Ana Babic
Current Organizations / Institutes
The organizations/institutes from the scholar's latest article
Previous Organizations / Institutes
The organizations / institutes from the scholar's other articles in the past ten years
Ana Babic has a total of 179 co-authors. Here are the top co-authors by number of shared publications:
- Brian M. Wolpin
Dana-Farber Cancer Institute, Boston, MA
87 shared publications - Howard D. Sesso
Division of Preventive Medicine, Department of Medicine, Brigham and Women’s Hospital and Harvard Medical School, Boston, USA
87 shared publications - Peter Kraft
Division of Cancer Epidemiology and Genetics, National Cancer Institute, National Institutes of Health , Rockville, MD
87 shared publications - Charles S. Fuchs
Yale Cancer Center, New Haven, CT
86 shared publications - Edward L. Giovannucci
Department of Nutrition, Harvard T.H. Chan School of Public Health, Boston, Massachusetts
86 shared publications - Jean Wactawski-Wende
Department of Epidemiology and Environmental Health, University at Buffalo , Buffalo, NY
86 shared publications
Latest Publications
- Open Access
- 26 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S1 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
Supplementary Table S1 shows results of univariate analysis for 77 chronically elevated serum alanine aminotransferase (cALT)-defined nonalcoholic fatty liver disease SNPs in the Pancreatic Cancer Cohort Consortium (PanScan) data
- 26 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S1 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
Supplementary Table S1 shows results of univariate analysis for 77 chronically elevated serum alanine aminotransferase (cALT)-defined nonalcoholic fatty liver disease SNPs in the Pancreatic Cancer Cohort Consortium (PanScan) data
- Open Access
- 26 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S2 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
Supplementary Table S2 shows results of univariate analysis for 22 imaging-defined nonalcoholic fatty liver disease SNPs in the PanScan sample
- 26 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S2 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
Supplementary Table S2 shows results of univariate analysis for 22 imaging-defined nonalcoholic fatty liver disease SNPs in the PanScan sample
- Open Access
- 26 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S3 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
Supplementary Table S3 shows results of univariate analysis for 36 biopsy-confirmed nonalcoholic fatty liver disease SNPs in the PanScan sample
- 26 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S3 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
Supplementary Table S3 shows results of univariate analysis for 36 biopsy-confirmed nonalcoholic fatty liver disease SNPs in the PanScan sample
- Open Access
- 26 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S1 from Sex-Specific Associations between Adiponectin and Leptin Signaling and Pancreatic Cancer Survival
Number of pancreatic cancer cases with available plasma adipokine levels and SNPs in adiponectin and leptin receptor genes
- 26 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S1 from Sex-Specific Associations between Adiponectin and Leptin Signaling and Pancreatic Cancer Survival
Number of pancreatic cancer cases with available plasma adipokine levels and SNPs in adiponectin and leptin receptor genes
- Open Access
- 26 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S8 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
Supplementary Table S8 shows results of univariate analysis for 17 imaging and biopsy validated SNPs in the PanC4 sample
- 26 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S8 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
Supplementary Table S8 shows results of univariate analysis for 17 imaging and biopsy validated SNPs in the PanC4 sample
- Open Access
- 26 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S6 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
Supplementary Table S6 shows results of univariate analysis for 22 imaging defined nonalcoholic fatty liver disease SNPs in the PanC4 sample
- 26 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S6 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
Supplementary Table S6 shows results of univariate analysis for 22 imaging defined nonalcoholic fatty liver disease SNPs in the PanC4 sample
- Open Access
- 26 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S2 from Sex-Specific Associations between Adiponectin and Leptin Signaling and Pancreatic Cancer Survival
Association between adiponectin levels and pancreatic cancer mortality using spline regression.
- 26 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S2 from Sex-Specific Associations between Adiponectin and Leptin Signaling and Pancreatic Cancer Survival
Association between adiponectin levels and pancreatic cancer mortality using spline regression.
- Open Access
- 26 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S9 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
Supplementary Table S9 shows descriptive Statistics for PanScan sample
- 26 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S9 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
Supplementary Table S9 shows descriptive Statistics for PanScan sample
- Open Access
- 26 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Table S5 from Sex-Specific Associations between Adiponectin and Leptin Signaling and Pancreatic Cancer Survival
SNPs in adiponectin and leptin receptor genes and their association with receptor expression
- 26 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Table S5 from Sex-Specific Associations between Adiponectin and Leptin Signaling and Pancreatic Cancer Survival
SNPs in adiponectin and leptin receptor genes and their association with receptor expression
- Open Access
- 26 November 2025
- Published by American Association for Cancer Research (AACR)
Supplementary Figure S1 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
Supplementary Figure S1 shows forest plot for minimally adjusted model for the PanScan sample.
- 26 November 2025
- Published by American Association for Cancer Research (AACR)
Open AccessSupplementary Figure S1 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization
Supplementary Figure S1 shows forest plot for minimally adjusted model for the PanScan sample.