Leora Witkowski
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Previous Organizations / Institutes
The organizations / institutes from the scholar's other articles in the past ten years
Leora Witkowski has a total of 127 co-authors. Here are the top co-authors by number of shared publications:
- William D. Foulkes
Department of Human Genetics McGill University Montreal QC Canada
23 shared publications - Morag Park
Department of Biochemistry, McGill University, Montreal, Canada
20 shared publications - Sidong Huang
Department of Biochemistry, McGill University, Montreal, Canada
20 shared publications - Yibo Xue
Department of Biochemistry, McGill University, Montreal, Canada
20 shared publications - W. Glenn McCluggage
Department of Pathology, Belfast Health and Social Care Trust, Belfast, UK
19 shared publications - Alexander James Roy Bishop
Greehey Children’s Cancer Research Institute, University of Texas Health at San Antonio, San Antonio, USA
18 shared publications
Latest Publications
- Open Access
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Figure S10 from SMARCA4 Loss Increases RNA Polymerase II Pausing and Elevates R-Loops to Inhibit BRCA1-Mediated Repair in Ovarian Cancer
Characterization of the pathogenic variants of SMARCA4 retaining protein expression
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S10 from SMARCA4 Loss Increases RNA Polymerase II Pausing and Elevates R-Loops to Inhibit BRCA1-Mediated Repair in Ovarian Cancer
Characterization of the pathogenic variants of SMARCA4 retaining protein expression
- Open Access
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Table S1 from SMARCA4 Loss Increases RNA Polymerase II Pausing and Elevates R-Loops to Inhibit BRCA1-Mediated Repair in Ovarian Cancer
Top 31 compounds identified from the FDA drug screen in BIN-67 cells.
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Open AccessTable S1 from SMARCA4 Loss Increases RNA Polymerase II Pausing and Elevates R-Loops to Inhibit BRCA1-Mediated Repair in Ovarian Cancer
Top 31 compounds identified from the FDA drug screen in BIN-67 cells.
- Open Access
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Figure S6 from SMARCA4 Loss Increases RNA Polymerase II Pausing and Elevates R-Loops to Inhibit BRCA1-Mediated Repair in Ovarian Cancer
SMARCA4 loss results in R-Loop accumulation
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S6 from SMARCA4 Loss Increases RNA Polymerase II Pausing and Elevates R-Loops to Inhibit BRCA1-Mediated Repair in Ovarian Cancer
SMARCA4 loss results in R-Loop accumulation
- Open Access
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Figure S11 from SMARCA4 Loss Increases RNA Polymerase II Pausing and Elevates R-Loops to Inhibit BRCA1-Mediated Repair in Ovarian Cancer
BioID proximity-labeling identifies the association between SMARCA4 and PAF1C
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S11 from SMARCA4 Loss Increases RNA Polymerase II Pausing and Elevates R-Loops to Inhibit BRCA1-Mediated Repair in Ovarian Cancer
BioID proximity-labeling identifies the association between SMARCA4 and PAF1C
- Open Access
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Table S3 from SMARCA4 Loss Increases RNA Polymerase II Pausing and Elevates R-Loops to Inhibit BRCA1-Mediated Repair in Ovarian Cancer
TSS bound by Pol II and TSS-associated mRNAs significantly regulated by SMARCA4.
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Open AccessTable S3 from SMARCA4 Loss Increases RNA Polymerase II Pausing and Elevates R-Loops to Inhibit BRCA1-Mediated Repair in Ovarian Cancer
TSS bound by Pol II and TSS-associated mRNAs significantly regulated by SMARCA4.
- Open Access
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Figure S9 from SMARCA4 Loss Increases RNA Polymerase II Pausing and Elevates R-Loops to Inhibit BRCA1-Mediated Repair in Ovarian Cancer
Removal of R-Loops in SCCOHT cells confers resistance to PARP inhibitors
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S9 from SMARCA4 Loss Increases RNA Polymerase II Pausing and Elevates R-Loops to Inhibit BRCA1-Mediated Repair in Ovarian Cancer
Removal of R-Loops in SCCOHT cells confers resistance to PARP inhibitors
- Open Access
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Figure S5 from SMARCA4 Loss Increases RNA Polymerase II Pausing and Elevates R-Loops to Inhibit BRCA1-Mediated Repair in Ovarian Cancer
SMARCA4-deficient ovarian cancer cells depend on BRCA1 for their survival
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S5 from SMARCA4 Loss Increases RNA Polymerase II Pausing and Elevates R-Loops to Inhibit BRCA1-Mediated Repair in Ovarian Cancer
SMARCA4-deficient ovarian cancer cells depend on BRCA1 for their survival
- Open Access
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Figure S4 from SMARCA4 Loss Increases RNA Polymerase II Pausing and Elevates R-Loops to Inhibit BRCA1-Mediated Repair in Ovarian Cancer
SCCOHT cells display homologous recombination repair impairment
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S4 from SMARCA4 Loss Increases RNA Polymerase II Pausing and Elevates R-Loops to Inhibit BRCA1-Mediated Repair in Ovarian Cancer
SCCOHT cells display homologous recombination repair impairment
- Open Access
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Figure S1 from SMARCA4 Loss Increases RNA Polymerase II Pausing and Elevates R-Loops to Inhibit BRCA1-Mediated Repair in Ovarian Cancer
Differential drug responses of SCCOHT cells and ovarian controls.
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S1 from SMARCA4 Loss Increases RNA Polymerase II Pausing and Elevates R-Loops to Inhibit BRCA1-Mediated Repair in Ovarian Cancer
Differential drug responses of SCCOHT cells and ovarian controls.
- Open Access
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Figure S2 from SMARCA4 Loss Increases RNA Polymerase II Pausing and Elevates R-Loops to Inhibit BRCA1-Mediated Repair in Ovarian Cancer
SMARCA4 modulates response to PARP inhibitors
- 15 August 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S2 from SMARCA4 Loss Increases RNA Polymerase II Pausing and Elevates R-Loops to Inhibit BRCA1-Mediated Repair in Ovarian Cancer
SMARCA4 modulates response to PARP inhibitors