David G. Edwards
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
David G. Edwards has a total of 77 co-authors. Here are the top co-authors by number of shared publications:
- Igor L. Bado
Icahn School of Medicine at Mount Sinai 12 , New York, New York
38 shared publications - Ling Wu
Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, USA
38 shared publications - Xiang H.-F. Zhang
Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, TX 77030, USA
38 shared publications - Xiaoxin Hao
Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, Texas
38 shared publications - Yi-Hsuan Wu
Lester and Sue Smith Breast Center, Baylor College of Medicine, Houston, Texas
38 shared publications - Jun Liu
Baylor College of Medicine 1 Lester and Sue Smith Breast Center Houston, Texas
34 shared publications
Latest Publications
- Open Access
- 14 August 2026
- Published by American Association for Cancer Research (AACR)
Table S3 from ICAM1high Neutrophils Sculpt Tumor Evolution and Metastasis Through Symbiotic Adhesion and Reverse Migration
Table S3 from ICAM1high Neutrophils Sculpt Tumor Evolution and Metastasis Through Symbiotic Adhesion and Reverse Migration
- 14 August 2026
- Published by American Association for Cancer Research (AACR)
Open AccessTable S3 from ICAM1high Neutrophils Sculpt Tumor Evolution and Metastasis Through Symbiotic Adhesion and Reverse Migration
Table S3 from ICAM1high Neutrophils Sculpt Tumor Evolution and Metastasis Through Symbiotic Adhesion and Reverse Migration
- Open Access
- 14 August 2026
- Published by American Association for Cancer Research (AACR)
Figure S6 from ICAM1high Neutrophils Sculpt Tumor Evolution and Metastasis Through Symbiotic Adhesion and Reverse Migration
Supplementary figure for ICAM1high neutrophils promote metastasis of epithelial cancer cells through reverse transendothelial migration
- 14 August 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S6 from ICAM1high Neutrophils Sculpt Tumor Evolution and Metastasis Through Symbiotic Adhesion and Reverse Migration
Supplementary figure for ICAM1high neutrophils promote metastasis of epithelial cancer cells through reverse transendothelial migration
- Open Access
- 14 August 2026
- Published by American Association for Cancer Research (AACR)
Figure S4 from ICAM1high Neutrophils Sculpt Tumor Evolution and Metastasis Through Symbiotic Adhesion and Reverse Migration
Supplementary figure for EMT disrupts the symbiotic interaction
- 14 August 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S4 from ICAM1high Neutrophils Sculpt Tumor Evolution and Metastasis Through Symbiotic Adhesion and Reverse Migration
Supplementary figure for EMT disrupts the symbiotic interaction
- Open Access
- 14 August 2026
- Published by American Association for Cancer Research (AACR)
Figure S8 from ICAM1high Neutrophils Sculpt Tumor Evolution and Metastasis Through Symbiotic Adhesion and Reverse Migration
Supplementary figure for clinical association of ICAM1high neutrophils in indicated cancer types
- 14 August 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S8 from ICAM1high Neutrophils Sculpt Tumor Evolution and Metastasis Through Symbiotic Adhesion and Reverse Migration
Supplementary figure for clinical association of ICAM1high neutrophils in indicated cancer types
- Open Access
- 14 August 2026
- Published by American Association for Cancer Research (AACR)
Figure S2 from ICAM1high Neutrophils Sculpt Tumor Evolution and Metastasis Through Symbiotic Adhesion and Reverse Migration
Supplementary figure for the symbiotic interaction between neutrophils and epithelial cancer cells
- 14 August 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S2 from ICAM1high Neutrophils Sculpt Tumor Evolution and Metastasis Through Symbiotic Adhesion and Reverse Migration
Supplementary figure for the symbiotic interaction between neutrophils and epithelial cancer cells
- Open Access
- 14 August 2026
- Published by American Association for Cancer Research (AACR)
Figure S5 from ICAM1high Neutrophils Sculpt Tumor Evolution and Metastasis Through Symbiotic Adhesion and Reverse Migration
Supplementary figure for ICAM1high neutrophils may promote metastasis of epithelial cancer cells through reverse transendothelial migration
- 14 August 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S5 from ICAM1high Neutrophils Sculpt Tumor Evolution and Metastasis Through Symbiotic Adhesion and Reverse Migration
Supplementary figure for ICAM1high neutrophils may promote metastasis of epithelial cancer cells through reverse transendothelial migration
- Open Access
- 14 August 2026
- Published by American Association for Cancer Research (AACR)
Figure S7 from ICAM1high Neutrophils Sculpt Tumor Evolution and Metastasis Through Symbiotic Adhesion and Reverse Migration
Supplementary figure for interaction among tumor cells, neutrophils and endothelial cells in human TNBC exhibits potential racial disparity
- 14 August 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S7 from ICAM1high Neutrophils Sculpt Tumor Evolution and Metastasis Through Symbiotic Adhesion and Reverse Migration
Supplementary figure for interaction among tumor cells, neutrophils and endothelial cells in human TNBC exhibits potential racial disparity
- Open Access
- 14 August 2026
- Published by American Association for Cancer Research (AACR)
Figure S1 from ICAM1high Neutrophils Sculpt Tumor Evolution and Metastasis Through Symbiotic Adhesion and Reverse Migration
Supplementary figure for ICAM1high neutrophils are enriched in solid epithelial tumors
- 14 August 2026
- Published by American Association for Cancer Research (AACR)
Open AccessFigure S1 from ICAM1high Neutrophils Sculpt Tumor Evolution and Metastasis Through Symbiotic Adhesion and Reverse Migration
Supplementary figure for ICAM1high neutrophils are enriched in solid epithelial tumors