Lishan Fang
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Previous Organizations / Institutes
The organizations / institutes from the scholar's other articles in the past ten years
Lishan Fang has a total of 49 co-authors. Here are the top co-authors by number of shared publications:
- Riqing Li
Shenzhen Inspection and Testing Center of Agricultural Product Quality and Safety, Shenzhen, China
41 shared publications - Hui Gong
Department of Endocrinology and Metabolic Diseases and Medical Research Center, The Eighth Affiliated Hospital, Sun Yat-Sun University, Shenzhen, China
39 shared publications - Jiahao Hu
Department of Endocrinology and Metabolic Diseases and Medical Research Center, The Eighth Affiliated Hospital, Sun Yat-Sun University, Shenzhen, China
39 shared publications - Libing Song
Department of Experimental Research, State Key Laboratory of Oncology in South China, Collaborative Innovation Center for Cancer Medicine, Sun Yat-sen University Cancer Center, Guangzhou, 510060, China
39 shared publications - Peihang Li
Department of Endocrinology and Metabolic Diseases and Medical Research Center, The Eighth Affiliated Hospital, Sun Yat-Sun University, Shenzhen, China
39 shared publications - Yifan Li
Laboratory Medicine Center, Shenzhen Nanshan People’s Hospital, Shenzhen, China
39 shared publications
Latest Publications
- Open Access
- 28 November 2025
- Published by American Association for Cancer Research (AACR)
Figure S5 from PRODH2-Mediated Metabolism in the Bone Microenvironment Promotes Breast Cancer Metastasis
Figure S5 shows Hyp metabolism regulates glutathione metabolism and ferroptosis through Hyp-mediated iron accumulation
- 28 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S5 from PRODH2-Mediated Metabolism in the Bone Microenvironment Promotes Breast Cancer Metastasis
Figure S5 shows Hyp metabolism regulates glutathione metabolism and ferroptosis through Hyp-mediated iron accumulation
- Open Access
- 28 November 2025
- Published by American Association for Cancer Research (AACR)
Figure S3 from PRODH2-Mediated Metabolism in the Bone Microenvironment Promotes Breast Cancer Metastasis
Figure S3 shows effects of cell death inhibitors on breast cancer bone metastasis
- 28 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S3 from PRODH2-Mediated Metabolism in the Bone Microenvironment Promotes Breast Cancer Metastasis
Figure S3 shows effects of cell death inhibitors on breast cancer bone metastasis
- Open Access
- 28 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S1 from PRODH2-Mediated Metabolism in the Bone Microenvironment Promotes Breast Cancer Metastasis
Supplementary Table S1 presents the baseline characteristics of the breast cancer patients included in the analysis.
- 28 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S1 from PRODH2-Mediated Metabolism in the Bone Microenvironment Promotes Breast Cancer Metastasis
Supplementary Table S1 presents the baseline characteristics of the breast cancer patients included in the analysis.
- Open Access
- 28 November 2025
- Published by American Association for Cancer Research (AACR)
Figure S1 from PRODH2-Mediated Metabolism in the Bone Microenvironment Promotes Breast Cancer Metastasis
Figure S1 shows Hyp metabolism regulates tumor cell adhesion and Hyp metabolism
- 28 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S1 from PRODH2-Mediated Metabolism in the Bone Microenvironment Promotes Breast Cancer Metastasis
Figure S1 shows Hyp metabolism regulates tumor cell adhesion and Hyp metabolism
- Open Access
- 28 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S3 from PRODH2-Mediated Metabolism in the Bone Microenvironment Promotes Breast Cancer Metastasis
Supplementary Table S3 lists the primer sequences used in this study.
- 28 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S3 from PRODH2-Mediated Metabolism in the Bone Microenvironment Promotes Breast Cancer Metastasis
Supplementary Table S3 lists the primer sequences used in this study.
- Open Access
- 28 November 2025
- Published by American Association for Cancer Research (AACR)
Figure S7 from PRODH2-Mediated Metabolism in the Bone Microenvironment Promotes Breast Cancer Metastasis
Figure S7 shows hydroxyproline metabolism mediates metabolic regulation modulates YY1 acetylation and downstream signaling
- 28 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S7 from PRODH2-Mediated Metabolism in the Bone Microenvironment Promotes Breast Cancer Metastasis
Figure S7 shows hydroxyproline metabolism mediates metabolic regulation modulates YY1 acetylation and downstream signaling
- Open Access
- 28 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S2 from PRODH2-Mediated Metabolism in the Bone Microenvironment Promotes Breast Cancer Metastasis
Supplementary Table S2 shows the correlation between PRODH2 expression and clinicopathological characteristics in breast cancer patients.
- 28 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S2 from PRODH2-Mediated Metabolism in the Bone Microenvironment Promotes Breast Cancer Metastasis
Supplementary Table S2 shows the correlation between PRODH2 expression and clinicopathological characteristics in breast cancer patients.
- Open Access
- 28 November 2025
- Published by American Association for Cancer Research (AACR)
Figure S6 from PRODH2-Mediated Metabolism in the Bone Microenvironment Promotes Breast Cancer Metastasis
Figure S6 shows co-culture system regulates IL-8 secretion and SLC7A11 expression
- 28 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S6 from PRODH2-Mediated Metabolism in the Bone Microenvironment Promotes Breast Cancer Metastasis
Figure S6 shows co-culture system regulates IL-8 secretion and SLC7A11 expression
- Open Access
- 28 November 2025
- Published by American Association for Cancer Research (AACR)
Figure S4 from PRODH2-Mediated Metabolism in the Bone Microenvironment Promotes Breast Cancer Metastasis
Figure S4 shows Collagen-mediated tumor-osteoclast crosstalk regulates ferroptosis sensitivity
- 28 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S4 from PRODH2-Mediated Metabolism in the Bone Microenvironment Promotes Breast Cancer Metastasis
Figure S4 shows Collagen-mediated tumor-osteoclast crosstalk regulates ferroptosis sensitivity
- Open Access
- 28 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S4 from PRODH2-Mediated Metabolism in the Bone Microenvironment Promotes Breast Cancer Metastasis
Supplementary Table S4 shows the incidence of bone metastasis (number of mice with metastasis / total mice).
- 28 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S4 from PRODH2-Mediated Metabolism in the Bone Microenvironment Promotes Breast Cancer Metastasis
Supplementary Table S4 shows the incidence of bone metastasis (number of mice with metastasis / total mice).