Sandra Larouche
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Sandra Larouche has a total of 28 co-authors. Here are the top co-authors by number of shared publications:
Alain Bouthillier
Division of Neurosurgery, Université de Montréal and CHUM, Montreal, Quebec H2L 4M1, Canada
1 shared publicationAlexander Flügel
Institute for Neuroimmunology and Multiple Sclerosis Research, University Medical Center Göttingen, Göttingen D-37073, Germany
1 shared publication- Alexandre Prat
Neuroimmunology Research Lab, Centre de Recherche du Centre Hospitalier de l’Université de Montréal (CRCHUM), Montreal, Canada
1 shared publication Ali Filali-Mouhim
Neuroimmunology Research Laboratory, Centre de Recherche du Centre Hospitalier de l’Université de Montréal (CRCHUM), Montreal, Quebec H2X 0A9, Canada
1 shared publication- Antoine Philippe Fournier
Institut Blood and Brain @ Caen‐Normandie (BB@C) INSERM UMR‐S U1237 Physiopathology and Imaging of Neurological Disorders (PhIND) GIP Cyceron UNICAEN, Université Caen Normandie Normandie University Caen France
1 shared publication - Boaz Lahav
Neuroimmunology Research Laboratory, Centre de Recherche du Centre Hospitalier de l’Université de Montréal (CRCHUM), Montreal, Quebec H2X 0A9, Canada
1 shared publication
Latest Publications
- 5 January 2022
- Published by American Association for the Advancement of Science (AAAS) in Science Translational Medicine
- Vol. 14 (626) , eabj0473
DICAM promotes T H 17 lymphocyte trafficking across the blood-brain barrier during autoimmune neuroinflammation
DICAM contributes to the pathogenesis of autoimmune neuroinflammation by promoting the migration of T H 17 lymphocytes across the blood-brain barrier.
- 5 January 2022
- Published by American Association for the Advancement of Science (AAAS) in Science Translational Medicine
- Vol. 14 (626) , eabj0473
DICAM promotes T H 17 lymphocyte trafficking across the blood-brain barrier during autoimmune neuroinflammation
DICAM contributes to the pathogenesis of autoimmune neuroinflammation by promoting the migration of T H 17 lymphocytes across the blood-brain barrier.