Diemmy Nguyen
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Previous Organizations / Institutes
The organizations / institutes from the scholar's other articles in the past ten years
Research Subjects
Diemmy Nguyen has a total of 59 co-authors. Here are the top co-authors by number of shared publications:
- Michael William Knitz
Washington University in St. Louis 4 Department of Radiation Oncology Saint Louis, Missouri
60 shared publications Benjamin Van Court
Department of Radiation Oncology, University of Colorado Denver
31 shared publicationsJacob Gadwa
Department of Radiation Oncology, University of Colorado Denver, Anschutz Medical Campus, Aurora, USA
31 shared publications- Sana D. Karam
Department of Radiation Oncology, University of Colorado Denver at Anschutz Medical Campus, Aurora, USA
31 shared publications Shilpa Bhatia
Department of Radiation Oncology, University of Colorado Denver
31 shared publications- Anthony J. Saviola
Department of Biochemistry and Molecular Genetics, School of Medicine, University of Colorado, Aurora, USA
30 shared publications
Latest Publications
- Open Access
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Supplemental Figure 11 from EphA4 Mediates Protumoral and Antitumoral Effects through Upstream Negative Regulation of AKT in Head and Neck Squamous Cell Carcinoma
Supplemental Figure 11. AKT Pharmacological Inhibition Reestablishes the Immunosuppressive Phenotype for EphA4 Knockout Tregs.
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplemental Figure 11 from EphA4 Mediates Protumoral and Antitumoral Effects through Upstream Negative Regulation of AKT in Head and Neck Squamous Cell Carcinoma
Supplemental Figure 11. AKT Pharmacological Inhibition Reestablishes the Immunosuppressive Phenotype for EphA4 Knockout Tregs.
- Open Access
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Supplemental Figure 12 from EphA4 Mediates Protumoral and Antitumoral Effects through Upstream Negative Regulation of AKT in Head and Neck Squamous Cell Carcinoma
Supplemental Figure 12. Gating Schematic for CD8+ T cells in CD8aCreEphA4fl/fl Mice.
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplemental Figure 12 from EphA4 Mediates Protumoral and Antitumoral Effects through Upstream Negative Regulation of AKT in Head and Neck Squamous Cell Carcinoma
Supplemental Figure 12. Gating Schematic for CD8+ T cells in CD8aCreEphA4fl/fl Mice.
- Open Access
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Supplemental Figure 13 from EphA4 Mediates Protumoral and Antitumoral Effects through Upstream Negative Regulation of AKT in Head and Neck Squamous Cell Carcinoma
Supplemental Figure 13. Flow Cytometric Gating for CD8+ T cells Treated with MK-2206.
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplemental Figure 13 from EphA4 Mediates Protumoral and Antitumoral Effects through Upstream Negative Regulation of AKT in Head and Neck Squamous Cell Carcinoma
Supplemental Figure 13. Flow Cytometric Gating for CD8+ T cells Treated with MK-2206.
- Open Access
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Supplemental Figure 14 from EphA4 Mediates Protumoral and Antitumoral Effects through Upstream Negative Regulation of AKT in Head and Neck Squamous Cell Carcinoma
Supplemental Figure 14. Gating Schematic for TAMs and T cells in LysMCreEphA4fl/fl Mice.
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplemental Figure 14 from EphA4 Mediates Protumoral and Antitumoral Effects through Upstream Negative Regulation of AKT in Head and Neck Squamous Cell Carcinoma
Supplemental Figure 14. Gating Schematic for TAMs and T cells in LysMCreEphA4fl/fl Mice.
- Open Access
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Supplemental Figure 18 from EphA4 Mediates Protumoral and Antitumoral Effects through Upstream Negative Regulation of AKT in Head and Neck Squamous Cell Carcinoma
Supplemental Figure 18. EphA4 Inhibition Does Not Affect Cancer Cell Invasion or Migration In Vitro.
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplemental Figure 18 from EphA4 Mediates Protumoral and Antitumoral Effects through Upstream Negative Regulation of AKT in Head and Neck Squamous Cell Carcinoma
Supplemental Figure 18. EphA4 Inhibition Does Not Affect Cancer Cell Invasion or Migration In Vitro.
- Open Access
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Supplemental Figure 17 from EphA4 Mediates Protumoral and Antitumoral Effects through Upstream Negative Regulation of AKT in Head and Neck Squamous Cell Carcinoma
Supplemental Figure 17. Macrophage Polarization Assay Gating.
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplemental Figure 17 from EphA4 Mediates Protumoral and Antitumoral Effects through Upstream Negative Regulation of AKT in Head and Neck Squamous Cell Carcinoma
Supplemental Figure 17. Macrophage Polarization Assay Gating.
- Open Access
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Supplemental Figure 15 from EphA4 Mediates Protumoral and Antitumoral Effects through Upstream Negative Regulation of AKT in Head and Neck Squamous Cell Carcinoma
Supplemental Figure 15. EphA4 Knockout BMDMs are Unable to Polarize into M1 Macrophages.
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplemental Figure 15 from EphA4 Mediates Protumoral and Antitumoral Effects through Upstream Negative Regulation of AKT in Head and Neck Squamous Cell Carcinoma
Supplemental Figure 15. EphA4 Knockout BMDMs are Unable to Polarize into M1 Macrophages.
- Open Access
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Supplemental Figure 16 from EphA4 Mediates Protumoral and Antitumoral Effects through Upstream Negative Regulation of AKT in Head and Neck Squamous Cell Carcinoma
Supplemental Figure 16. EphA4 Pharmacological Inhibition Continues to Limit Tumor Growth During EphA4 Genetic Knockout in Myeloid Cells.
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplemental Figure 16 from EphA4 Mediates Protumoral and Antitumoral Effects through Upstream Negative Regulation of AKT in Head and Neck Squamous Cell Carcinoma
Supplemental Figure 16. EphA4 Pharmacological Inhibition Continues to Limit Tumor Growth During EphA4 Genetic Knockout in Myeloid Cells.
- Open Access
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Supplemental Figure 21 from EphA4 Mediates Protumoral and Antitumoral Effects through Upstream Negative Regulation of AKT in Head and Neck Squamous Cell Carcinoma
Supplemental Figure 21. MOC2 EphA4 KO Cells Proteomic Analysis Reflects an Upregulation of Pathways Related to Cellular Proliferation.
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplemental Figure 21 from EphA4 Mediates Protumoral and Antitumoral Effects through Upstream Negative Regulation of AKT in Head and Neck Squamous Cell Carcinoma
Supplemental Figure 21. MOC2 EphA4 KO Cells Proteomic Analysis Reflects an Upregulation of Pathways Related to Cellular Proliferation.
- Open Access
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Supplemental Figure 19 from EphA4 Mediates Protumoral and Antitumoral Effects through Upstream Negative Regulation of AKT in Head and Neck Squamous Cell Carcinoma
Supplemental Figure 19. EphA4 Knockout in HNSCC Cells Promotes Upregulated Cellular Proliferation Pathways.
- 3 November 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplemental Figure 19 from EphA4 Mediates Protumoral and Antitumoral Effects through Upstream Negative Regulation of AKT in Head and Neck Squamous Cell Carcinoma
Supplemental Figure 19. EphA4 Knockout in HNSCC Cells Promotes Upregulated Cellular Proliferation Pathways.