Kaitlin Long
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Kaitlin Long has a total of 15 co-authors. Here are the top co-authors by number of shared publications:
- Aleksandar Z. Obradovic
Columbia Center for Translational Immunology, Department of Medicine, Columbia University Medical Center, New York, New York
49 shared publications - An Xu
Department of Integrative Biology and Pharmacology, McGovern Medical School, The University of Texas Health Science Center, The University of Texas MD Anderson Cancer Center, Houston, Texas
49 shared publications - Ana Maria Low-Calle
Department of Biological Sciences, Columbia University, New York, New York, USA
49 shared publications - Ananya Raghavan
Department of Biological Sciences, Columbia University, New York, New York
49 shared publications - Asja Guzman
Department of Biological Sciences, Columbia University, New York, New York
49 shared publications - Bita Alaghebandan
Department of Biological Sciences, Columbia University, New York, New York
49 shared publications
Latest Publications
- Open Access
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Data 3 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
Differentially expressed genes in osteoblasts derived from normal and LFS individuals
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Data 3 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
Differentially expressed genes in osteoblasts derived from normal and LFS individuals
- Open Access
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Data 1 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
Differentially expressed genes in U2OS cells varying in p53 allelic status
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Data 1 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
Differentially expressed genes in U2OS cells varying in p53 allelic status
- Open Access
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Data 2 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
Differentially expressed genes in U2OS cells varying in p53 allelic status combined from two independent RNA library preparations
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Data 2 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
Differentially expressed genes in U2OS cells varying in p53 allelic status combined from two independent RNA library preparations
- Open Access
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table 2 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
Characteristics of LFS patients' fibroblasts and derivatives
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table 2 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
Characteristics of LFS patients' fibroblasts and derivatives
- Open Access
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Data 6 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
P53 ChIPseq peaks from osteoblasts derived from normal and LFS individuals
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Data 6 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
P53 ChIPseq peaks from osteoblasts derived from normal and LFS individuals
- Open Access
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table 1 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
Summary of GOF vs. LOF activities of p53(A347D) in U2OS cells
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table 1 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
Summary of GOF vs. LOF activities of p53(A347D) in U2OS cells
- Open Access
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Data 5 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
P53 ChIPseq peaks from U2OS cells varying in p53 allelic status
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Data 5 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
P53 ChIPseq peaks from U2OS cells varying in p53 allelic status
- Open Access
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Figures from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
Supplementary Figures 1-9 demonstrating the GOF and LOF activities of p53(A347D)
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figures from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
Supplementary Figures 1-9 demonstrating the GOF and LOF activities of p53(A347D)
- Open Access
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Data 4 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
P53 ChIPseq peaks from U2OS cells varying in p53 allelic status
- 11 December 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Data 4 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
P53 ChIPseq peaks from U2OS cells varying in p53 allelic status
- Open Access
- 4 May 2023
- Published by American Association for Cancer Research (AACR)
Supplementary Data 3 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
Differentially expressed genes in osteoblasts derived from normal and LFS individuals
- 4 May 2023
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Data 3 from Li–Fraumeni Syndrome–Associated Dimer-Forming Mutant p53 Promotes Transactivation-Independent Mitochondrial Cell Death
Differentially expressed genes in osteoblasts derived from normal and LFS individuals