Miguel G. Silva

Technical University of Munich

*This is an algorithmically generated author record based on publicly available information.

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The organizations/institutes from the scholar's latest article

Technical University of Munich

Previous Organizations / Institutes

The organizations / institutes from the scholar's other articles in the past ten years

TUM Klinikum

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Research Interests

Miguel G. Silva has a total of 63 co-authors. Here are the top co-authors by number of shared publications:

Markus Tschurtschenthaler
Center for Translational Cancer Research (TranslaTUM), Klinikum rechts der Isar, School of Medicine and Health, Technical University of Munich, Munich, Germany
26 shared publications
Moritz Jesinghaus
Institute of Pathology, Philipps-Universität Marburg, Marburg, Germany
26 shared publications
Dieter Saur
Department of Internal Medicine II, Klinikum rechts der Isar, Technische Universität München, Munich, Germany
25 shared publications
Frederick Klauschen
Institute of Pathology, Ludwig Maximilians University Hospital Munich, Munich, Germany
24 shared publications
Heiko Hermeking
Experimental and Molecular Pathology, Institute of Pathology, Ludwig-Maximilians-University München, Thalkirchnerstrasse 59, Munich, D-80337, Germany.
24 shared publications
Jens Neumann
Institute of Pathology, Ludwig-Maximilians-Universität, Medical Faculty, Munich, Germany; Universitätsinstitut für Pathologie, Paracelsus Medizinische Universität, Klinikum Nürnberg, Nürnberg, Germany
24 shared publications

Latest Publications

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Open Access
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- 24 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary table S3 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
by Leon P. Loevenich, Markus Tschurtschenthaler, Matjaz Rokavec, Miguel G. Silva, Moritz Jesinghaus, Thomas Kirchner, Frederick Klauschen, Dieter Saur, Jens Neumann, Heiko Hermeking, et al.
Upregulated genes in SMAD4 mutant tumor organoid lines and DepMAP Project dependency scores.
Website
Google Scholar
- 24 November 2025
- Published by American Association for Cancer Research (AACR)
Open Access
Supplementary table S3 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
by Leon P. Loevenich, Markus Tschurtschenthaler, Matjaz Rokavec, Miguel G. Silva, Moritz Jesinghaus, Thomas Kirchner, Frederick Klauschen, Dieter Saur, Jens Neumann, Heiko Hermeking, et al.
Upregulated genes in SMAD4 mutant tumor organoid lines and DepMAP Project dependency scores.
Open Access
Website
Google Scholar
- 24 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary table S1 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
by Leon P. Loevenich, Markus Tschurtschenthaler, Matjaz Rokavec, Miguel G. Silva, Moritz Jesinghaus, Thomas Kirchner, Frederick Klauschen, Dieter Saur, Jens Neumann, Heiko Hermeking, et al.
Characteristics of tumor organoid lines used for CRISPR/Cas9-mediated SMAD4 deletion.
Website
Google Scholar
- 24 November 2025
- Published by American Association for Cancer Research (AACR)
Open Access
Supplementary table S1 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
by Leon P. Loevenich, Markus Tschurtschenthaler, Matjaz Rokavec, Miguel G. Silva, Moritz Jesinghaus, Thomas Kirchner, Frederick Klauschen, Dieter Saur, Jens Neumann, Heiko Hermeking, et al.
Characteristics of tumor organoid lines used for CRISPR/Cas9-mediated SMAD4 deletion.
Open Access
Website
Google Scholar
- 24 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Data from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
by Leon P. Loevenich, Markus Tschurtschenthaler, Matjaz Rokavec, Miguel G. Silva, Moritz Jesinghaus, Thomas Kirchner, Frederick Klauschen, Dieter Saur, Jens Neumann, Heiko Hermeking, et al.
Supplementary Figures S1-5 S1: Effect of SMAD4 loss on intestinal stem cell, c-MYC, and NLE1 gene expression in CRC organoids S2: Effects of NLE1 ablation on growth and clonogenicity of CRC cell lines S3: Effects of NLE1 ablation on growth and clonogenicity of CRC PDTOs S4: Effects of NLE1 over-expression in human colonic epithelial cells and CRC organoids S5: NLE1 loss-mediated cell cycle arrest and apoptosis in HCT116 CRC cells and HCEC-1CT normal human colonic epithelial cells
Website
Google Scholar
- 24 November 2025
- Published by American Association for Cancer Research (AACR)
Open Access
Supplementary Data from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
by Leon P. Loevenich, Markus Tschurtschenthaler, Matjaz Rokavec, Miguel G. Silva, Moritz Jesinghaus, Thomas Kirchner, Frederick Klauschen, Dieter Saur, Jens Neumann, Heiko Hermeking, et al.
Supplementary Figures S1-5 S1: Effect of SMAD4 loss on intestinal stem cell, c-MYC, and NLE1 gene expression in CRC organoids S2: Effects of NLE1 ablation on growth and clonogenicity of CRC cell lines S3: Effects of NLE1 ablation on growth and clonogenicity of CRC PDTOs S4: Effects of NLE1 over-expression in human colonic epithelial cells and CRC organoids S5: NLE1 loss-mediated cell cycle arrest and apoptosis in HCT116 CRC cells and HCEC-1CT normal human colonic epithelial cells
Open Access
Website
Google Scholar
- 24 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Data from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
by Leon P. Loevenich, Markus Tschurtschenthaler, Matjaz Rokavec, Miguel G. Silva, Moritz Jesinghaus, Thomas Kirchner, Frederick Klauschen, Dieter Saur, Jens Neumann, Heiko Hermeking, et al.
Supplementary Materials Oligonucleotides, qRT-PCR primer pairs, and Taqman probe assays used in this study.
Website
Google Scholar
- 24 November 2025
- Published by American Association for Cancer Research (AACR)
Open Access
Supplementary Data from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
by Leon P. Loevenich, Markus Tschurtschenthaler, Matjaz Rokavec, Miguel G. Silva, Moritz Jesinghaus, Thomas Kirchner, Frederick Klauschen, Dieter Saur, Jens Neumann, Heiko Hermeking, et al.
Supplementary Materials Oligonucleotides, qRT-PCR primer pairs, and Taqman probe assays used in this study.
Open Access
Website
Google Scholar
- 24 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary table S2 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
by Leon P. Loevenich, Markus Tschurtschenthaler, Matjaz Rokavec, Miguel G. Silva, Moritz Jesinghaus, Thomas Kirchner, Frederick Klauschen, Dieter Saur, Jens Neumann, Heiko Hermeking, et al.
Website
Google Scholar
- 24 November 2025
- Published by American Association for Cancer Research (AACR)
Open Access
Supplementary table S2 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
by Leon P. Loevenich, Markus Tschurtschenthaler, Matjaz Rokavec, Miguel G. Silva, Moritz Jesinghaus, Thomas Kirchner, Frederick Klauschen, Dieter Saur, Jens Neumann, Heiko Hermeking, et al.
Open Access
Website
Google Scholar
- 24 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary video file from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
by Leon P. Loevenich, Markus Tschurtschenthaler, Matjaz Rokavec, Miguel G. Silva, Moritz Jesinghaus, Thomas Kirchner, Frederick Klauschen, Dieter Saur, Jens Neumann, Heiko Hermeking, et al.
Endoscopy-guided orthotopic injection of colorectal cancer organoids into the colonic wall of immune-deficient mice.
Website
Google Scholar
- 24 November 2025
- Published by American Association for Cancer Research (AACR)
Open Access
Supplementary video file from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
by Leon P. Loevenich, Markus Tschurtschenthaler, Matjaz Rokavec, Miguel G. Silva, Moritz Jesinghaus, Thomas Kirchner, Frederick Klauschen, Dieter Saur, Jens Neumann, Heiko Hermeking, et al.
Endoscopy-guided orthotopic injection of colorectal cancer organoids into the colonic wall of immune-deficient mice.
Open Access
Website
Google Scholar
- 24 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary table S4 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
by Leon P. Loevenich, Markus Tschurtschenthaler, Matjaz Rokavec, Miguel G. Silva, Moritz Jesinghaus, Thomas Kirchner, Frederick Klauschen, Dieter Saur, Jens Neumann, Heiko Hermeking, et al.
Differentially expressed genes in PDTOS4 versus PDTO cells in the presence of recombinant TGF-beta1
Website
Google Scholar
- 24 November 2025
- Published by American Association for Cancer Research (AACR)
Open Access
Supplementary table S4 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
by Leon P. Loevenich, Markus Tschurtschenthaler, Matjaz Rokavec, Miguel G. Silva, Moritz Jesinghaus, Thomas Kirchner, Frederick Klauschen, Dieter Saur, Jens Neumann, Heiko Hermeking, et al.
Differentially expressed genes in PDTOS4 versus PDTO cells in the presence of recombinant TGF-beta1
Open Access
Website
Google Scholar
- 31 March 2023
- Published by American Association for Cancer Research (AACR)
Supplementary table S2 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
by Leon P. Loevenich, Markus Tschurtschenthaler, Matjaz Rokavec, Miguel G. Silva, Moritz Jesinghaus, Thomas Kirchner, Frederick Klauschen, Dieter Saur, Jens Neumann, Heiko Hermeking, et al.
Website
Google Scholar
- 31 March 2023
- Published by American Association for Cancer Research (AACR)
Open Access
Supplementary table S2 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
by Leon P. Loevenich, Markus Tschurtschenthaler, Matjaz Rokavec, Miguel G. Silva, Moritz Jesinghaus, Thomas Kirchner, Frederick Klauschen, Dieter Saur, Jens Neumann, Heiko Hermeking, et al.
Open Access
Website
Google Scholar
- 31 March 2023
- Published by American Association for Cancer Research (AACR)
Supplementary Data from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
by Leon P. Loevenich, Markus Tschurtschenthaler, Matjaz Rokavec, Miguel G. Silva, Moritz Jesinghaus, Thomas Kirchner, Frederick Klauschen, Dieter Saur, Jens Neumann, Heiko Hermeking, et al.
Supplementary Materials Oligonucleotides, qRT-PCR primer pairs, and Taqman probe assays used in this study.
Website
Google Scholar
- 31 March 2023
- Published by American Association for Cancer Research (AACR)
Open Access
Supplementary Data from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
by Leon P. Loevenich, Markus Tschurtschenthaler, Matjaz Rokavec, Miguel G. Silva, Moritz Jesinghaus, Thomas Kirchner, Frederick Klauschen, Dieter Saur, Jens Neumann, Heiko Hermeking, et al.
Supplementary Materials Oligonucleotides, qRT-PCR primer pairs, and Taqman probe assays used in this study.
Open Access
Website
Google Scholar
- 31 March 2023
- Published by American Association for Cancer Research (AACR)
Supplementary table S3 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
by Leon P. Loevenich, Markus Tschurtschenthaler, Matjaz Rokavec, Miguel G. Silva, Moritz Jesinghaus, Thomas Kirchner, Frederick Klauschen, Dieter Saur, Jens Neumann, Heiko Hermeking, et al.
Upregulated genes in SMAD4 mutant tumor organoid lines and DepMAP Project dependency scores.
Website
Google Scholar
- 31 March 2023
- Published by American Association for Cancer Research (AACR)
Open Access
Supplementary table S3 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
by Leon P. Loevenich, Markus Tschurtschenthaler, Matjaz Rokavec, Miguel G. Silva, Moritz Jesinghaus, Thomas Kirchner, Frederick Klauschen, Dieter Saur, Jens Neumann, Heiko Hermeking, et al.
Upregulated genes in SMAD4 mutant tumor organoid lines and DepMAP Project dependency scores.

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