Hongjun Dong

Photocatalytic CO ₂ reduction to C ₂ H ₄ offers a sustainable route for hydrocarbon production but is hindered by poor selectivity and rapid charge recombination. Herein, we develop a Cu/S/N codoped orthorhombic WO ₃ photocatalyst through a facile solvothermal strategy. The charge‐compensated cation/anion codoping simultaneously reduces oxygen vacancies, relieves lattice strain, stabilizes the metastable orthorhombic phase, and boosts carrier separation and interfacial charge transfer. Meanwhile, the preferentially exposed (100) facet provides an abundant unsaturated coordination microenvironment that promotes CO ₂ adsorption and reduces the C─C coupling barrier, thereby synergistically driving C ₂ H ₄ formation. The optimal Cu/S/N‐WO ₃ ‐5 achieves a C ₂ H ₄ yield 29.8‐fold and selectivity 1.75‐fold those of the pristine WO ₃ within 4 h, respectively. In situ diffuse reflectance infrared Fourier transform spectroscopy (DRIFTS) identifies key intermediates and reveals three parallel C ₂ H ₄ generation pathways, namely *CO dimerization, *CHO‐mediated coupling, and *CH ₂ direct dimerization. This work demonstrates that charge‐compensated codoping combined with crystal plane engineering offers a versatile approach for stabilizing metastable phases and steering product selectivity toward valuable multicarbon products in photocatalytic CO ₂ reduction.

Designing electrocatalysts for industrial‐level currents across a wide pH range is challenging. Here, we report a hierarchical RuMo alloy nanoflower synthesized via a one‐step solvothermal method. The unique three‐dimensional nanoflower architecture provides abundant accessible active sites, enhanced mass transport, and excellent structural robustness, while Mo alloying tailors the electronic structure, optimizes hydrogen adsorption energy, and accelerates water dissociation kinetics. The optimal RuMo‐1.5 sample requires ultralow overpotentials of only 223 mV in alkaline, 389 mV in neutral, and 306 mV in acidic electrolytes to achieve a current density of 1000 mA cm ⁻² , remarkably outperforming commercial Pt/C and most state‐of‐the‐art Ru‐based catalysts. Moreover, it sustains continuous operation at 1000 mA cm ⁻² for over 120 h in all pH media without noticeable degradation, demonstrating outstanding long‐term durability. This work provides a highly efficient and durable universal‐pH electrocatalyst for large‐scale hydrogen production and establishes a rational design strategy, integrating morphological engineering with electronic modulation.

Achieving the exposure of high‐energy active crystal planes is a very challenging issue due to the thermodynamic limitations. Herein, Pd nanosheets with exposed high‐energy (1–10) crystal planes were constructed on IRMOF via layer‐by‐layer (LBL) electrodeposition. The metal–organic framework (MOF) matrix prevents Pd nanosheets aggregation through spatial isolation, while Pd─N bonds with ─NH ₂ groups in IRMOF stabilize the high‐energy Pd (1–10) crystal planes, enabling their continued activity under acidic hydrogen evolution reaction (HER) conditions. Benefiting from the intrinsically favorable hydrogen adsorption thermodynamics of the Pd (1–10) crystal plane and the high density of low‐coordination Pd sites, the optimized Pd/IRMOF‐3 achieves an ultralow overpotential of 15 and 370 mV at 10 and 1000 mA cm ⁻² , respectively, outperforming commercial Pt/C. Moreover, it exhibits outstanding durability, sustaining HER for 320 h at 10 mA cm ⁻² , accompanied by preserved crystallinity and surface chemistry after long‐term HER. This work demonstrates that MOF‐stabilized Pd nanosheets with exposed active crystal planes offer a design for achieving simultaneously high activity and long‐term stability of electrocatalysts, providing insights for the rational construction of next‐generation electrocatalysts.

The rational design of low-cost bifunctional electrocatalysts for overall water-splitting constitutes a pivotal scientific challenge on the path to carbon-neutral energy cycles. Herein, we report a unique NiPd/Pd heterodimer synthesized via an in situ etching-growth route, which functions as a highly efficient bifunctional catalyst for both the hydrogen evolution reaction (HER) and the oxygen evolution reaction (OER). The optimized NiPd/Pd-2 heterodimer delivers exceptional activity, requiring ultralow overpotentials of only 15 mV for HER and 300 mV for OER to achieve a current density of 10 mA cm^-2, substantially outperforming the commercial Pt/C (39 mV) and RuO₂ (370 mV), respectively. Remarkably, when configured as a symmetric NiPd/Pd-2||NiPd/Pd-2 electrolyzer, it requires a cell voltage of merely 1.36 V to drive overall water splitting at 10 mA cm^-2, surpassing the performance of the Pt/C||RuO₂ couple (1.70 V). Structural and chemical characterizations after prolonged HER/OER reveal the preserved heterodimeric architecture of NiPd/Pd, with the increased content of Ni-O-O-Pd motif imparting enhanced stability in acidic conditions. Consequently, the NiPd/Pd heterodimer can maintain stable operation of HER, OER, and overall water electrolysis for 24 h without significant attenuation. Furthermore, the electrolyzer can be efficiently powered by simulated renewable sources, highlighting its great potential for sustainable energy-conversion devices.

A novel fcc -RuCo@ hcp -Ru core–shell heterostructure is created via cobalt-induced phase transformation. Its abundant fcc/hcp interfaces and electronic synergy deliver superior activity and stability for alkaline hydrogen evolution.

Sudden sensorineural hearing loss (SSNHL) is a common auditory disorder, with oxidative stress and ferroptosis implicated in its pathogenesis. Notoginsenoside R1, a major component of Panax notoginseng, exhibits antioxidant and cytoprotective effects, but its role in ferroptosis regulation remains unclear. In this study, a mouse model of SSNHL and an oxygen‐glucose deprivation/reoxygenation (OGD/R)‐induced Ear Institute‐Organ of Corti 1 (HEI‐OC1) cell model were used to evaluate the effects of Notoginsenoside R1. Notoginsenoside R1 significantly reduced auditory brainstem response (ABR) thresholds, reduced cochlear hair cell apoptosis, and alleviated oxidative damage. It inhibited ferroptosis by decreasing Fe ^{2 +} , ROS, MDA, and LPO levels, while upregulating SOD, GSH, GCL, and GPX4. Mechanistically, nuclear factor erythroid 2‐related factor 2 (NRF2) was identified as a key responsive target through target prediction and qPCR validation, while molecular docking and microscale thermophoresis (MST) assays demonstrated a direct interaction between Notoginsenoside R1 and SQSTM1 (p62). Besides, Notoginsenoside R1 enhanced p62 expression and promoted NRF2 nuclear translocation, restoring expression of GCLC, GCLM, and GPX4. Both p62 knockout and siRNA‐mediated knockdown progressively attenuated these protective effects. These findings suggest that Notoginsenoside R1 protects against SSNHL by inhibiting ferroptosis and oxidative stress via activation of the p62‐NRF2‐GCL‐GPX4 axis.