Alexa Corker
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Alexa Corker has a total of 18 co-authors. Here are the top co-authors by number of shared publications:
- Amy D. Bradshaw
Medical University of South Carolina
2 shared publications - Kristine Y. DeLeon-Pennell
Medical University of South Carolina
2 shared publications - Amanda C. LaRue
Research Service, Ralph H. Johnson VA Health Care System, Charleston, USA
1 shared publication - Amber Hazzard
College of Graduate Studies, Medical University of South Carolina, 171 Ashley Ave, Charleston, SC 29425, United States; Department of Medicine, Division of Cardiology, Medical University of South Carolina, 171 Ashley Ave, Charleston, SC 29425, United States; RResearch Service, Ralph H. Johnson Veterans Affairs Health Care System, 109 Bee St, Charleston, SC 29401, United States
1 shared publication - An Van Laer
Department of Medicine, Division of Cardiology, Medical University of South Carolina, 171 Ashley Ave, Charleston, SC 29425, United States; RResearch Service, Ralph H. Johnson Veterans Affairs Health Care System, 109 Bee St, Charleston, SC 29401, United States
1 shared publication - Jeffrey A. Jones
Department of Surgery, Division of Cardiothoracic Surgery, Medical University of South Carolina, Charleston, SC 29425, USA
1 shared publication
Latest Publications
- Open Access
- 18 February 2025
- Published by Elsevier in Journal of Molecular and Cellular Cardiology
- Vol. 201, 32-43
Mouse model of post-traumatic stress disorder negatively impacts cardiac homeostasis
- 18 February 2025
- Published by Elsevier in Journal of Molecular and Cellular Cardiology
- Vol. 201, 32-43
Open AccessMouse model of post-traumatic stress disorder negatively impacts cardiac homeostasis
- Open Access
- 22 December 2021
- Published by MDPI AG in Biomolecules
- Vol. 12 (1) , 11
Organized Chaos: Deciphering Immune Cell Heterogeneity’s Role in Inflammation in the Heart
During homeostasis, immune cells perform daily housekeeping functions to maintain heart health by acting as sentinels for tissue damage and foreign particles. Resident immune cells compose 5% of the cellular population in healthy human ventricular tissue. In response to injury, there is an increase in inflammation within the heart due to the influx of immune cells. Some of the most common immune cells recruited to the heart are macrophages, dendritic cells, neutrophils, and T-cells. In this review, we will discuss what is known about cardiac immune cell heterogeneity during homeostasis, how these cell populations change in response to a pathology such as myocardial infarction or pressure overload, and what stimuli are regulating these processes. In addition, we will summarize technologies used to evaluate cell heterogeneity in models of cardiovascular disease.
- 22 December 2021
- Published by MDPI AG in Biomolecules
- Vol. 12 (1) , 11
Open AccessOrganized Chaos: Deciphering Immune Cell Heterogeneity’s Role in Inflammation in the Heart
During homeostasis, immune cells perform daily housekeeping functions to maintain heart health by acting as sentinels for tissue damage and foreign particles. Resident immune cells compose 5% of the cellular population in healthy human ventricular tissue. In response to injury, there is an increase in inflammation within the heart due to the influx of immune cells. Some of the most common immune cells recruited to the heart are macrophages, dendritic cells, neutrophils, and T-cells. In this review, we will discuss what is known about cardiac immune cell heterogeneity during homeostasis, how these cell populations change in response to a pathology such as myocardial infarction or pressure overload, and what stimuli are regulating these processes. In addition, we will summarize technologies used to evaluate cell heterogeneity in models of cardiovascular disease.