Manoswini Dash
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Previous Organizations / Institutes
The organizations / institutes from the scholar's other articles in the past ten years
Research Subjects
Research Interests
Manoswini Dash has a total of 9 co-authors. Here are the top co-authors by number of shared publications:
- Abhinav Sinha
ICMR-National Institute of Malaria Research, New Delhi, India
2 shared publications - Aparna Tiwari
ICMR-National Institute of Malaria Research, New Delhi, India
2 shared publications - Auley De
New Delhi, India
1 shared publication - Kristan Alexander Schneider
Department of Internal Medicine, School of Medicine, University of New Mexico Health Sciences Center, 1 University of New Mexico, MSC10 5550, Albuquerque, NM, 87131, United States, 1 505-272-9709
1 shared publication - Lisa Cartwright Ranford-Cartwright
University of Glasgow
1 shared publication - Lokesh Kori
ICMR-National Institute of Malaria Research, New Delhi, India
1 shared publication
Latest Publications
- 19 October 2024
- Published by Elsevier in Trends in Parasitology
- Vol. 40 (11) , 959-961
Microhaplotypes: the QR-code for Plasmodium vivax infection dynamics
- 19 October 2024
- Published by Elsevier in Trends in Parasitology
- Vol. 40 (11) , 959-961
Microhaplotypes: the QR-code for Plasmodium vivax infection dynamics
- 16 March 2023
- Published by Elsevier in Trends in Parasitology
- Vol. 39 (5) , 307-313
India-EMBO Lecture Course: understanding malaria from molecular epidemiology, population genetics, and evolutionary perspectives
- 16 March 2023
- Published by Elsevier in Trends in Parasitology
- Vol. 39 (5) , 307-313
India-EMBO Lecture Course: understanding malaria from molecular epidemiology, population genetics, and evolutionary perspectives
- Open Access
- 1 October 2021
- Published by The Royal Society in Open Biology
- Vol. 11 (10) , 210213
Malaria, COVID-19 and angiotensin-converting enzyme 2: what does the available population data say?
The etiopathogenesis of COVID-19 and its differential geographic spread suggest some populations are apparently ‘less affected’ through many host-related factors that involve angiotensin-converting enzyme 2 (ACE2) protein, which is also the entry receptor for SARS-CoV-2. The role of ACE2 has been well studied in COVID-19 but not in the context of malaria and COVID-19. We have previously suggested how malaria might intersect with COVID-19 through ACE2 mutation and here we evaluate the currently available data that could provide a link between the two diseases. Based on the existing global and Indian data on malaria, COVID-19 and the suggested ACE2 mutation, the association could not be examined robustly, neither accepting nor refuting the suggested hypothesis. We strongly recommend targeted evaluation of this hypothesis through carefully designed robust molecular epidemiological studies.
- 1 October 2021
- Published by The Royal Society in Open Biology
- Vol. 11 (10) , 210213
Open AccessMalaria, COVID-19 and angiotensin-converting enzyme 2: what does the available population data say?
The etiopathogenesis of COVID-19 and its differential geographic spread suggest some populations are apparently ‘less affected’ through many host-related factors that involve angiotensin-converting enzyme 2 (ACE2) protein, which is also the entry receptor for SARS-CoV-2. The role of ACE2 has been well studied in COVID-19 but not in the context of malaria and COVID-19. We have previously suggested how malaria might intersect with COVID-19 through ACE2 mutation and here we evaluate the currently available data that could provide a link between the two diseases. Based on the existing global and Indian data on malaria, COVID-19 and the suggested ACE2 mutation, the association could not be examined robustly, neither accepting nor refuting the suggested hypothesis. We strongly recommend targeted evaluation of this hypothesis through carefully designed robust molecular epidemiological studies.