Elihu D. Richter
Research Subjects
Research Interests
Elihu D. Richter has a total of 42 co-authors. Here are the top co-authors by number of shared publications:
Tamar Berman
Hebrew University-Hadassah School of Public Health and Community Medicine, Jerusalem, Israel
4 shared publications- Lee S. Friedman
Division of Environmental and Occupational Health Sciences, School of Public Health, University of Illinois Chicago, Chicago, IL, USA
3 shared publications E Ben-Michael
Hebrew University-Hadassah School of Community Medicine and Public Health, Unit of Occupational and Environmental Medicine, Betts Injury Prevention Center, Jerusalem, Israel
2 shared publications- Paul Barach
College of Population Health Thomas Jefferson University Philadelphia Pennsylvania USA
2 shared publications T Berman
2 shared publicationsAbed Abu-Hamda
Kupat Holim Meuhedet, Yerka, Israel
1 shared publication
Latest Publications
- Open Access
- 18 May 2007
- Published by Oxford University Press (OUP) in European Journal of Public Health
- Vol. 18 (2) , 204-209
'Ethnic cleansing' bleaches the atrocities of genocide
Genocide has been the leading cause of preventable violent death in the 20th–21st century, taking even more lives than war. The term ‘ethnic cleansing’ is used as a euphemism for genocide despite it having no legal status. Like ‘Judenrein’ and ‘racial hygiene’ in Nazi medicine, it expropriates pseudo-medical terminology to justify massacre. Use of the term reifies a dehumanized view of the victims as sources of filth and disease, and propagates the reversed social ethics of the perpetrators. Timelines for recent genocides (Bosnia, 1991–1996, 200 000; Kosovo 1998–2000, 10 000–20 000; Rwanda, 1994, 800 000; Darfur 2002–2006, >400 000) show that its use bears no relationship to death tolls or the scale of atrocity. Bystanders’ use of the term ‘ethnic cleansing’ signals the lack of will to stop genocide, resulting in huge increases in deaths, and undermines international legal obligations to acknowledge genocide. The term ‘ethnic cleansing’ corrupts observation, interpretation, ethical judgment and decision-making, thereby undermining the aim of public health. Public health should lead the way in expunging the term ‘ethnic cleansing’ from official use. ‘Ethnic cleansing’ bleaches the atrocities of genocide, leading to inaction in preventing current and future genocides.
- 18 May 2007
- Published by Oxford University Press (OUP) in European Journal of Public Health
- Vol. 18 (2) , 204-209
Open Access'Ethnic cleansing' bleaches the atrocities of genocide
Genocide has been the leading cause of preventable violent death in the 20th–21st century, taking even more lives than war. The term ‘ethnic cleansing’ is used as a euphemism for genocide despite it having no legal status. Like ‘Judenrein’ and ‘racial hygiene’ in Nazi medicine, it expropriates pseudo-medical terminology to justify massacre. Use of the term reifies a dehumanized view of the victims as sources of filth and disease, and propagates the reversed social ethics of the perpetrators. Timelines for recent genocides (Bosnia, 1991–1996, 200 000; Kosovo 1998–2000, 10 000–20 000; Rwanda, 1994, 800 000; Darfur 2002–2006, >400 000) show that its use bears no relationship to death tolls or the scale of atrocity. Bystanders’ use of the term ‘ethnic cleansing’ signals the lack of will to stop genocide, resulting in huge increases in deaths, and undermines international legal obligations to acknowledge genocide. The term ‘ethnic cleansing’ corrupts observation, interpretation, ethical judgment and decision-making, thereby undermining the aim of public health. Public health should lead the way in expunging the term ‘ethnic cleansing’ from official use. ‘Ethnic cleansing’ bleaches the atrocities of genocide, leading to inaction in preventing current and future genocides.
- Open Access
- 1 June 2006
- Published by American Chemical Society (ACS) in Environmental Health Perspectives
- Vol. 114 (6) , 917-922
Childhood Lead Exposure in the Palestinian Authority, Israel, and Jordan: Results from the Middle Eastern Regional Cooperation Project, 1996–2000
In the Middle East, the major sources of lead exposure have been leaded gasoline, lead-contaminated flour from traditional stone mills, focal exposures from small battery plants and smelters, and kohl (blue color) in cosmetics. In 1998–2000, we ...
- 1 June 2006
- Published by American Chemical Society (ACS) in Environmental Health Perspectives
- Vol. 114 (6) , 917-922
Open AccessChildhood Lead Exposure in the Palestinian Authority, Israel, and Jordan: Results from the Middle Eastern Regional Cooperation Project, 1996–2000
In the Middle East, the major sources of lead exposure have been leaded gasoline, lead-contaminated flour from traditional stone mills, focal exposures from small battery plants and smelters, and kohl (blue color) in cosmetics. In 1998–2000, we ...
- 3 April 2006
- Published by BMJ in Injury Prevention
- Vol. 12 (2) , 135.1-135
Resources for terror and road injury prevention
The follow up period (1994–2003) is cut off before the terror attacks in Madrid (191 dead) and London (52 dead), and the …
- 3 April 2006
- Published by BMJ in Injury Prevention
- Vol. 12 (2) , 135.1-135
Resources for terror and road injury prevention
The follow up period (1994–2003) is cut off before the terror attacks in Madrid (191 dead) and London (52 dead), and the …
- 1 April 2006
- Published by Annual Reviews in Annual Review of Public Health
- Vol. 27 (1) , 125-152
SPEED, ROAD INJURY, AND PUBLIC HEALTH
▪ Abstract We review milestones in the history of increases in speed limits and travel speeds (“speed creep”) and risks for road deaths and injury. Reduced speed limits, speed-camera networks, and speed calming substantially reduce these tolls in absolute numbers—a trend that is apparent in the United Kingdom, Australia, France, and other countries, but not in the United States, which has raised speed limits and does not have speed-camera networks. Newtonian relationships between the fourth power of small increases or reductions in speed and large increases or reductions in deaths state the case for speed control. Speed adaptation and the interaction between speed and other determinants of injury risks, including congestion and countermeasures, enter into these relationships. Speed-camera networks and speed calming lead to large, sustainable, and highly cost-effective drops in road deaths and injuries and should target entire populations, not merely high-risk subgroups or situations. Yet, there are major barriers to preventive strategies based on the discovery that speed kills. Modal shifts from speed on roads to speed on rail, lower maximum vehicle speeds, and speed-camera networks are required for progress toward Vision Zero—the goal of no road deaths—through Killing Speed. The human cost of the delay in killing speed in the United States may be as high as 20,000 lives lost per year.
- 1 April 2006
- Published by Annual Reviews in Annual Review of Public Health
- Vol. 27 (1) , 125-152
SPEED, ROAD INJURY, AND PUBLIC HEALTH
▪ Abstract We review milestones in the history of increases in speed limits and travel speeds (“speed creep”) and risks for road deaths and injury. Reduced speed limits, speed-camera networks, and speed calming substantially reduce these tolls in absolute numbers—a trend that is apparent in the United Kingdom, Australia, France, and other countries, but not in the United States, which has raised speed limits and does not have speed-camera networks. Newtonian relationships between the fourth power of small increases or reductions in speed and large increases or reductions in deaths state the case for speed control. Speed adaptation and the interaction between speed and other determinants of injury risks, including congestion and countermeasures, enter into these relationships. Speed-camera networks and speed calming lead to large, sustainable, and highly cost-effective drops in road deaths and injuries and should target entire populations, not merely high-risk subgroups or situations. Yet, there are major barriers to preventive strategies based on the discovery that speed kills. Modal shifts from speed on roads to speed on rail, lower maximum vehicle speeds, and speed-camera networks are required for progress toward Vision Zero—the goal of no road deaths—through Killing Speed. The human cost of the delay in killing speed in the United States may be as high as 20,000 lives lost per year.
- Open Access
- 1 April 2005
- Published by BMJ in Injury Prevention
- Vol. 11 (2) , 110-114
Working conditions and fatigue in professional truck drivers at Israeli ports
Background: Trucks represent 6% of all vehicles, but truck crashes account for 20% of road deaths in Israel, even though travel distances are usually short (Objective: To determine occupational and individual predictors of fatigue, falling asleep at the wheel, and involvement in crashes with injuries and deaths in truck drivers. Setting and methods: We carried out field interviews of 160 port truck drivers regarding driver characteristics, workplace and driving conditions, employer-employee relations, medical conditions, sleep quality and fatigue, falling asleep at the wheel, and involvement in road crashes. Results: One day before interview, 38.1% of the drivers had worked more than the 12 hour legal limit. More than 30% reported falling asleep at the wheel recently, and 13% had prior involvement in a sleep related crash. Sixty seven (41.9%) drivers said that their employer forced them to work beyond the legal 12 hour daily limit. Involvement in a crash with casualties was associated with poor sleep quality (adjusted OR = 2.9; p = 0.042) and frequent difficulty finding parking when tired (OR = 3.7; p = 0.049). Self assessment of fatigue underestimated fatigue from the Pittsburgh Sleep Quality Questionnaire. However fatigue occurred in many drivers without sleep problems and many crashes occurred without fatigue. Conclusions: Prevention requires measures to reduce work stresses, screening drivers, speed control, and modal shifts. The work risks and adverse outcomes of truck drivers in large countries with long overnight journeys occur in a small country with small distances, relatively short work journeys, and little overnight travel.
- 1 April 2005
- Published by BMJ in Injury Prevention
- Vol. 11 (2) , 110-114
Open AccessWorking conditions and fatigue in professional truck drivers at Israeli ports
Background: Trucks represent 6% of all vehicles, but truck crashes account for 20% of road deaths in Israel, even though travel distances are usually short (Objective: To determine occupational and individual predictors of fatigue, falling asleep at the wheel, and involvement in crashes with injuries and deaths in truck drivers. Setting and methods: We carried out field interviews of 160 port truck drivers regarding driver characteristics, workplace and driving conditions, employer-employee relations, medical conditions, sleep quality and fatigue, falling asleep at the wheel, and involvement in road crashes. Results: One day before interview, 38.1% of the drivers had worked more than the 12 hour legal limit. More than 30% reported falling asleep at the wheel recently, and 13% had prior involvement in a sleep related crash. Sixty seven (41.9%) drivers said that their employer forced them to work beyond the legal 12 hour daily limit. Involvement in a crash with casualties was associated with poor sleep quality (adjusted OR = 2.9; p = 0.042) and frequent difficulty finding parking when tired (OR = 3.7; p = 0.049). Self assessment of fatigue underestimated fatigue from the Pittsburgh Sleep Quality Questionnaire. However fatigue occurred in many drivers without sleep problems and many crashes occurred without fatigue. Conclusions: Prevention requires measures to reduce work stresses, screening drivers, speed control, and modal shifts. The work risks and adverse outcomes of truck drivers in large countries with long overnight journeys occur in a small country with small distances, relatively short work journeys, and little overnight travel.
- 1 April 2004
- Published by American Public Health Association in American Journal of Public Health
- Vol. 94 (4) , 568-574
Raised Speed Limits, Speed Spillover, Case-Fatality Rates, and Road Deaths in Israel: A 5-Year Follow-Up
Objectives. We assessed the 5-year, nationwide impact on road deaths of the raise in the speed limit (November 1, 1993) on 3 major interurban highways in Israel from 90 to 100 kph. Methods. We compared before–after trends in deaths as well as case fatality—an outcome independent of exposure (defined as vehicle-kilometers traveled). Results. After the raise, speeds rose by 4.5%–9.1%. Over 5 years, there was a sustained increase in deaths (15%) and case fatality rates (38%) on all interurban roads. Corresponding increases in deaths (13%) and case fatality (24%) on urban roads indicated “speed spillover.” Conclusions. Immediate increases in case fatality predicted and tracked the sustained increase in deaths from increased speeds of impact. Newtonian fourth power models predicted the effects of “small” increases in speed on large rises in case fatality rates. Countermeasures and congestion reduced the impact on deaths and case-fatality rates by more than half.
- 1 April 2004
- Published by American Public Health Association in American Journal of Public Health
- Vol. 94 (4) , 568-574
Raised Speed Limits, Speed Spillover, Case-Fatality Rates, and Road Deaths in Israel: A 5-Year Follow-Up
Objectives. We assessed the 5-year, nationwide impact on road deaths of the raise in the speed limit (November 1, 1993) on 3 major interurban highways in Israel from 90 to 100 kph. Methods. We compared before–after trends in deaths as well as case fatality—an outcome independent of exposure (defined as vehicle-kilometers traveled). Results. After the raise, speeds rose by 4.5%–9.1%. Over 5 years, there was a sustained increase in deaths (15%) and case fatality rates (38%) on all interurban roads. Corresponding increases in deaths (13%) and case fatality (24%) on urban roads indicated “speed spillover.” Conclusions. Immediate increases in case fatality predicted and tracked the sustained increase in deaths from increased speeds of impact. Newtonian fourth power models predicted the effects of “small” increases in speed on large rises in case fatality rates. Countermeasures and congestion reduced the impact on deaths and case-fatality rates by more than half.
- 1 January 2004
- Vol. 17 (1) , 9-16
The Precautionary Principle, epidemiology and the ethics of delay.
Ethics tells us: do good and do no harm and invokes the norms of justice, equity and respect for autonomy in protecting and promoting health and well-being. The Precautionary Principle, a contemporary re-definition of Bradford Hill's case for action, gives us a common sense rule for doing good by preventing harm to public health from delay: when in doubt about the presence of a hazard, there should be no doubt about its prevention or removal. It shifts the burden of proof from showing presence of risk to showing absence of risk, aims to do good by preventing harm, and subsumes the upstream strategies of the Driving Forces Pressure Stress Exposure Effect Action model and downstream strategies from molecular epidemiology for detection and prevention of risk. The Precautionary Principle has emerged because of the ethical import of delays in detection of risks to human health and the environment. Ethical principles, the Precautionary Principle, the DPSEEA model and molecular epidemiology all imply re-emphasizing epidemiology's classic role for early detection and prevention. Delays in recognizing risks from past exposures and acting on the findings (e.g., cigarette smoking and lung cancer, asbestos, organochlorines and endocrine disruption, radiofrequency, raised travel speeds) were examples of failures that were not only scientific, but ethical, since they resulted in preventable harm to exposed populations. These may delay results from, among other things, external and internal determinants of epidemiologic investigations of hazard and risk, including misuse of tests of statistical significance. Furthermore, applying the Precautionary Principle to ensure justice, equity, and respect for autonomy raises questions concerning the short-term costs of implementation to achieve long-term goals and the principles that guide compensation.
- 1 January 2004
- Vol. 17 (1) , 9-16
The Precautionary Principle, epidemiology and the ethics of delay.
Ethics tells us: do good and do no harm and invokes the norms of justice, equity and respect for autonomy in protecting and promoting health and well-being. The Precautionary Principle, a contemporary re-definition of Bradford Hill's case for action, gives us a common sense rule for doing good by preventing harm to public health from delay: when in doubt about the presence of a hazard, there should be no doubt about its prevention or removal. It shifts the burden of proof from showing presence of risk to showing absence of risk, aims to do good by preventing harm, and subsumes the upstream strategies of the Driving Forces Pressure Stress Exposure Effect Action model and downstream strategies from molecular epidemiology for detection and prevention of risk. The Precautionary Principle has emerged because of the ethical import of delays in detection of risks to human health and the environment. Ethical principles, the Precautionary Principle, the DPSEEA model and molecular epidemiology all imply re-emphasizing epidemiology's classic role for early detection and prevention. Delays in recognizing risks from past exposures and acting on the findings (e.g., cigarette smoking and lung cancer, asbestos, organochlorines and endocrine disruption, radiofrequency, raised travel speeds) were examples of failures that were not only scientific, but ethical, since they resulted in preventable harm to exposed populations. These may delay results from, among other things, external and internal determinants of epidemiologic investigations of hazard and risk, including misuse of tests of statistical significance. Furthermore, applying the Precautionary Principle to ensure justice, equity, and respect for autonomy raises questions concerning the short-term costs of implementation to achieve long-term goals and the principles that guide compensation.
- 16 October 2002
- Published by Elsevier in Medical Hypotheses
- Vol. 59 (6) , 703-705
Cellular telephones and effects on the brain: The head as an antenna and brain tissue as a radio receiver
- 16 October 2002
- Published by Elsevier in Medical Hypotheses
- Vol. 59 (6) , 703-705
Cellular telephones and effects on the brain: The head as an antenna and brain tissue as a radio receiver
- 1 July 2002
- Published by Taylor & Francis in Archives of Environmental Health: An International Journal
- Vol. 57 (4) , 270-272
Brain Cancer with Induction Periods of Less Than 10 Years in Young Military Radar Workers
The authors have reported on 5 young patients who had brain tumors that appeared within 10 yr of initial occupational exposures to radar. Four of the patients were less than 30 yr of age when the diagnoses were initially made. Brief induction periods that follow high exposures in individual sentinel patients are a recognized indicator of impending group risk, and these periods call attention to the need for precautionary measures. Similarly, reports of short induction periods for brain cancer on the side of the head in which there has been prior use of cell phones may also indicate increased risk.
- 1 July 2002
- Published by Taylor & Francis in Archives of Environmental Health: An International Journal
- Vol. 57 (4) , 270-272
Brain Cancer with Induction Periods of Less Than 10 Years in Young Military Radar Workers
The authors have reported on 5 young patients who had brain tumors that appeared within 10 yr of initial occupational exposures to radar. Four of the patients were less than 30 yr of age when the diagnoses were initially made. Brief induction periods that follow high exposures in individual sentinel patients are a recognized indicator of impending group risk, and these periods call attention to the need for precautionary measures. Similarly, reports of short induction periods for brain cancer on the side of the head in which there has been prior use of cell phones may also indicate increased risk.
- Open Access
- 1 September 2001
- Published by BMJ in Injury Prevention
- Vol. 7 (3) , 176-178
Death and injury from motor vehicle crashes: a public health failure, not an achievement: Figure 1
- 1 September 2001
- Published by BMJ in Injury Prevention
- Vol. 7 (3) , 176-178
Open AccessDeath and injury from motor vehicle crashes: a public health failure, not an achievement: Figure 1