Ke Xu
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Previous Organizations / Institutes
The organizations / institutes from the scholar's other articles in the past ten years
Research Subjects
Ke Xu has a total of 207 co-authors. Here are the top co-authors by number of shared publications:
- Abir Kumar Panda
NIAID/NIH
14 shared publications - Alexander J. Rossi
Department of Surgery, Yale School of Medicine, New Haven, CT 06510, USA
14 shared publications - Allen J. Luna
28School of Medicine, Vanderbilt University, Nashville, Tennessee
14 shared publications - Alok Ranjan
18Department of Pharmacology and Toxicology, Massey Cancer Center, Richmond, Virginia
14 shared publications - Alyssa V. Eade
Surgical Oncology Program, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD
14 shared publications - Amber Leila Sarvestani
21Department of Surgery, University of Missouri Kansas City, Kansas City, Missouri
14 shared publications
Latest Publications
- Open Access
- 3 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S5 from Expression of IMPACT Curtails Metabolic Plasticity and Augments NK Cell Killing to Abrogate Metastatic Growth
TRANSITE prediction of RNA binding proteins whose motifs are overrepresented in the 3’-UTR transcriptomes of GCN1 KD (SH1 and SH2) KLM1 cells.
- 3 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S5 from Expression of IMPACT Curtails Metabolic Plasticity and Augments NK Cell Killing to Abrogate Metastatic Growth
TRANSITE prediction of RNA binding proteins whose motifs are overrepresented in the 3’-UTR transcriptomes of GCN1 KD (SH1 and SH2) KLM1 cells.
- Open Access
- 3 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S4 from Expression of IMPACT Curtails Metabolic Plasticity and Augments NK Cell Killing to Abrogate Metastatic Growth
Figure S4. IMPACT abrogates GCN1-mediated immune cell evasion
- 3 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S4 from Expression of IMPACT Curtails Metabolic Plasticity and Augments NK Cell Killing to Abrogate Metastatic Growth
Figure S4. IMPACT abrogates GCN1-mediated immune cell evasion
- Open Access
- 3 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S1 from Expression of IMPACT Curtails Metabolic Plasticity and Augments NK Cell Killing to Abrogate Metastatic Growth
List of all hits identified in the cDNA screen and categorized by function. cDNAs highlighted in red were identified in the 3 KB library fraction. The number of tumor lesions identifying the cDNA is given.
- 3 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S1 from Expression of IMPACT Curtails Metabolic Plasticity and Augments NK Cell Killing to Abrogate Metastatic Growth
List of all hits identified in the cDNA screen and categorized by function. cDNAs highlighted in red were identified in the 3 KB library fraction. The number of tumor lesions identifying the cDNA is given.
- Open Access
- 3 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S2 from Expression of IMPACT Curtails Metabolic Plasticity and Augments NK Cell Killing to Abrogate Metastatic Growth
Figure S2. GCN1 drives metastatic growth through activation of ATF4
- 3 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S2 from Expression of IMPACT Curtails Metabolic Plasticity and Augments NK Cell Killing to Abrogate Metastatic Growth
Figure S2. GCN1 drives metastatic growth through activation of ATF4
- Open Access
- 3 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S7 from Expression of IMPACT Curtails Metabolic Plasticity and Augments NK Cell Killing to Abrogate Metastatic Growth
Figure S7. Expression of IMPACT is lost through genomic deletion and epigenetic silencing
- 3 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S7 from Expression of IMPACT Curtails Metabolic Plasticity and Augments NK Cell Killing to Abrogate Metastatic Growth
Figure S7. Expression of IMPACT is lost through genomic deletion and epigenetic silencing
- Open Access
- 3 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S3 from Expression of IMPACT Curtails Metabolic Plasticity and Augments NK Cell Killing to Abrogate Metastatic Growth
GCN1 cDNA sequence in 4964-POP and 4964-HOP cells.
- 3 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S3 from Expression of IMPACT Curtails Metabolic Plasticity and Augments NK Cell Killing to Abrogate Metastatic Growth
GCN1 cDNA sequence in 4964-POP and 4964-HOP cells.
- Open Access
- 3 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S3 from Expression of IMPACT Curtails Metabolic Plasticity and Augments NK Cell Killing to Abrogate Metastatic Growth
Figure S3. IMPACT limits ATF4 activation through competitive inhibition of GCN1
- 3 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S3 from Expression of IMPACT Curtails Metabolic Plasticity and Augments NK Cell Killing to Abrogate Metastatic Growth
Figure S3. IMPACT limits ATF4 activation through competitive inhibition of GCN1
- Open Access
- 3 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S6 from Expression of IMPACT Curtails Metabolic Plasticity and Augments NK Cell Killing to Abrogate Metastatic Growth
Extent of IMPACT CpG island promoter methylation in mouse cell lines (KPC, 4964-POP, and 4964-HOP), and five human pancreatic liver metastases compared to three primary PDACs, and two normal pancreas and liver tissue.
- 3 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S6 from Expression of IMPACT Curtails Metabolic Plasticity and Augments NK Cell Killing to Abrogate Metastatic Growth
Extent of IMPACT CpG island promoter methylation in mouse cell lines (KPC, 4964-POP, and 4964-HOP), and five human pancreatic liver metastases compared to three primary PDACs, and two normal pancreas and liver tissue.
- Open Access
- 3 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S5 from Expression of IMPACT Curtails Metabolic Plasticity and Augments NK Cell Killing to Abrogate Metastatic Growth
Figure S5. NK cells eliminate IMPACT-expressing tumor cells through NKG2D recognition
- 3 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S5 from Expression of IMPACT Curtails Metabolic Plasticity and Augments NK Cell Killing to Abrogate Metastatic Growth
Figure S5. NK cells eliminate IMPACT-expressing tumor cells through NKG2D recognition
- Open Access
- 3 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S2 from Expression of IMPACT Curtails Metabolic Plasticity and Augments NK Cell Killing to Abrogate Metastatic Growth
Fold change in antioxidant responsive genes upon GCN1 KD in KPC cells grown at physiologic levels of glutamine
- 3 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S2 from Expression of IMPACT Curtails Metabolic Plasticity and Augments NK Cell Killing to Abrogate Metastatic Growth
Fold change in antioxidant responsive genes upon GCN1 KD in KPC cells grown at physiologic levels of glutamine