Juan Carlos Alamilla-Presuel
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Juan Carlos Alamilla-Presuel has a total of 7 co-authors. Here are the top co-authors by number of shared publications:
- Antonio Manuel Burgos-Molina
Surgery, Biochemistry and Immunology Department, School of Medicine, University of Malaga, 29010 Malaga, Spain
3 shared publications - Francisco Sendra-Portero
Department of Radiology and Physical Medicine, Facultad de Medicina, Universidad de Málaga, Málaga, Spain
3 shared publications - Miguel Jose Ruiz-Gomez
Department of Radiology and Physical Medicine, Faculty of Medicine, University of Málaga, Málaga, Spain
3 shared publications - Alejandro González-Vidal
Departamento de Radiología y Medicina Física, Facultad de Medicina, Universidad de Málaga, 29010 Málaga, Spain
2 shared publications - Miguel Alcaraz
Radiology and Physical Medicine Department, School of Medicine, Campus de Excelencia Internacional de Ámbito Regional (CEIR)-Campus Mare Nostrum (CMN), University of Murcia, 30100 Murcia, Spain
2 shared publications - Casimiro Cárdenas
Central Research Support Services (SCAI), University of Málaga, Campus de Teatinos s/n, E-29071 Málaga, Spain
1 shared publication
Latest Publications
- Open Access
- 12 August 2026
- Published by Elsevier in Radiation Physics and Chemistry
- Vol. 250
Proteomic identification of candidate biomarkers associated with acquired tolerance to ionizing radiation
- 12 August 2026
- Published by Elsevier in Radiation Physics and Chemistry
- Vol. 250
Open AccessProteomic identification of candidate biomarkers associated with acquired tolerance to ionizing radiation
- Open Access
- 30 August 2023
- Published by MDPI AG in Antioxidants
- Vol. 12 (9) , 1690
Molecular Mechanisms of Resistance to Ionizing Radiation in S. cerevisiae and Its Relationship with Aging, Oxidative Stress, and Antioxidant Activity
The repair of the damage produced to the genome and proteome by the action of ionizing radiation, oxidizing agents, and during aging is important to maintain cellular homeostasis. Many of the metabolic pathways influence multiple processes. In this way, this work aims to study the relationship between resistance/response to ionizing radiation, cellular aging, and the response mechanisms to oxidative stress, free radicals, reactive oxygen species (ROS), and antioxidant activity in the yeast S. cerevisiae. Systems biology allows us to use tools that reveal the molecular mechanisms common to different cellular response phenomena. The results found indicate that homologous recombination, non-homologous end joining, and base excision repair pathways are the most important common processes necessary to maintain cellular homeostasis. The metabolic routes of longevity regulation are those that jointly contribute to the three phenomena studied. This study proposes eleven common biomarkers for response/resistance to ionizing radiation and aging (EXO1, MEC1, MRE11, RAD27, RAD50, RAD51, RAD52, RAD55, RAD9, SGS1, YKU70) and two biomarkers for response/resistance to radiation and oxidative stress, free radicals, ROS, and antioxidant activity (NTG1, OGG1). In addition, it is important to highlight that the HSP104 protein could be a good biomarker common to the three phenomena studied.
- 30 August 2023
- Published by MDPI AG in Antioxidants
- Vol. 12 (9) , 1690
Open AccessMolecular Mechanisms of Resistance to Ionizing Radiation in S. cerevisiae and Its Relationship with Aging, Oxidative Stress, and Antioxidant Activity
The repair of the damage produced to the genome and proteome by the action of ionizing radiation, oxidizing agents, and during aging is important to maintain cellular homeostasis. Many of the metabolic pathways influence multiple processes. In this way, this work aims to study the relationship between resistance/response to ionizing radiation, cellular aging, and the response mechanisms to oxidative stress, free radicals, reactive oxygen species (ROS), and antioxidant activity in the yeast S. cerevisiae. Systems biology allows us to use tools that reveal the molecular mechanisms common to different cellular response phenomena. The results found indicate that homologous recombination, non-homologous end joining, and base excision repair pathways are the most important common processes necessary to maintain cellular homeostasis. The metabolic routes of longevity regulation are those that jointly contribute to the three phenomena studied. This study proposes eleven common biomarkers for response/resistance to ionizing radiation and aging (EXO1, MEC1, MRE11, RAD27, RAD50, RAD51, RAD52, RAD55, RAD9, SGS1, YKU70) and two biomarkers for response/resistance to radiation and oxidative stress, free radicals, ROS, and antioxidant activity (NTG1, OGG1). In addition, it is important to highlight that the HSP104 protein could be a good biomarker common to the three phenomena studied.
- 7 March 2022
- Published by Taylor & Francis in International Journal of Radiation Biology
- Vol. 98 (8) , 1301-1315
Factors and molecular mechanisms of radiation resistance in cancer cells
The aim of this work is to review the published studies on radiation resistance mechanisms and molecular markers involved in different tumors. The revision has been focused in the last 5 years (201...
- 7 March 2022
- Published by Taylor & Francis in International Journal of Radiation Biology
- Vol. 98 (8) , 1301-1315
Factors and molecular mechanisms of radiation resistance in cancer cells
The aim of this work is to review the published studies on radiation resistance mechanisms and molecular markers involved in different tumors. The revision has been focused in the last 5 years (201...