Michele Yip-Schneider
Department of Surgery, Indiana University School of Medicine, Indianapolis, Indiana
232 shared publications - Anirban Maitra
Laura and Isaac Perlmutter Cancer Center, New York University Grossman School of Medicine, NYU Langone Health, New York City, New York, USA
205 shared publications - Johannes Francois Fahrmann
Department of Clinical Cancer Prevention, The University of Texas MD Anderson Cancer Center, Houston, TX 77030, USA
204 shared publications - Mark W. Hurd
Ahmed Center for Pancreatic Cancer Research, University of Texas MD Anderson Cancer Center, Houston, TX
204 shared publications - Kimal I. Rajapakshe
Department of Gastrointestinal Radiation Oncology, University of Texas MD Anderson Cancer Center, Houston, TX
188 shared publications - Paola A. Guerrero
Department of Translational Molecular Pathology, Sheikh Ahmed Center for Pancreatic Cancer Research, The University of Texas MD Anderson Cancer Center
188 shared publications
Latest Publications
- Open Access
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Table S20 from Spatial Transcriptomics of Intraductal Papillary Mucinous Neoplasms of the Pancreas Identifies NKX6-2 as a Driver of Gastric Differentiation and Indolent Biological Potential
Overlap between genes differentially expressed in the NKX6-2 overexpressing and control Kras;Gnas cell line and genes differentially expressed in Nkx6-2 positive and negative spots from the Kras;Gnas ST epithelial spots.
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessTable S20 from Spatial Transcriptomics of Intraductal Papillary Mucinous Neoplasms of the Pancreas Identifies NKX6-2 as a Driver of Gastric Differentiation and Indolent Biological Potential
Overlap between genes differentially expressed in the NKX6-2 overexpressing and control Kras;Gnas cell line and genes differentially expressed in Nkx6-2 positive and negative spots from the Kras;Gnas ST epithelial spots.
- Open Access
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Table S5 from Spatial Transcriptomics of Intraductal Papillary Mucinous Neoplasms of the Pancreas Identifies NKX6-2 as a Driver of Gastric Differentiation and Indolent Biological Potential
Differentially expressed genes between LG and HGPDAC juxtalesional spots.
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessTable S5 from Spatial Transcriptomics of Intraductal Papillary Mucinous Neoplasms of the Pancreas Identifies NKX6-2 as a Driver of Gastric Differentiation and Indolent Biological Potential
Differentially expressed genes between LG and HGPDAC juxtalesional spots.
- Open Access
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Table S9 from Spatial Transcriptomics of Intraductal Papillary Mucinous Neoplasms of the Pancreas Identifies NKX6-2 as a Driver of Gastric Differentiation and Indolent Biological Potential
Patient and clinical information for the IPMN samples included in the COMET cohort.
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessTable S9 from Spatial Transcriptomics of Intraductal Papillary Mucinous Neoplasms of the Pancreas Identifies NKX6-2 as a Driver of Gastric Differentiation and Indolent Biological Potential
Patient and clinical information for the IPMN samples included in the COMET cohort.
- Open Access
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Table S21 from Spatial Transcriptomics of Intraductal Papillary Mucinous Neoplasms of the Pancreas Identifies NKX6-2 as a Driver of Gastric Differentiation and Indolent Biological Potential
Overlap between genes differentially expressed in the NKX6-2 overexpressing and control Kras;Gnas cell line and genes differentially expressed in NKX6-2 positive and negative epilesional spots from the human LG IPMN dataset.
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessTable S21 from Spatial Transcriptomics of Intraductal Papillary Mucinous Neoplasms of the Pancreas Identifies NKX6-2 as a Driver of Gastric Differentiation and Indolent Biological Potential
Overlap between genes differentially expressed in the NKX6-2 overexpressing and control Kras;Gnas cell line and genes differentially expressed in NKX6-2 positive and negative epilesional spots from the human LG IPMN dataset.
- Open Access
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Table S7 from Spatial Transcriptomics of Intraductal Papillary Mucinous Neoplasms of the Pancreas Identifies NKX6-2 as a Driver of Gastric Differentiation and Indolent Biological Potential
Differentially expressed genes between LG, HG, and PDAC samples per region.
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessTable S7 from Spatial Transcriptomics of Intraductal Papillary Mucinous Neoplasms of the Pancreas Identifies NKX6-2 as a Driver of Gastric Differentiation and Indolent Biological Potential
Differentially expressed genes between LG, HG, and PDAC samples per region.
- Open Access
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Table S13 from Spatial Transcriptomics of Intraductal Papillary Mucinous Neoplasms of the Pancreas Identifies NKX6-2 as a Driver of Gastric Differentiation and Indolent Biological Potential
Correlation between percentage of NKX6-2 positive nuclei and GATA-6, MUC5AC, TFF1, and VIM protein expression.
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessTable S13 from Spatial Transcriptomics of Intraductal Papillary Mucinous Neoplasms of the Pancreas Identifies NKX6-2 as a Driver of Gastric Differentiation and Indolent Biological Potential
Correlation between percentage of NKX6-2 positive nuclei and GATA-6, MUC5AC, TFF1, and VIM protein expression.
- Open Access
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Table S15 from Spatial Transcriptomics of Intraductal Papillary Mucinous Neoplasms of the Pancreas Identifies NKX6-2 as a Driver of Gastric Differentiation and Indolent Biological Potential
Predicted NKX6-2 targets and transcription factors associated with NKX6-2 expression evaluated by GENIE3.
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessTable S15 from Spatial Transcriptomics of Intraductal Papillary Mucinous Neoplasms of the Pancreas Identifies NKX6-2 as a Driver of Gastric Differentiation and Indolent Biological Potential
Predicted NKX6-2 targets and transcription factors associated with NKX6-2 expression evaluated by GENIE3.
- Open Access
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Table S19 from Spatial Transcriptomics of Intraductal Papillary Mucinous Neoplasms of the Pancreas Identifies NKX6-2 as a Driver of Gastric Differentiation and Indolent Biological Potential
Differentially expressed genes between NKX6-2 overexpressing and control Kras;Gnas cell line.
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessTable S19 from Spatial Transcriptomics of Intraductal Papillary Mucinous Neoplasms of the Pancreas Identifies NKX6-2 as a Driver of Gastric Differentiation and Indolent Biological Potential
Differentially expressed genes between NKX6-2 overexpressing and control Kras;Gnas cell line.
- Open Access
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Table S4 from Spatial Transcriptomics of Intraductal Papillary Mucinous Neoplasms of the Pancreas Identifies NKX6-2 as a Driver of Gastric Differentiation and Indolent Biological Potential
Differentially expressed genes between LG and HGPDAC epilesional spots.
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessTable S4 from Spatial Transcriptomics of Intraductal Papillary Mucinous Neoplasms of the Pancreas Identifies NKX6-2 as a Driver of Gastric Differentiation and Indolent Biological Potential
Differentially expressed genes between LG and HGPDAC epilesional spots.