Tengyue Hu
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Research Interests
Tengyue Hu has a total of 7 co-authors. Here are the top co-authors by number of shared publications:
- Hong Tang
Center of Infectious Diseases, West China Hospital, Sichuan University, Chengdu 610041, China
1 shared publication - Jiayi Wang
Center of Infectious Diseases West China Hospital of Sichuan University Chengdu China
1 shared publication - Lingyao Du
Center of Infectious Diseases, West China Hospital of Sichuan University, Chengdu 610041, China
1 shared publication - Man Yuan
Center of Infectious Diseases West China Hospital of Sichuan University Chengdu China
1 shared publication - Miao Liu
Center of Infectious Diseases, West China Hospital of Sichuan University, Chengdu 610041, China
1 shared publication - Ning Han
Center of Infectious Diseases, West China Hospital of Sichuan University, Chengdu 610041, China
1 shared publication
Latest Publications
- Open Access
Endoplasmic Reticulum Stress in Hepatitis B Virus and Hepatitis C Virus Infection
Endoplasmic reticulum (ER) stress, a type of cellular stress, always occurs when unfolded or misfolded proteins accumulating in the ER exceed the protein folding capacity. Because of the demand for rapid viral protein synthesis after viral infection, viral infections become a risk factor for ER stress. The hepatocyte is a cell with large and well-developed ER, and hepatitis virus infection is widespread in the population, indicating the interaction between hepatitis viruses and ER stress may have significance for managing liver diseases. In this paper, we review the process that is initiated by the hepatocyte through ER stress against HBV and HCV infection and explain how this information can be helpful in the treatment of HBV/HCV-related diseases.
Open AccessEndoplasmic Reticulum Stress in Hepatitis B Virus and Hepatitis C Virus Infection
Endoplasmic reticulum (ER) stress, a type of cellular stress, always occurs when unfolded or misfolded proteins accumulating in the ER exceed the protein folding capacity. Because of the demand for rapid viral protein synthesis after viral infection, viral infections become a risk factor for ER stress. The hepatocyte is a cell with large and well-developed ER, and hepatitis virus infection is widespread in the population, indicating the interaction between hepatitis viruses and ER stress may have significance for managing liver diseases. In this paper, we review the process that is initiated by the hepatocyte through ER stress against HBV and HCV infection and explain how this information can be helpful in the treatment of HBV/HCV-related diseases.