Mingqun Qin
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Research Interests
Mingqun Qin has a total of 5 co-authors. Here are the top co-authors by number of shared publications:
- Fuqiu Niu
Department of Parasitology, School of Basic Medicine, Guilin Medical University, Guangxi 541004, China
1 shared publication - Riming Wei
School of Intelligent Medicine and Biotechbology, Guilin Medical University, Lingui District, Guilin, People’s Republic of China
1 shared publication - Shenmei Li
School of Stomatology, Guilin Medical University, Guangxi 541004, China
1 shared publication - Shigui Chong
School of Stomatology, Guilin Medical University, Guangxi 541004, China
1 shared publication - Yumin Zhao
Department of Parasitology, School of Basic Medicine, Guilin Medical University, Guangxi 541004, China
1 shared publication
Latest Publications
- Open Access
Mechanism of Fibrosis Induced by Echinococcus spp.
Infection with Echinococcus spp. causes fibrosis in various vital organs, including the liver and lungs. Hepatic fibrosis is a pathological feature of Echinococcus infection that destroys normal liver tissue, leading to jaundice, cholecystitis, portal hypertension, etc. Severe Echinococcus multilocularis infections lead to liver failure and hepatic encephalopathy. The formation of peripheral fiberboards around the metacestode is a major reason as to why antiparasitic drugs fail to be effectively transported to the lesion site. Studies on the mechanism of hepatic fibrosis caused by Echinococcus are important for treatment in patients. Recent studies have focused on miRNA and TGF-β. More recent findings have focused on the generation of collagen fibers around the metacestode. In this review paper we focus on the mechanism by which the Echinococcus parasite induces fibrosis in liver and some other organs in intermediate hosts—animals as well as human beings.
Open AccessMechanism of Fibrosis Induced by Echinococcus spp.
Infection with Echinococcus spp. causes fibrosis in various vital organs, including the liver and lungs. Hepatic fibrosis is a pathological feature of Echinococcus infection that destroys normal liver tissue, leading to jaundice, cholecystitis, portal hypertension, etc. Severe Echinococcus multilocularis infections lead to liver failure and hepatic encephalopathy. The formation of peripheral fiberboards around the metacestode is a major reason as to why antiparasitic drugs fail to be effectively transported to the lesion site. Studies on the mechanism of hepatic fibrosis caused by Echinococcus are important for treatment in patients. Recent studies have focused on miRNA and TGF-β. More recent findings have focused on the generation of collagen fibers around the metacestode. In this review paper we focus on the mechanism by which the Echinococcus parasite induces fibrosis in liver and some other organs in intermediate hosts—animals as well as human beings.