Winson Wing-Yin Lam
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Research Subjects
Winson Wing-Yin Lam has a total of 8 co-authors. Here are the top co-authors by number of shared publications:
- Hui-Yao Lan
Department of Medicine & Therapeutics, The Chinese University of Hong Kong, Hong Kong, China
1 shared publication - Jeff Yat-Fai Chung
Department of Anatomical and Cellular Pathology, State Key Laboratory of Translational Oncology, The Chinese University of Hong Kong, Hong Kong, China
1 shared publication - Ka-Fai To
Department of Anatomical and Cellular Pathology, The Chinese University of Hong Kong, Hong Kong SAR, China
1 shared publication - Max Kam-Kwan Chan
Department of Anatomical and Cellular Pathology, State Key Laboratory of Translational Oncology, Prince of Wales Hospital, The Chinese University of Hong Kong, Hong Kong 999077, China
1 shared publication - Patrick Ming-Kuen Tang
Department of Anatomical and Cellular Pathology, State Key Laboratory of Translational Oncology, The Chinese University of Hong Kong, Hong Kong, China
1 shared publication - Philip Chiu-Tsun Tang
Department of Anatomical and Cellular Pathology, State Key Laboratory of Translational Oncology, The Chinese University of Hong Kong, Shatin, Hong Kong
1 shared publication
Latest Publications
- Open Access
- 4 June 2020
- Published by MDPI AG in International Journal of Molecular Sciences
- Vol. 21 (11) , 4018
The Emerging Role of Innate Immunity in Chronic Kidney Diseases
Renal fibrosis is a common fate of chronic kidney diseases. Emerging studies suggest that unsolved inflammation will progressively transit into tissue fibrosis that finally results in an irreversible end-stage renal disease (ESRD). Renal inflammation recruits and activates immunocytes, which largely promotes tissue scarring of the diseased kidney. Importantly, studies have suggested a crucial role of innate immunity in the pathologic basis of kidney diseases. This review provides an update of both clinical and experimental information, focused on how innate immune signaling contributes to renal fibrogenesis. A better understanding of the underlying mechanisms may uncover a novel therapeutic strategy for ESRD.
- 4 June 2020
- Published by MDPI AG in International Journal of Molecular Sciences
- Vol. 21 (11) , 4018
Open AccessThe Emerging Role of Innate Immunity in Chronic Kidney Diseases
Renal fibrosis is a common fate of chronic kidney diseases. Emerging studies suggest that unsolved inflammation will progressively transit into tissue fibrosis that finally results in an irreversible end-stage renal disease (ESRD). Renal inflammation recruits and activates immunocytes, which largely promotes tissue scarring of the diseased kidney. Importantly, studies have suggested a crucial role of innate immunity in the pathologic basis of kidney diseases. This review provides an update of both clinical and experimental information, focused on how innate immune signaling contributes to renal fibrogenesis. A better understanding of the underlying mechanisms may uncover a novel therapeutic strategy for ESRD.