SLShao-Ting Li
Shao-Ting Li
*This is an algorithmically generated author record based on publicly available information.
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Research Interests
Shao-Ting Li has a total of 8 co-authors. Here are the top co-authors by number of shared publications:
- Cheng-Heng Kao
Center of General Education, Chang Gung University, Taoyuan, Taiwan
2 shared publications - Liang-Tung Yang
Institute of Cellular and System Medicine, National Health Research Institutes, Zhunan, Taiwan; Graduate Institute of Biomedical Sciences, China Medical University, Taichung, Taiwan
2 shared publications - Chien-Hui Hong
Kaohsiung Veterans General Hospital
1 shared publication - Chih-Hung Lee
Department of Dermatology, Kaohsiung Chang Gung Memorial Hospital and Chang Gung University College of Medicine
1 shared publication - Li-Ying Huang
Institute of Cellular and System Medicine, National Health Research Institutes, Zhunan, Taiwan
1 shared publication - Ming-Kai Yang
Institute of Cellular and System Medicine, National Health Research Institutes, Zhunan, Taiwan, R.O.C.
1 shared publication
Latest Publications
- 23 March 2023
- Published by Elsevier in Journal of Investigative Dermatology
- Vol. 143 (9) , 1735-1745.e11
Gasdermin A Is Required for Epidermal Cornification during Skin Barrier Regeneration and in an Atopic Dermatitis-Like Model
- 23 March 2023
- Published by Elsevier in Journal of Investigative Dermatology
- Vol. 143 (9) , 1735-1745.e11
Gasdermin A Is Required for Epidermal Cornification during Skin Barrier Regeneration and in an Atopic Dermatitis-Like Model
- 1 November 2020
- Published by Elsevier in Journal of Investigative Dermatology
- Vol. 140 (11) , 2117-2128
Gasdermin A3–Mediated Cell Death Causes Niche Collapse and Precocious Activation of Hair Follicle Stem Cells
- 1 November 2020
- Published by Elsevier in Journal of Investigative Dermatology
- Vol. 140 (11) , 2117-2128
Gasdermin A3–Mediated Cell Death Causes Niche Collapse and Precocious Activation of Hair Follicle Stem Cells