Ying Li
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Previous Organizations / Institutes
The organizations / institutes from the scholar's other articles in the past ten years
Research Subjects
Ying Li has a total of 193 co-authors. Here are the top co-authors by number of shared publications:
- Henry Yang
Cancer Science Institute of Singapore, National University of Singapore, Singapore, Singapore
75 shared publications - Khine Nyein Myint
Cancer Science Institute of Singapore, National University of Singapore, Singapore
33 shared publications - Akihiro Yamamura
Cancer Science Institute of Singapore, National University of Singapore, Singapore
29 shared publications - Atsushi Kaneda
Chiba University Graduate School of Medicine
29 shared publications - Atsushi Okabe
Chiba University Graduate School of Medicine, Chiba, Japan
29 shared publications - Dominic Chih-Cheng Voon
Division of Genetics, Cancer Research Institute, Kanazawa University, Kanazawa, Japan
29 shared publications
Latest Publications
- Open Access
- 24 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary file from Targeting RNA Exonuclease XRN1 Potentiates Efficacy of Cancer Immunotherapy
This file contains supplementary methods, Figs. S1 to S6 and Tables S1 to S5.
- 24 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary file from Targeting RNA Exonuclease XRN1 Potentiates Efficacy of Cancer Immunotherapy
This file contains supplementary methods, Figs. S1 to S6 and Tables S1 to S5.
- Open Access
- 3 July 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S4 from B-cell Receptor Silencing Reveals the Origin and Dependencies of High-Grade B-cell Lymphomas with MYC and BCL2 Rearrangements
IGH and CD79B expression and interaction in HGBCL-DH-BCL2(-BCL6), and relation to follicular lymphoma.
- 3 July 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S4 from B-cell Receptor Silencing Reveals the Origin and Dependencies of High-Grade B-cell Lymphomas with MYC and BCL2 Rearrangements
IGH and CD79B expression and interaction in HGBCL-DH-BCL2(-BCL6), and relation to follicular lymphoma.
- Open Access
- 3 July 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S5 from B-cell Receptor Silencing Reveals the Origin and Dependencies of High-Grade B-cell Lymphomas with MYC and BCL2 Rearrangements
Configuration of IGH, MYC and BCL2 loci in HGBCL-DH-BCL2(-BCL6) and RAG1/2 involvement in t(8;22).
- 3 July 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S5 from B-cell Receptor Silencing Reveals the Origin and Dependencies of High-Grade B-cell Lymphomas with MYC and BCL2 Rearrangements
Configuration of IGH, MYC and BCL2 loci in HGBCL-DH-BCL2(-BCL6) and RAG1/2 involvement in t(8;22).
- Open Access
- 3 July 2025
- Published by American Association for Cancer Research (AACR)
Supplementary figure S6 from B-cell Receptor Silencing Reveals the Origin and Dependencies of High-Grade B-cell Lymphomas with MYC and BCL2 Rearrangements
Cytogenetic of COH-DHL1 and COH-THL1 HGBCL-DH-BCL2(-BCL6) cell line models (A-D); In vivo secondary Ighv gene rearrangements in BCR-less murine lambda-MYC lymphomas (E-J)
- 3 July 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary figure S6 from B-cell Receptor Silencing Reveals the Origin and Dependencies of High-Grade B-cell Lymphomas with MYC and BCL2 Rearrangements
Cytogenetic of COH-DHL1 and COH-THL1 HGBCL-DH-BCL2(-BCL6) cell line models (A-D); In vivo secondary Ighv gene rearrangements in BCR-less murine lambda-MYC lymphomas (E-J)
- Open Access
- 3 July 2025
- Published by American Association for Cancer Research (AACR)
Supplementary figure S4 from B-cell Receptor Silencing Reveals the Origin and Dependencies of High-Grade B-cell Lymphomas with MYC and BCL2 Rearrangements
IGH and CD79B expression, complex formation and IGH isotype choice in DLBCL and HGBCL-DH-BCL2
- 3 July 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary figure S4 from B-cell Receptor Silencing Reveals the Origin and Dependencies of High-Grade B-cell Lymphomas with MYC and BCL2 Rearrangements
IGH and CD79B expression, complex formation and IGH isotype choice in DLBCL and HGBCL-DH-BCL2
- Open Access
- 3 July 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S1 from B-cell Receptor Silencing Reveals the Origin and Dependencies of High-Grade B-cell Lymphomas with MYC and BCL2 Rearrangements
DLBCL cases included in the study.
- 3 July 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S1 from B-cell Receptor Silencing Reveals the Origin and Dependencies of High-Grade B-cell Lymphomas with MYC and BCL2 Rearrangements
DLBCL cases included in the study.
- Open Access
- 3 July 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S7 from B-cell Receptor Silencing Reveals the Origin and Dependencies of High-Grade B-cell Lymphomas with MYC and BCL2 Rearrangements
Supplementary Table S7 from B-cell Receptor Silencing Reveals the Origin and Dependencies of High-Grade B-cell Lymphomas with MYC and BCL2 Rearrangements
- 3 July 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S7 from B-cell Receptor Silencing Reveals the Origin and Dependencies of High-Grade B-cell Lymphomas with MYC and BCL2 Rearrangements
Supplementary Table S7 from B-cell Receptor Silencing Reveals the Origin and Dependencies of High-Grade B-cell Lymphomas with MYC and BCL2 Rearrangements
- Open Access
- 3 July 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S2 from B-cell Receptor Silencing Reveals the Origin and Dependencies of High-Grade B-cell Lymphomas with MYC and BCL2 Rearrangements
Bulk and spatial gene expression data of MYC and BCL2 dual expressor DLBCL and HGBCL-DH-BCL2 (-BCL6).
- 3 July 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S2 from B-cell Receptor Silencing Reveals the Origin and Dependencies of High-Grade B-cell Lymphomas with MYC and BCL2 Rearrangements
Bulk and spatial gene expression data of MYC and BCL2 dual expressor DLBCL and HGBCL-DH-BCL2 (-BCL6).
- Open Access
- 3 July 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S3 from B-cell Receptor Silencing Reveals the Origin and Dependencies of High-Grade B-cell Lymphomas with MYC and BCL2 Rearrangements
Non synonymus SNVs, IGHV gene rearrangements, AID expression status and N-glycosylation motifs in IGHV domains of HGBCL-DH-BCL2(-BCL6).
- 3 July 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S3 from B-cell Receptor Silencing Reveals the Origin and Dependencies of High-Grade B-cell Lymphomas with MYC and BCL2 Rearrangements
Non synonymus SNVs, IGHV gene rearrangements, AID expression status and N-glycosylation motifs in IGHV domains of HGBCL-DH-BCL2(-BCL6).