Dexter X. Jin
Research Subjects
Dexter X. Jin has a total of 117 co-authors. Here are the top co-authors by number of shared publications:
- Ethan S. Sokol
Foundation Medicine , Cambridge, MA
62 shared publications - Garrett M. Frampton
Computational Discovery, Foundation Medicine, Inc., Cambridge, USA
50 shared publications - Smruthy Sivakumar
Computational Discovery, Foundation Medicine, Boston, MA
48 shared publications - David L. Corcoran
University of North Carolina at Chapel Hill
47 shared publications - Jay A. Moore
Foundation Medicine, Inc., Boston, Massachusetts. 5
47 shared publications - Jeffrey S. Damrauer
Lineberger Comprehensive Cancer Center, University of North Carolina at Chapel Hill, Chapel Hill, USA
47 shared publications
Latest Publications
- Open Access
- 2 September 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S4 from Deficiency in POLE Exonuclease Causes Synthetic Lethality in Highly Aneuploid Cancer Cells
Association analyses of aneuploidy score with AR, GID4, AKT3, FGF10, EWSR1, and POLD1
- 2 September 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S4 from Deficiency in POLE Exonuclease Causes Synthetic Lethality in Highly Aneuploid Cancer Cells
Association analyses of aneuploidy score with AR, GID4, AKT3, FGF10, EWSR1, and POLD1
- Open Access
- 2 September 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S8 from Deficiency in POLE Exonuclease Causes Synthetic Lethality in Highly Aneuploid Cancer Cells
Structural analysis of the exonuclease domain of POLE
- 2 September 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S8 from Deficiency in POLE Exonuclease Causes Synthetic Lethality in Highly Aneuploid Cancer Cells
Structural analysis of the exonuclease domain of POLE
- Open Access
- 2 September 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S3 from Deficiency in POLE Exonuclease Causes Synthetic Lethality in Highly Aneuploid Cancer Cells
Detailed characterization of TP53 alterations in breast cancer, colorectal cancer, NSCLC, ovarian cancer, pancreatic cancer and prostate cancer
- 2 September 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S3 from Deficiency in POLE Exonuclease Causes Synthetic Lethality in Highly Aneuploid Cancer Cells
Detailed characterization of TP53 alterations in breast cancer, colorectal cancer, NSCLC, ovarian cancer, pancreatic cancer and prostate cancer
- Open Access
- 2 September 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S6 from Deficiency in POLE Exonuclease Causes Synthetic Lethality in Highly Aneuploid Cancer Cells
Characterization of whole genome doubling status
- 2 September 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S6 from Deficiency in POLE Exonuclease Causes Synthetic Lethality in Highly Aneuploid Cancer Cells
Characterization of whole genome doubling status
- Open Access
- 2 September 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Table S1 from Deficiency in POLE Exonuclease Causes Synthetic Lethality in Highly Aneuploid Cancer Cells
Trinucleotide motif counts on human genome
- 2 September 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S1 from Deficiency in POLE Exonuclease Causes Synthetic Lethality in Highly Aneuploid Cancer Cells
Trinucleotide motif counts on human genome
- Open Access
- 2 September 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S7 from Deficiency in POLE Exonuclease Causes Synthetic Lethality in Highly Aneuploid Cancer Cells
Additional analyses of the all-in-one inducible adenine base editor
- 2 September 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S7 from Deficiency in POLE Exonuclease Causes Synthetic Lethality in Highly Aneuploid Cancer Cells
Additional analyses of the all-in-one inducible adenine base editor
- Open Access
- 2 September 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S5 from Deficiency in POLE Exonuclease Causes Synthetic Lethality in Highly Aneuploid Cancer Cells
Association analyses of aneuploidy score with TMB, mutational signatures, and microsatellite instability
- 2 September 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S5 from Deficiency in POLE Exonuclease Causes Synthetic Lethality in Highly Aneuploid Cancer Cells
Association analyses of aneuploidy score with TMB, mutational signatures, and microsatellite instability
- Open Access
- 2 September 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Table S3 from Deficiency in POLE Exonuclease Causes Synthetic Lethality in Highly Aneuploid Cancer Cells
SgRNA counts from the base editor screening
- 2 September 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S3 from Deficiency in POLE Exonuclease Causes Synthetic Lethality in Highly Aneuploid Cancer Cells
SgRNA counts from the base editor screening
- Open Access
- 2 September 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Table S6 from Deficiency in POLE Exonuclease Causes Synthetic Lethality in Highly Aneuploid Cancer Cells
Elastic net regression for TMB
- 2 September 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S6 from Deficiency in POLE Exonuclease Causes Synthetic Lethality in Highly Aneuploid Cancer Cells
Elastic net regression for TMB
- Open Access
- 2 September 2026
- Published by American Association for Cancer Research (AACR)
Supplementary Table S2 from Deficiency in POLE Exonuclease Causes Synthetic Lethality in Highly Aneuploid Cancer Cells
- 2 September 2026
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S2 from Deficiency in POLE Exonuclease Causes Synthetic Lethality in Highly Aneuploid Cancer Cells